Lista de obras - Valeria Crippa - Dominio Público Uruguay

17-AAG increases autophagic removal of mutant androgen receptor in spinal and bulbar muscular atrophy.

artículo científico publicado en 2010

A role of small heat shock protein B8 (HspB8) in the autophagic removal of misfolded proteins responsible for neurodegenerative diseases

artículo científico publicado en 2010

ALS-related misfolded protein management in motor neurons and muscle cells

artículo científico publicado en 2014

Aberrant Autophagic Response in The Muscle of A Knock-in Mouse Model of Spinal and Bulbar Muscular Atrophy.

artículo científico publicado en 2015

Aggregation and proteasome: the case of elongated polyglutamine aggregation in spinal and bulbar muscular atrophy.

artículo científico publicado en 2006

Alteration of protein folding and degradation in motor neuron diseases: Implications and protective functions of small heat shock proteins.

artículo científico publicado en 2011

Clearance of the mutant androgen receptor in motoneuronal models of spinal and bulbar muscular atrophy.

artículo científico publicado en 2013

Differences in protein quality control correlate with phenotype variability in 2 mouse models of familial amyotrophic lateral sclerosis.

artículo científico publicado en 2014

Different anti-aggregation and pro-degradative functions of the members of the mammalian sHSP family in neurological disorders.

artículo científico

Differential autophagy power in the spinal cord and muscle of transgenic ALS mice.

artículo científico publicado en 2013

Dysfunction of constitutive and inducible ubiquitin-proteasome system in amyotrophic lateral sclerosis: implication for protein aggregation and immune response.

artículo científico

Dysregulation of axonal transport and motorneuron diseases.

artículo científico publicado en 2011

HSC70 expression is reduced in lymphomonocytes of sporadic ALS patients and contributes to TDP-43 accumulation

artículo científico publicado en 2019

Inhibition of retrograde transport modulates misfolded protein accumulation and clearance in motoneuron diseases.

artículo científico publicado en 2017

Loss-of-function mutations in the SIGMAR1 gene cause distal hereditary motor neuropathy by impairing ER-mitochondria tethering and Ca2+ signalling

artículo científico publicado en 2016

Modulators of estrogen receptor inhibit proliferation and migration of prostate cancer cells.

artículo científico publicado en 2013

Motoneuronal and muscle-selective removal of ALS-related misfolded proteins.

artículo científico

Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis

artículo científico publicado en 2011

Mutation of SOD1 in ALS: a gain of a loss of function

artículo científico publicado en 2007

Proteasomal and autophagic degradative activities in spinal and bulbar muscular atrophy.

artículo científico publicado en 2010

Retinoic Acid Downregulates HSPB8 Gene Expression in Human Breast Cancer Cells MCF-7

artículo científico publicado en 2021

Synergic prodegradative activity of Bicalutamide and trehalose on the mutant androgen receptor responsible for spinal and bulbar muscular atrophy

artículo científico publicado en 2014

Tdp-25 Routing to Autophagy and Proteasome Ameliorates its Aggregation in Amyotrophic Lateral Sclerosis Target Cells

artículo científico publicado en 2018

The Role of the Heat Shock Protein B8 (HSPB8) in Motoneuron Diseases.

artículo científico publicado en 2017

The Role of the Protein Quality Control System in SBMA.

artículo científico publicado en 2015

The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis

artículo científico publicado en 2011

The chaperone HSPB8 reduces the accumulation of truncated TDP-43 species in cells and protects against TDP-43-mediated toxicity.

artículo científico publicado en 2016

The role of the polyglutamine tract in androgen receptor.

artículo científico publicado en 2007

The small heat shock protein B8 (HSPB8) efficiently removes aggregating species of dipeptides produced in C9ORF72-related neurodegenerative diseases.

artículo científico publicado en 2017

The small heat shock protein B8 (HSPB8) modulates proliferation and migration of breast cancer cells

artículo científico publicado en 2017

The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).

artículo científico publicado en 2010

Transcriptional induction of the heat shock protein B8 mediates the clearance of misfolded proteins responsible for motor neuron diseases

artículo científico publicado en 2016

Trehalose induces autophagy via lysosomal-mediated TFEB activation in models of motoneuron degeneration

scientific article published on 05 November 2018

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

artículo científico publicado en 2016

Lista de obras de Valeria Crippa

17-AAG increases autophagic removal of mutant androgen receptor in spinal and bulbar muscular atrophy.

A role of small heat shock protein B8 (HspB8) in the autophagic removal of misfolded proteins responsible for neurodegenerative diseases

ALS-related misfolded protein management in motor neurons and muscle cells

Aberrant Autophagic Response in The Muscle of A Knock-in Mouse Model of Spinal and Bulbar Muscular Atrophy.

Aggregation and proteasome: the case of elongated polyglutamine aggregation in spinal and bulbar muscular atrophy.

Alteration of protein folding and degradation in motor neuron diseases: Implications and protective functions of small heat shock proteins.

Clearance of the mutant androgen receptor in motoneuronal models of spinal and bulbar muscular atrophy.

Differences in protein quality control correlate with phenotype variability in 2 mouse models of familial amyotrophic lateral sclerosis.

Different anti-aggregation and pro-degradative functions of the members of the mammalian sHSP family in neurological disorders.

Differential autophagy power in the spinal cord and muscle of transgenic ALS mice.

Dysfunction of constitutive and inducible ubiquitin-proteasome system in amyotrophic lateral sclerosis: implication for protein aggregation and immune response.

Dysregulation of axonal transport and motorneuron diseases.

HSC70 expression is reduced in lymphomonocytes of sporadic ALS patients and contributes to TDP-43 accumulation

Inhibition of retrograde transport modulates misfolded protein accumulation and clearance in motoneuron diseases.

Loss-of-function mutations in the SIGMAR1 gene cause distal hereditary motor neuropathy by impairing ER-mitochondria tethering and Ca2+ signalling

Modulators of estrogen receptor inhibit proliferation and migration of prostate cancer cells.

Motoneuronal and muscle-selective removal of ALS-related misfolded proteins.

Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis

Mutation of SOD1 in ALS: a gain of a loss of function

Proteasomal and autophagic degradative activities in spinal and bulbar muscular atrophy.

Retinoic Acid Downregulates HSPB8 Gene Expression in Human Breast Cancer Cells MCF-7

Synergic prodegradative activity of Bicalutamide and trehalose on the mutant androgen receptor responsible for spinal and bulbar muscular atrophy

Tdp-25 Routing to Autophagy and Proteasome Ameliorates its Aggregation in Amyotrophic Lateral Sclerosis Target Cells

The Role of the Heat Shock Protein B8 (HSPB8) in Motoneuron Diseases.

The Role of the Protein Quality Control System in SBMA.

The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis

The chaperone HSPB8 reduces the accumulation of truncated TDP-43 species in cells and protects against TDP-43-mediated toxicity.

The role of the polyglutamine tract in androgen receptor.

The small heat shock protein B8 (HSPB8) efficiently removes aggregating species of dipeptides produced in C9ORF72-related neurodegenerative diseases.

The small heat shock protein B8 (HSPB8) modulates proliferation and migration of breast cancer cells

The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).

Transcriptional induction of the heat shock protein B8 mediates the clearance of misfolded proteins responsible for motor neuron diseases

Trehalose induces autophagy via lysosomal-mediated TFEB activation in models of motoneuron degeneration

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)