Lista de obras -

Competition between influenza A virus genome segments

artículo científico publicado en 2012

Coxsackievirus mutants that can bypass host factor PI4KIIIβ and the need for high levels of PI4P lipids for replication

artículo científico publicado en 2012

Display of the human mucinome with defined O-glycans by gene engineered cells

artículo científico publicado en 2021

Dissection of the influenza A virus endocytic routes reveals macropinocytosis as an alternative entry pathway

artículo científico publicado en 2011

Enhanced Inhibition of Influenza A Virus Adhesion by Di- and Trivalent Hemagglutinin Inhibitors

scientific article published on 28 June 2019

Enterovirus D68 receptor requirements unveiled by haploid genetics.

artículo científico publicado en 2016

Evolution of the hemagglutinin protein of the new pandemic H1N1 influenza virus: maintaining optimal receptor binding by compensatory substitutions.

artículo científico publicado en 2013

Genetic versus antigenic differences among highly pathogenic H5N1 avian influenza A viruses: Consequences for vaccine strain selection

artículo científico publicado en 2017

Glycan-dependent immunogenicity of recombinant soluble trimeric hemagglutinin

artículo científico publicado en 2012

Highly Pathogenic Influenza A(H5Nx) Viruses with Altered H5 Receptor-Binding Specificity

scientific article published on 15 February 2017

Human-type sialic acid receptors contribute to avian influenza A virus binding and entry by hetero-multivalent interactions

artículo científico publicado en 2022

Identification of Residues That Affect Oligomerization and/or Enzymatic Activity of Influenza Virus H5N1 Neuraminidase Proteins

artículo científico publicado en 2016

Influenza A Virus Hemagglutinin-Neuraminidase-Receptor Balance: Preserving Virus Motility

artículo científico publicado en 2020

Influenza A virus entry into cells lacking sialylated N-glycans

artículo científico publicado en 2012

Inhibition of the ubiquitin-proteasome system affects influenza A virus infection at a postfusion step

artículo científico publicado en 2010

Kinetic analysis of the influenza A virus HA/NA balance reveals contribution of NA to virus-receptor binding and NA-dependent rolling on receptor-containing surfaces

artículo científico publicado en 2018

Mutation of the Second Sialic Acid-Binding Site, Resulting in Reduced Neuraminidase Activity, Preceded the Emergence of H7N9 Influenza A Virus

artículo científico publicado en 2017

Mutation of the second sialic acid-binding site of influenza A virus neuraminidase drives compensatory mutations in hemagglutinin

artículo científico publicado en 2020

Only two residues are responsible for the dramatic difference in receptor binding between swine and new pandemic H1 hemagglutinin

artículo científico publicado en 2010

Rapid Emergence of Highly Pathogenic Avian Influenza Subtypes from a Subtype H5N1 Hemagglutinin Variant

artículo científico publicado en 2015

Role of enhanced receptor engagement in the evolution of a pandemic acute hemorrhagic conjunctivitis virus ⬇️

artículo científico publicado en 2017

Second sialic acid-binding site of influenza A virus neuraminidase: Binding receptors for efficient release

artículo científico publicado en 2020

Substitutions T200A and E227A in the hemagglutinin of pandemic 2009 influenza A virus increase lethality but decrease transmission

artículo científico publicado en 2013

Substrate Binding by the Second Sialic Acid-Binding Site of Influenza A Virus N1 Neuraminidase Contributes to Enzymatic Activity

scholarly article by Wenjuan Du published in October 2018

The 2nd sialic acid-binding site of influenza A virus neuraminidase is an important determinant of the hemagglutinin-neuraminidase-receptor balance.

artículo científico publicado en 2019

The influenza A virus hemagglutinin glycosylation state affects receptor-binding specificity

artículo científico publicado en 2010

Lista de obras de

Competition between influenza A virus genome segments

Coxsackievirus mutants that can bypass host factor PI4KIIIβ and the need for high levels of PI4P lipids for replication

Display of the human mucinome with defined O-glycans by gene engineered cells

Dissection of the influenza A virus endocytic routes reveals macropinocytosis as an alternative entry pathway

Enhanced Inhibition of Influenza A Virus Adhesion by Di- and Trivalent Hemagglutinin Inhibitors

Enterovirus D68 receptor requirements unveiled by haploid genetics.

Evolution of the hemagglutinin protein of the new pandemic H1N1 influenza virus: maintaining optimal receptor binding by compensatory substitutions.

Genetic versus antigenic differences among highly pathogenic H5N1 avian influenza A viruses: Consequences for vaccine strain selection

Glycan-dependent immunogenicity of recombinant soluble trimeric hemagglutinin

Highly Pathogenic Influenza A(H5Nx) Viruses with Altered H5 Receptor-Binding Specificity

Human-type sialic acid receptors contribute to avian influenza A virus binding and entry by hetero-multivalent interactions

Identification of Residues That Affect Oligomerization and/or Enzymatic Activity of Influenza Virus H5N1 Neuraminidase Proteins

Influenza A Virus Hemagglutinin-Neuraminidase-Receptor Balance: Preserving Virus Motility

Influenza A virus entry into cells lacking sialylated N-glycans

Inhibition of the ubiquitin-proteasome system affects influenza A virus infection at a postfusion step

Kinetic analysis of the influenza A virus HA/NA balance reveals contribution of NA to virus-receptor binding and NA-dependent rolling on receptor-containing surfaces

Mutation of the Second Sialic Acid-Binding Site, Resulting in Reduced Neuraminidase Activity, Preceded the Emergence of H7N9 Influenza A Virus

Mutation of the second sialic acid-binding site of influenza A virus neuraminidase drives compensatory mutations in hemagglutinin

Only two residues are responsible for the dramatic difference in receptor binding between swine and new pandemic H1 hemagglutinin

Rapid Emergence of Highly Pathogenic Avian Influenza Subtypes from a Subtype H5N1 Hemagglutinin Variant

Role of enhanced receptor engagement in the evolution of a pandemic acute hemorrhagic conjunctivitis virus ⬇️

Second sialic acid-binding site of influenza A virus neuraminidase: Binding receptors for efficient release

Substitutions T200A and E227A in the hemagglutinin of pandemic 2009 influenza A virus increase lethality but decrease transmission

Substrate Binding by the Second Sialic Acid-Binding Site of Influenza A Virus N1 Neuraminidase Contributes to Enzymatic Activity

The 2nd sialic acid-binding site of influenza A virus neuraminidase is an important determinant of the hemagglutinin-neuraminidase-receptor balance.

The influenza A virus hemagglutinin glycosylation state affects receptor-binding specificity