Lista de obras -

A transcriptome-based classifier to determine molecular subtypes in medulloblastoma

scientific article published on 29 October 2020

ATR-Chk1 axis protects BCR/ABL leukemia cells from the lethal effect of DNA double-strand breaks.

artículo científico publicado en 2006

BCR/ABL and other kinases from chronic myeloproliferative disorders stimulate single-strand annealing, an unfaithful DNA double-strand break repair

artículo científico publicado en 2008

BCR/ABL inhibits mismatch repair to protect from apoptosis and induce point mutations

artículo científico publicado en 2008

BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

artículo científico publicado en 2006

BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells

artículo científico publicado en 2005

BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species-dependent DNA double-strand breaks

scientific article published on 10 August 2004

BCR/ABL recruits p53 tumor suppressor protein to induce drug resistance

artículo científico publicado en 2004

BLM helicase is activated in BCR/ABL leukemia cells to modulate responses to cisplatin.

artículo científico publicado en 2005

Crk and CrkL are required for cell transformation by v-fos and v-ras

artículo científico publicado en 2015

Fibroblast Growth Requires CT10 Regulator of Kinase (Crk) and Crk-like (CrkL).

artículo científico publicado en 2016

Genomic instability may originate from imatinib-refractory chronic myeloid leukemia stem cells

artículo científico publicado en 2013

HGG-40. HIGH GRADE GLIOMA CELL LINE COHORT AS AN EXAMPLE OF CHILDREN’S BRAIN TUMOR TISSUE CONSORTIUM TUMOR SPECIMEN PROCESSING PIPELINE

IRS-1-Rad51 nuclear interaction sensitizes JCV T-antigen positive medulloblastoma cells to genotoxic treatment

artículo científico publicado en 2006

Imatinib sensitivity in BCR-ABL1-positive chronic myeloid leukemia cells is regulated by the remaining normal ABL1 allele

artículo científico publicado en 2011

Inhibition of Stat5a/b Enhances Proteasomal Degradation of Androgen Receptor Liganded by Antiandrogens in Prostate Cancer.

artículo científico publicado en 2014

Integrated Proteogenomic Characterization across Major Histological Types of Pediatric Brain Cancer

artículo científico publicado en 2020

MODL-28. Patient-derived, three-dimensional organoid platform for pediatric brain tumor modeling

scientific article published in 2022

MODL-30. Children’s Brain Tumor Network preclinical tumor models development and sharing platform: collaborative model empowering pediatric brain tumor discovery and global research.

scientific article published in 2022

Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases

artículo científico publicado en 2016

OTHR-07. CIRCULATING MIRNAS AS A NON-INVASIVE MONITORING TOOL FOR PEDIATRIC BRAIN TUMORS

scientific article published in 2023

OTHR-43. Composition of cell-free miRNA in cerebrospinal fluid and plasma as a monitoring tool for pediatric brain tumors

scientific article published in 2022

Pediatric high-grade glioma resources from the Children's Brain Tumor Tissue Consortium

artículo científico publicado en 2020

Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors

artículo científico publicado en 2012

Signal transducer and activator of transcription 5a/b: biomarker and therapeutic target in prostate and breast cancer

artículo científico publicado en 2011

Lista de obras de

A transcriptome-based classifier to determine molecular subtypes in medulloblastoma

ATR-Chk1 axis protects BCR/ABL leukemia cells from the lethal effect of DNA double-strand breaks.

BCR/ABL and other kinases from chronic myeloproliferative disorders stimulate single-strand annealing, an unfaithful DNA double-strand break repair

BCR/ABL inhibits mismatch repair to protect from apoptosis and induce point mutations

BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells

BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species-dependent DNA double-strand breaks

BCR/ABL recruits p53 tumor suppressor protein to induce drug resistance

BLM helicase is activated in BCR/ABL leukemia cells to modulate responses to cisplatin.

Crk and CrkL are required for cell transformation by v-fos and v-ras

Fibroblast Growth Requires CT10 Regulator of Kinase (Crk) and Crk-like (CrkL).

Genomic instability may originate from imatinib-refractory chronic myeloid leukemia stem cells

HGG-40. HIGH GRADE GLIOMA CELL LINE COHORT AS AN EXAMPLE OF CHILDREN’S BRAIN TUMOR TISSUE CONSORTIUM TUMOR SPECIMEN PROCESSING PIPELINE

IRS-1-Rad51 nuclear interaction sensitizes JCV T-antigen positive medulloblastoma cells to genotoxic treatment

Imatinib sensitivity in BCR-ABL1-positive chronic myeloid leukemia cells is regulated by the remaining normal ABL1 allele

Inhibition of Stat5a/b Enhances Proteasomal Degradation of Androgen Receptor Liganded by Antiandrogens in Prostate Cancer.

Integrated Proteogenomic Characterization across Major Histological Types of Pediatric Brain Cancer

MODL-28. Patient-derived, three-dimensional organoid platform for pediatric brain tumor modeling

MODL-30. Children’s Brain Tumor Network preclinical tumor models development and sharing platform: collaborative model empowering pediatric brain tumor discovery and global research.

Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases

OTHR-07. CIRCULATING MIRNAS AS A NON-INVASIVE MONITORING TOOL FOR PEDIATRIC BRAIN TUMORS

OTHR-43. Composition of cell-free miRNA in cerebrospinal fluid and plasma as a monitoring tool for pediatric brain tumors

Pediatric high-grade glioma resources from the Children's Brain Tumor Tissue Consortium

Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors

Signal transducer and activator of transcription 5a/b: biomarker and therapeutic target in prostate and breast cancer

TMOD-18. AN INTEGRATED SET OF PEDIATRIC HIGH GRADE GLIOMA RESOURCES FOR TRANSLATIONAL STUDIES

TMOD-20. THE PEDIATRIC BRAIN TUMOR ATLAS: AN INITIATIVE BY THE CHILDREN’S BRAIN TUMOR TISSUE CONSORTIUM AND PACIFIC PEDIATRIC NEUROONCOLOGY CONSORTIUM