Lista de obras -

Activation of pruritogenic TGR5, MrgprA3, and MrgprC11 on colon-innervating afferents induces visceral hypersensitivity

scientific article published on 17 October 2019

CO-EXPRESSION OF μ AND ∂ OPIOID RECEPTORS BY MOUSE COLONIC NOCICEPTORS.

artículo científico publicado en 2018

Chronic linaclotide treatment reduces colitis-induced neuroplasticity and reverses persistent bladder dysfunction

article

Colonic afferent input and dorsal horn neuron activation differs between the thoracolumbar and lumbosacral spinal cord

artículo científico publicado en 2019

Conopeptide-Derived κ-Opioid Agonists (Conorphins): Potent, Selective, and Metabolic Stable Dynorphin A Mimetics with Antinociceptive Properties.

artículo científico publicado en 2016

Contribution of membrane receptor signalling to chronic visceral pain

artículo científico publicado en 2018

Cyclic analogues of α-conotoxin Vc1.1 inhibit colonic nociceptors and provide analgesia in a mouse model of chronic abdominal pain

artículo científico publicado en 2017

Enterochromaffin Cells Are Gut Chemosensors that Couple to Sensory Neural Pathways.

artículo científico publicado en 2017

Histamine induces peripheral and central hypersensitivity to bladder distension via the histamine H1 receptor and TRPV1

artículo científico publicado en 2019

Linaclotide treatment reduces endometriosis-associated vaginal hyperalgesia and mechanical allodynia through viscerovisceral cross-talk

artículo científico publicado en 2019

Multiple sodium channel isoforms mediate the pathological effects of Pacific ciguatoxin-1.

artículo científico publicado en 2017

NaV 1.6 regulates excitability of mechanosensitive sensory neurons

artículo científico publicado en 2019

NaV1.1 inhibition can reduce visceral hypersensitivity

artículo científico publicado en 2018

Protease-activated receptor-2 in endosomes signals persistent pain of irritable bowel syndrome

article published in the Proceedings of the National Academy of Sciences of the United States of America

Selective spider toxins reveal a role for the Nav1.1 channel in mechanical pain

scientific journal article

Selenoether oxytocin analogues have analgesic properties in a mouse model of chronic abdominal pain

scientific article published in Nature Communications

Structure-Activity Studies of Cysteine-Rich α-Conotoxins that Inhibit High-Voltage-Activated Calcium Channels via GABA(B) Receptor Activation Reveal a Minimal Functional Motif.

artículo científico publicado en 2016

Structure–Activity Studies Reveal the Molecular Basis for GABAB-Receptor Mediated Inhibition of High Voltage-Activated Calcium Channels by α-Conotoxin Vc1.1

artículo científico publicado en 2018

Translating peripheral bladder afferent mechanosensitivity to neuronal activation within the lumbosacral spinal cord of mice

scientific article published on 01 April 2019

Voltage-gated sodium channels: (NaV )igating the field to determine their contribution to visceral nociception.

artículo científico publicado en 2018

α-Conotoxin Vc1.1 inhibits human dorsal root ganglion neuroexcitability and mouse colonic nociception via GABAB receptors.

artículo científico publicado en 2016

Lista de obras de

Activation of pruritogenic TGR5, MrgprA3, and MrgprC11 on colon-innervating afferents induces visceral hypersensitivity

CO-EXPRESSION OF μ AND ∂ OPIOID RECEPTORS BY MOUSE COLONIC NOCICEPTORS.

Chronic linaclotide treatment reduces colitis-induced neuroplasticity and reverses persistent bladder dysfunction

Colonic afferent input and dorsal horn neuron activation differs between the thoracolumbar and lumbosacral spinal cord

Conopeptide-Derived κ-Opioid Agonists (Conorphins): Potent, Selective, and Metabolic Stable Dynorphin A Mimetics with Antinociceptive Properties.

Contribution of membrane receptor signalling to chronic visceral pain

Cyclic analogues of α-conotoxin Vc1.1 inhibit colonic nociceptors and provide analgesia in a mouse model of chronic abdominal pain

Enterochromaffin Cells Are Gut Chemosensors that Couple to Sensory Neural Pathways.

Histamine induces peripheral and central hypersensitivity to bladder distension via the histamine H1 receptor and TRPV1

Linaclotide treatment reduces endometriosis-associated vaginal hyperalgesia and mechanical allodynia through viscerovisceral cross-talk

Multiple sodium channel isoforms mediate the pathological effects of Pacific ciguatoxin-1.

NaV 1.6 regulates excitability of mechanosensitive sensory neurons

NaV1.1 inhibition can reduce visceral hypersensitivity

Protease-activated receptor-2 in endosomes signals persistent pain of irritable bowel syndrome

Selective spider toxins reveal a role for the Nav1.1 channel in mechanical pain

Selenoether oxytocin analogues have analgesic properties in a mouse model of chronic abdominal pain

Structure-Activity Studies of Cysteine-Rich α-Conotoxins that Inhibit High-Voltage-Activated Calcium Channels via GABA(B) Receptor Activation Reveal a Minimal Functional Motif.

Structure–Activity Studies Reveal the Molecular Basis for GABAB-Receptor Mediated Inhibition of High Voltage-Activated Calcium Channels by α-Conotoxin Vc1.1

Translating peripheral bladder afferent mechanosensitivity to neuronal activation within the lumbosacral spinal cord of mice

Voltage-gated sodium channels: (NaV )igating the field to determine their contribution to visceral nociception.

α-Conotoxin Vc1.1 inhibits human dorsal root ganglion neuroexcitability and mouse colonic nociception via GABAB receptors.