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Comment: postpartum hormones oxytocin and prolactin cause pro-arrhythmic prolongation of cardiac repolarization in long QT syndrome type 2-Authors' reply

artículo científico publicado en 2019

Electro-mechanical (dys-)function in long QT syndrome type 1

scientific article published on 09 July 2018

Electro-mechanical dysfunction in long QT syndrome: Role for arrhythmogenic risk prediction and modulation by sex and sex hormones

artículo científico publicado en 2015

Increased risk due to ventricular arrhythmias after transmyocardial laser-revascularisation? Preliminary results.

artículo científico publicado en 1997

Nicorandil normalizes prolonged repolarisation in the first transgenic rabbit model with Long-QT syndrome 1 both in vitro and in vivo ⬇️

artículo científico publicado el 17 de octubre de 2010

Pacemaker therapy in very elderly patients: long-term survival and prognostic parameters ⬇️

artículo científico publicado el 1 de noviembre de 2003

Postpartum hormones oxytocin and prolactin cause pro-arrhythmic prolongation of cardiac repolarization in long QT syndrome type 2

scientific article published on 01 July 2019

Risk stratification in long QT syndrome: Are we finally getting closer to a mutation-specific assessment of an individual patient’s arrhythmogenic risk? ⬇️

artículo científico publicado el 13 de febrero de 2013

SAP97 interacts with Kv1.5 in heterologous expression systems ⬇️

artículo científico publicado el 1 de diciembre de 2001

Spatial correlation of action potential duration and diastolic dysfunction in transgenic and drug-induced LQT2 rabbits.

artículo científico publicado en 2013

Transgenic short-QT syndrome 1 rabbits mimic the human disease phenotype with QT/action potential duration shortening in the atria and ventricles and increased ventricular tachycardia/ventricular fibrillation inducibility

scientific article published on 01 March 2019

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