Lista de obras -

Allele-dependent thermodynamic and structural perturbations in ApoE variants associated with the correction of dyslipidemia and formation of spherical ApoE-containing HDL particles.

artículo científico publicado en 2012

Alteration of myocardial GRK2 produces a global metabolic phenotype.

artículo científico publicado en 2019

Amyloid-Beta (1-40) Peptide and Subclinical Cardiovascular Disease

scientific article published on 01 August 2018

Cardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications ⬇️

artículo científico publicado el 1 de junio de 2013

Cardiac Myocyte KLF5 Regulates Ppara Expression and Cardiac Function

artículo científico publicado en 2015

Cardiac myocyte KLF5 regulates body weight via alteration of cardiac FGF21

scientific article published on 26 April 2019

Ceramide is a cardiotoxin in lipotoxic cardiomyopathy

artículo científico publicado en 2008

Chemically synthesized Secoisolariciresinol diglucoside (LGM2605) improves mitochondrial function in cardiac myocytes and alleviates septic cardiomyopathy

scientific article published on 03 January 2019

DNA Checkpoint and Repair Factors Are Nuclear Sensors for Intracellular Organelle Stresses-Inflammations and Cancers Can Have High Genomic Risks.

artículo científico publicado en 2018

Dual peroxisome-proliferator-activated-receptor-α/γ activation inhibits SIRT1-PGC1α axis and causes cardiac dysfunction

scientific article published on 08 August 2019

Endothelial cell CD36 optimizes tissue fatty acid uptake

scientific article published on 26 July 2018

Enhanced efferocytosis of apoptotic cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction.

artículo científico publicado en 2013

Fatty Acid Oxidation Promotes Cardiomyocyte Proliferation Rate but Does Not Change Cardiomyocyte Number in Infant Mice

scientific article published on 22 March 2019

Fatty old hearts: role of cardiac lipotoxicity in age-related cardiomyopathy

artículo científico publicado en 2016

Fish oil selectively improves heart function in a mouse model of lipid-induced cardiomyopathy

artículo científico publicado en 2013

GRK2 compromises cardiomyocyte mitochondrial function by diminishing fatty acid-mediated oxygen consumption and increasing superoxide levels

artículo científico publicado en 2015

Human aldose reductase expression accelerates atherosclerosis in diabetic apolipoprotein E-/- mice

artículo científico publicado en 2011

Hyperhomocysteinemia potentiates diabetes-impaired EDHF-induced vascular relaxation: Role of insufficient hydrogen sulfide.

artículo científico publicado en 2018

Increased de novo ceramide synthesis and accumulation in failing myocardium

artículo científico publicado en 2017

Increased de novo ceramide synthesis and accumulation in failing myocardium

scientific article published on 04 May 2017

Inhibition of NADPH oxidase 2 (NOX2) prevents sepsis-induced cardiomyopathy by improving calcium handling and mitochondrial function.

artículo científico publicado en 2017

Inhibition of c-Jun-N-terminal kinase increases cardiac peroxisome proliferator-activated receptor alpha expression and fatty acid oxidation and prevents lipopolysaccharide-induced heart dysfunction.

artículo científico publicado en 2011

Krüppel-like factors: Crippling and un-crippling metabolic pathways.

artículo científico publicado en 2018

Lipid Use and Misuse by the Heart.

scientific article published on May 2016

Lipolysis, and not hepatic lipogenesis, is the primary modulator of triglyceride levels in streptozotocin-induced diabetic mice.

artículo científico publicado en 2014

Molecular etiology of a dominant form of type III hyperlipoproteinemia caused by R142C substitution in apoE4

artículo científico publicado en 2010

Myocardial Strain and Cardiac Output are Preferable Measurements for Cardiac Dysfunction and Can Predict Mortality in Septic Mice.

artículo científico publicado en 2019

Nutriepigenetics and cardiovascular disease

artículo científico publicado en 2018

PPARs: Protectors or Opponents of Myocardial Function?

artículo científico publicado en 2015

Palmitic acid and DGAT1 deficiency enhance osteoclastogenesis, while oleic acid-induced triglyceride formation prevents it

artículo científico publicado en 2014

Pathophysiology of sepsis-related cardiac dysfunction: driven by inflammation, energy mismanagement, or both?

artículo científico publicado en 2015

Residues Leu261, Trp264, and Phe265 account for apolipoprotein E-induced dyslipidemia and affect the formation of apolipoprotein E-containing high-density lipoprotein.

artículo científico publicado en 2007

Restricting mitochondrial GRK2 post-ischemia confers cardioprotection by reducing myocyte death and maintaining glucose oxidation

scientific article published on 11 December 2018

Savings precede spending: fatty acid utilization relies on triglyceride formation for cardiac energetics.

artículo científico publicado en 2014

Sequestration of fatty acids in triglycerides prevents endoplasmic reticulum stress in an in vitro model of cardiomyocyte lipotoxicity

artículo científico publicado en 2014

miR-195 Regulates Metabolism in Failing Myocardium via Alterations in SIRT3 Expression and Mitochondrial Protein Acetylation.

artículo científico publicado en 2018

Lista de obras de

Allele-dependent thermodynamic and structural perturbations in ApoE variants associated with the correction of dyslipidemia and formation of spherical ApoE-containing HDL particles.

Alteration of myocardial GRK2 produces a global metabolic phenotype.

Amyloid-Beta (1-40) Peptide and Subclinical Cardiovascular Disease

Cardiac Lipotoxicity: Molecular Pathways and Therapeutic Implications ⬇️

Cardiac Myocyte KLF5 Regulates Ppara Expression and Cardiac Function

Cardiac myocyte KLF5 regulates body weight via alteration of cardiac FGF21

Ceramide is a cardiotoxin in lipotoxic cardiomyopathy

Chemically synthesized Secoisolariciresinol diglucoside (LGM2605) improves mitochondrial function in cardiac myocytes and alleviates septic cardiomyopathy

DNA Checkpoint and Repair Factors Are Nuclear Sensors for Intracellular Organelle Stresses-Inflammations and Cancers Can Have High Genomic Risks.

Dual peroxisome-proliferator-activated-receptor-α/γ activation inhibits SIRT1-PGC1α axis and causes cardiac dysfunction

Endothelial cell CD36 optimizes tissue fatty acid uptake

Enhanced efferocytosis of apoptotic cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction.

Fatty Acid Oxidation Promotes Cardiomyocyte Proliferation Rate but Does Not Change Cardiomyocyte Number in Infant Mice

Fatty old hearts: role of cardiac lipotoxicity in age-related cardiomyopathy

Fish oil selectively improves heart function in a mouse model of lipid-induced cardiomyopathy

GRK2 compromises cardiomyocyte mitochondrial function by diminishing fatty acid-mediated oxygen consumption and increasing superoxide levels

Human aldose reductase expression accelerates atherosclerosis in diabetic apolipoprotein E-/- mice

Hyperhomocysteinemia potentiates diabetes-impaired EDHF-induced vascular relaxation: Role of insufficient hydrogen sulfide.

Increased de novo ceramide synthesis and accumulation in failing myocardium

Increased de novo ceramide synthesis and accumulation in failing myocardium

Inhibition of NADPH oxidase 2 (NOX2) prevents sepsis-induced cardiomyopathy by improving calcium handling and mitochondrial function.

Inhibition of c-Jun-N-terminal kinase increases cardiac peroxisome proliferator-activated receptor alpha expression and fatty acid oxidation and prevents lipopolysaccharide-induced heart dysfunction.

Krüppel-like factors: Crippling and un-crippling metabolic pathways.

Lipid Use and Misuse by the Heart.

Lipolysis, and not hepatic lipogenesis, is the primary modulator of triglyceride levels in streptozotocin-induced diabetic mice.

Molecular etiology of a dominant form of type III hyperlipoproteinemia caused by R142C substitution in apoE4

Myocardial Strain and Cardiac Output are Preferable Measurements for Cardiac Dysfunction and Can Predict Mortality in Septic Mice.

Nutriepigenetics and cardiovascular disease

PPARs: Protectors or Opponents of Myocardial Function?

Palmitic acid and DGAT1 deficiency enhance osteoclastogenesis, while oleic acid-induced triglyceride formation prevents it

Pathophysiology of sepsis-related cardiac dysfunction: driven by inflammation, energy mismanagement, or both?

Residues Leu261, Trp264, and Phe265 account for apolipoprotein E-induced dyslipidemia and affect the formation of apolipoprotein E-containing high-density lipoprotein.

Restricting mitochondrial GRK2 post-ischemia confers cardioprotection by reducing myocyte death and maintaining glucose oxidation

Savings precede spending: fatty acid utilization relies on triglyceride formation for cardiac energetics.

Sequestration of fatty acids in triglycerides prevents endoplasmic reticulum stress in an in vitro model of cardiomyocyte lipotoxicity

miR-195 Regulates Metabolism in Failing Myocardium via Alterations in SIRT3 Expression and Mitochondrial Protein Acetylation.