Lista de obras -

A Chemically Modified Curcumin (CMC 2.24) Inhibits Nuclear Factor κB Activation and Inflammatory Bone Loss in Murine Models of LPS-Induced Experimental Periodontitis and Diabetes-Associated Natural Periodontitis.

artículo científico publicado en 2017

A novel chemically modified curcumin reduces severity of experimental periodontal disease in rats: initial observations

artículo científico publicado en 2014

Chemopreventive activity of systemically administered curcumin on oral cancer in the 4-nitroquinoline 1-oxide model

artículo científico publicado en 2015

Curcumin abrogates LPS-induced pro-inflammatory cytokines in RAW 264.7 macrophages. Evidence for novel mechanisms involving SOCS-1, -3 and p38 MAPK

artículo científico publicado en 2013

Differential effects of natural Curcumin and chemically modified curcumin on inflammation and bone resorption in model of experimental periodontitis.

artículo científico publicado en 2018

Differential regulation of MMP-13 expression in two models of experimentally induced periodontal disease in rats.

artículo científico publicado en 2009

Effects of long-term FK 506 therapy on the alveolar bone and cementum of rats.

artículo científico publicado en 2009

Experimental development of bisphosphonate-related osteonecrosis of the jaws in rodents

artículo científico publicado en 2013

Influence of antiplatelet drugs in the pathogenesis of experimental periodontitis and periodontal repair in rats

artículo científico publicado en 2010

Overexpression of Bcl-2, SOCS 1, 3 and Cdh 1, 2 are associated with the early neoplasic changes in modified 4-nitroquinoline 1-oxide-induced murine oral cancer model.

artículo científico publicado en 2016

Potent anti-inflammatory effects of systemically administered curcumin modulate periodontal disease in vivo.

artículo científico publicado en 2011

Protective effects of Tacrolimus, a calcineurin inhibitor, in experimental periodontitis in rats

scientific article published on 23 March 2007

Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells.