Lista de obras -

Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis.

artículo científico publicado en 2011

Abnormalities in α/β-CaMKII and related mechanisms suggest synaptic dysfunction in hippocampus of LPA1 receptor knockout mice.

artículo científico publicado en 2010

Acute Inescapable Stress Rapidly Increases Synaptic Energy Metabolism in Prefrontal Cortex and Alters Working Memory Performance

artículo científico publicado en 2019

Acute stress is not acute: sustained enhancement of glutamate release after acute stress involves readily releasable pool size and synapsin I activation.

artículo científico publicado en 2016

Acute stress rapidly increases the readily releasable pool of glutamate vesicles in prefrontal and frontal cortex through non-genomic action of corticosterone.

artículo científico publicado en 2014

Altered mechanisms underlying the abnormal glutamate release in amyotrophic lateral sclerosis at a pre-symptomatic stage of the disease

artículo científico publicado en 2016

Antidepressant Treatments Change 5-HT2C Receptor mRNA Expression in Rat Prefrontal/Frontal Cortex and Hippocampus

artículo científico publicado en 2011

Blockade of stress-induced increase of glutamate release in the rat prefrontal/frontal cortex by agomelatine involves synergy between melatonergic and 5-HT2C receptor-dependent pathways

artículo científico publicado en 2010

Chronic antidepressant treatments induce a time-dependent up-regulation of AMPA receptor subunit protein levels.

artículo científico publicado en 2011

Chronic antidepressants induce redistribution and differential activation of alphaCaM kinase II between presynaptic compartments.

artículo científico publicado en 2007

Chronic antidepressants reduce depolarization-evoked glutamate release and protein interactions favoring formation of SNARE complex in hippocampus.

artículo científico publicado en 2005

Correction: Acute Stress Increases Depolarization-Evoked Glutamate Release in the Rat Prefrontal/Frontal Cortex: The Dampening Action of Antidepressants.

artículo científico publicado en 2010

Depression-Associated Gene Negr1-Fgfr2 Pathway Is Altered by Antidepressant Treatment

scientific article published on 31 July 2020

Early induction of CREB activation and CREB-regulating signalling by antidepressants.

artículo científico publicado en 2009

Early raise of BDNF in hippocampus suggests induction of posttranscriptional mechanisms by antidepressants

artículo científico publicado en 2009

Expression and Dendritic Trafficking of BDNF-6 Splice Variant are Impaired in Knock-In Mice Carrying Human BDNF Val66Met Polymorphism.

artículo científico publicado en 2015

Expression profiling of a genetic animal model of depression reveals novel molecular pathways underlying depressive-like behaviours

artículo científico publicado en 2010

Functional and structural remodeling of glutamate synapses in prefrontal and frontal cortex induced by behavioral stress.

artículo científico publicado en 2015

Glutamate hypothesis of depression and its consequences for antidepressant treatments.

artículo científico publicado en 2012

Long-term soluble Abeta1-40 activates CaM kinase II in organotypic hippocampal cultures.

artículo científico publicado en 2006

Mode of action of agomelatine: synergy between melatonergic and 5-HT2C receptors.

artículo científico publicado en 2011

Pharmacological characterization of BDNF promoters I, II and IV reveals that serotonin and norepinephrine input is sufficient for transcription activation

artículo científico publicado en 2014

Physical Exercise and Antidepressants Enhance BDNF Targeting in Hippocampal CA3 Dendrites: Further Evidence of a Spatial Code for BDNF Splice Variants ⬇️

artículo científico publicado el 8 de febrero de 2012

Physical exercise and acute restraint stress differentially modulate hippocampal brain-derived neurotrophic factor transcripts and epigenetic mechanisms in mice.

artículo científico publicado en 2015

Regulation of editing and expression of glutamate alpha-amino-propionic-acid (AMPA)/kainate receptors by antidepressant drugs

artículo científico publicado en 2006

Signaling pathways regulating gene expression, neuroplasticity, and neurotrophic mechanisms in the action of antidepressants: a critical overview.

artículo científico publicado en 2006

Stress and corticosterone increase the readily releasable pool of glutamate vesicles in synaptic terminals of prefrontal and frontal cortex.

artículo científico publicado en 2014

Stress, glucocorticoids and glutamate release: Effects of antidepressant drugs ⬇️

artículo científico publicado el 13 de junio de 2011

The action of antidepressants on the glutamate system: regulation of glutamate release and glutamate receptors.

artículo científico

Time-dependent activation of MAPK/Erk1/2 and Akt/GSK3 cascades: modulation by agomelatine.

artículo científico publicado en 2014

miRNome Profiling Detects miR-101-3p and miR-142-5p as Putative Blood Biomarkers of Frailty Syndrome

artículo científico publicado en 2022

Lista de obras de

Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis.

Abnormalities in α/β-CaMKII and related mechanisms suggest synaptic dysfunction in hippocampus of LPA1 receptor knockout mice.

Acute Inescapable Stress Rapidly Increases Synaptic Energy Metabolism in Prefrontal Cortex and Alters Working Memory Performance

Acute stress is not acute: sustained enhancement of glutamate release after acute stress involves readily releasable pool size and synapsin I activation.

Acute stress rapidly increases the readily releasable pool of glutamate vesicles in prefrontal and frontal cortex through non-genomic action of corticosterone.

Altered mechanisms underlying the abnormal glutamate release in amyotrophic lateral sclerosis at a pre-symptomatic stage of the disease

Antidepressant Treatments Change 5-HT2C Receptor mRNA Expression in Rat Prefrontal/Frontal Cortex and Hippocampus

Blockade of stress-induced increase of glutamate release in the rat prefrontal/frontal cortex by agomelatine involves synergy between melatonergic and 5-HT2C receptor-dependent pathways

Chronic antidepressant treatments induce a time-dependent up-regulation of AMPA receptor subunit protein levels.

Chronic antidepressants induce redistribution and differential activation of alphaCaM kinase II between presynaptic compartments.

Chronic antidepressants reduce depolarization-evoked glutamate release and protein interactions favoring formation of SNARE complex in hippocampus.

Correction: Acute Stress Increases Depolarization-Evoked Glutamate Release in the Rat Prefrontal/Frontal Cortex: The Dampening Action of Antidepressants.

Depression-Associated Gene Negr1-Fgfr2 Pathway Is Altered by Antidepressant Treatment

Early induction of CREB activation and CREB-regulating signalling by antidepressants.

Early raise of BDNF in hippocampus suggests induction of posttranscriptional mechanisms by antidepressants

Expression and Dendritic Trafficking of BDNF-6 Splice Variant are Impaired in Knock-In Mice Carrying Human BDNF Val66Met Polymorphism.

Expression profiling of a genetic animal model of depression reveals novel molecular pathways underlying depressive-like behaviours

Functional and structural remodeling of glutamate synapses in prefrontal and frontal cortex induced by behavioral stress.

Glutamate hypothesis of depression and its consequences for antidepressant treatments.

Long-term soluble Abeta1-40 activates CaM kinase II in organotypic hippocampal cultures.

Mode of action of agomelatine: synergy between melatonergic and 5-HT2C receptors.

Pharmacological characterization of BDNF promoters I, II and IV reveals that serotonin and norepinephrine input is sufficient for transcription activation

Physical Exercise and Antidepressants Enhance BDNF Targeting in Hippocampal CA3 Dendrites: Further Evidence of a Spatial Code for BDNF Splice Variants ⬇️

Physical exercise and acute restraint stress differentially modulate hippocampal brain-derived neurotrophic factor transcripts and epigenetic mechanisms in mice.

Regulation of editing and expression of glutamate alpha-amino-propionic-acid (AMPA)/kainate receptors by antidepressant drugs

Signaling pathways regulating gene expression, neuroplasticity, and neurotrophic mechanisms in the action of antidepressants: a critical overview.

Stress and corticosterone increase the readily releasable pool of glutamate vesicles in synaptic terminals of prefrontal and frontal cortex.

Stress, glucocorticoids and glutamate release: Effects of antidepressant drugs ⬇️

The action of antidepressants on the glutamate system: regulation of glutamate release and glutamate receptors.

Time-dependent activation of MAPK/Erk1/2 and Akt/GSK3 cascades: modulation by agomelatine.

miRNome Profiling Detects miR-101-3p and miR-142-5p as Putative Blood Biomarkers of Frailty Syndrome