Lista de obras -

APP depletion alters selective pre- and post-synaptic proteins

scientific article published on 11 February 2019

Capillary cerebral amyloid angiopathy is associated with vessel occlusion and cerebral blood flow disturbances

scientific article published on 21 March 2008

Co-occurrence of Alzheimer's disease ß-amyloid and τ pathologies at synapses.

artículo científico publicado en 2008

Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease

article

Corrigendum to "Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease"

scientific article published on 29 January 2019

Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice.

artículo científico publicado en 2012

Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice.

artículo científico publicado en 2010

High-resolution 3D reconstruction reveals intra-synaptic amyloid fibrils.

artículo científico publicado en 2011

Inter-laboratory comparison of neuropathological assessments of β-amyloid protein: a study of the BrainNet Europe consortium

artículo científico publicado en 2008

Intraneuronal Aβ accumulation, amyloid plaques, and synapse pathology in Alzheimer's disease.

artículo científico publicado en 2012

Mangiferin and Morin Attenuate Oxidative Stress, Mitochondrial Dysfunction, and Neurocytotoxicity, Induced by Amyloid Beta Oligomers.

artículo científico publicado en 2018

New, Fully Implantable Device for Selective Clearance of CSF-Target Molecules: Proof of Concept in a Murine Model of Alzheimer’s Disease

artículo científico publicado en 2022

P2X4 receptor controls microglia activation and favors remyelination in autoimmune encephalitis

artículo científico publicado en 2018

Selective vulnerability of different types of commissural neurons for amyloid beta-protein-induced neurodegeneration in APP23 mice correlates with dendritic tree morphology.

artículo científico publicado en 2006

Synaptic activity protects against AD and FTD-like pathology via autophagic-lysosomal degradation.

artículo científico

Targeting Beta-Amyloid at the CSF: A New Therapeutic Strategy in Alzheimer's Disease.

artículo científico publicado en 2018

The development of amyloid beta protein deposits in the aged brain.

artículo científico publicado en 2006

The endocytic pathway in microglia during health, aging and Alzheimer's disease

artículo científico publicado en 2016

Tracing of temporo-entorhinal connections in the human brain: cognitively impaired argyrophilic grain disease cases show dendritic alterations but no axonal disconnection of temporo-entorhinal association neurons.

artículo científico publicado en 2007

Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology.

artículo científico publicado en 2012

Vesicular glutamate release from axons in white matter.

artículo científico publicado en 2007

Lista de obras de

APP depletion alters selective pre- and post-synaptic proteins

Capillary cerebral amyloid angiopathy is associated with vessel occlusion and cerebral blood flow disturbances

Co-occurrence of Alzheimer's disease ß-amyloid and τ pathologies at synapses.

Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease

Corrigendum to "Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease"

Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice.

Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice.

High-resolution 3D reconstruction reveals intra-synaptic amyloid fibrils.

Inter-laboratory comparison of neuropathological assessments of β-amyloid protein: a study of the BrainNet Europe consortium

Intraneuronal Aβ accumulation, amyloid plaques, and synapse pathology in Alzheimer's disease.

Mangiferin and Morin Attenuate Oxidative Stress, Mitochondrial Dysfunction, and Neurocytotoxicity, Induced by Amyloid Beta Oligomers.

New, Fully Implantable Device for Selective Clearance of CSF-Target Molecules: Proof of Concept in a Murine Model of Alzheimer’s Disease

P2X4 receptor controls microglia activation and favors remyelination in autoimmune encephalitis

Selective vulnerability of different types of commissural neurons for amyloid beta-protein-induced neurodegeneration in APP23 mice correlates with dendritic tree morphology.

Synaptic activity protects against AD and FTD-like pathology via autophagic-lysosomal degradation.

Targeting Beta-Amyloid at the CSF: A New Therapeutic Strategy in Alzheimer's Disease.

The development of amyloid beta protein deposits in the aged brain.

The endocytic pathway in microglia during health, aging and Alzheimer's disease

Tracing of temporo-entorhinal connections in the human brain: cognitively impaired argyrophilic grain disease cases show dendritic alterations but no axonal disconnection of temporo-entorhinal association neurons.

Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology.

Vesicular glutamate release from axons in white matter.