Lista de obras -

A Proapoptotic Peptide Derived from Reovirus Outer Capsid Protein μ1 Has Membrane-Destabilizing Activity ⬇️

artículo científico publicado el 24 de noviembre de 2010

Assaying for Structural Variation in the Parvovirus Capsid and Its Role in Infection ⬇️

artículo científico publicado el 10 de octubre de 1998

Bacterial filtration efficiency of green soy protein based nanofiber air filter

artículo científico publicado en 2014

Conserved surface residues on the feline calicivirus (FCV) capsid are essential for interaction with its receptor feline junctional adhesion molecule A (fJAM-A).

artículo científico publicado en 2018

Distribution of the feline calicivirus receptor junctional adhesion molecule a in feline tissues.

artículo científico publicado en 2010

Feline caliciviruses (FCVs) isolated from cats with virulent systemic disease possess in vitro phenotypes distinct from those of other FCV isolates

artículo científico publicado en 2007

Identification of regions and residues in feline junctional adhesion molecule required for feline calicivirus binding and infection.

artículo científico publicado en 2007

Increased ubiquitination and other covariant phenotypes attributed to a strain- and temperature-dependent defect of reovirus core protein mu2.

artículo científico publicado en 2004

Mammalian orthoreovirus Infection is Enhanced in Cells Pre-Treated with Sodium Arsenite

scientific article published on 18 June 2019

Micro-total analysis system for virus detection: microfluidic pre-concentration coupled to liposome-based detection.

artículo científico publicado en 2011

Molecular virology of feline calicivirus.

artículo científico publicado en 2008

Nucleoside and RNA triphosphatase activities of orthoreovirus transcriptase cofactor mu2

artículo científico publicado en 2004

Putative autocleavage of outer capsid protein micro1, allowing release of myristoylated peptide micro1N during particle uncoating, is critical for cell entry by reovirus.

artículo científico publicado en 2004

Reovirus core protein mu2 determines the filamentous morphology of viral inclusion bodies by interacting with and stabilizing microtubules.

artículo científico publicado en 2002

Reovirus infection or ectopic expression of outer capsid protein micro1 induces apoptosis independently of the cellular proapoptotic proteins Bax and Bak.

artículo científico publicado en 2010

Reovirus σ3 protein limits interferon expression and cell death induction

artículo científico publicado en 2020

The cellular chaperone hsc70 is specifically recruited to reovirus viral factories independently of its chaperone function.

artículo científico publicado en 2011

The delta region of outer-capsid protein micro 1 undergoes conformational change and release from reovirus particles during cell entry.

artículo científico publicado en 2003

The multi-functional reovirus σ3 protein is a virulence factor that suppresses stress granule formation and is associated with myocardial injury

artículo científico publicado en 2021

The natural host range shift and subsequent evolution of canine parvovirus resulted from virus-specific binding to the canine transferrin receptor ⬇️

artículo científico publicado en 2003

Virus-mediated compartmentalization of the host translational machinery.

artículo científico publicado en 2014

Lista de obras de

A Proapoptotic Peptide Derived from Reovirus Outer Capsid Protein μ1 Has Membrane-Destabilizing Activity ⬇️

Assaying for Structural Variation in the Parvovirus Capsid and Its Role in Infection ⬇️

Bacterial filtration efficiency of green soy protein based nanofiber air filter

Conserved surface residues on the feline calicivirus (FCV) capsid are essential for interaction with its receptor feline junctional adhesion molecule A (fJAM-A).

Distribution of the feline calicivirus receptor junctional adhesion molecule a in feline tissues.

Feline caliciviruses (FCVs) isolated from cats with virulent systemic disease possess in vitro phenotypes distinct from those of other FCV isolates

Identification of regions and residues in feline junctional adhesion molecule required for feline calicivirus binding and infection.

Increased ubiquitination and other covariant phenotypes attributed to a strain- and temperature-dependent defect of reovirus core protein mu2.

Mammalian orthoreovirus Infection is Enhanced in Cells Pre-Treated with Sodium Arsenite

Micro-total analysis system for virus detection: microfluidic pre-concentration coupled to liposome-based detection.

Molecular virology of feline calicivirus.

Nucleoside and RNA triphosphatase activities of orthoreovirus transcriptase cofactor mu2

Putative autocleavage of outer capsid protein micro1, allowing release of myristoylated peptide micro1N during particle uncoating, is critical for cell entry by reovirus.

Reovirus core protein mu2 determines the filamentous morphology of viral inclusion bodies by interacting with and stabilizing microtubules.

Reovirus infection or ectopic expression of outer capsid protein micro1 induces apoptosis independently of the cellular proapoptotic proteins Bax and Bak.

Reovirus σ3 protein limits interferon expression and cell death induction

The cellular chaperone hsc70 is specifically recruited to reovirus viral factories independently of its chaperone function.

The delta region of outer-capsid protein micro 1 undergoes conformational change and release from reovirus particles during cell entry.

The multi-functional reovirus σ3 protein is a virulence factor that suppresses stress granule formation and is associated with myocardial injury

The natural host range shift and subsequent evolution of canine parvovirus resulted from virus-specific binding to the canine transferrin receptor ⬇️

Virus-mediated compartmentalization of the host translational machinery.