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A novel non genomic glucocorticoid signaling mediated by a membrane palmitoylated glucocorticoid receptor cross talks with GnRH in gonadotrope cells

artículo científico publicado en 2017

Accessory proteins for G proteins: partners in signaling.

artículo científico publicado en 2006

Carbon Black Nanoparticles Inhibit Aromatase Expression and Estradiol Secretion in Human Granulosa Cells Through the ERK1/2 Pathway.

artículo científico publicado en 2017

Concomitant increase of G protein-coupled receptor kinase activity and uncoupling of beta-adrenergic receptors in rat myometrium at parturition

artículo científico publicado en 2001

Decoding high Gonadotropin-releasing hormone pulsatility: a role for GnRH receptor coupling to the cAMP pathway?

artículo científico publicado en 2012

Distribution of activator of G-protein signaling 3 within the aggresomal pathway: role of specific residues in the tetratricopeptide repeat domain and differential regulation by the AGS3 binding partners Gi(alpha) and mammalian inscuteable.

artículo científico publicado en 2010

Endogenous G protein-coupled receptor kinase 6 triggers homologous beta-adrenergic receptor desensitization in primary uterine smooth muscle cells

scientific journal article

GnRH regulates the expression of its receptor accessory protein SET in pituitary gonadotropes

artículo científico publicado en 2018

Influence of the accessory protein SET on M3 muscarinic receptor phosphorylation and G protein coupling.

artículo científico publicado en 2012

Mechanisms underlying the tissue-specific and regulated activity of the Gnrhr promoter in mammals.

artículo científico publicado en 2012

SET protein interacts with intracellular domains of the gonadotropin-releasing hormone receptor and differentially regulates receptor signaling to cAMP and calcium in gonadotrope cells.

artículo científico publicado en 2012

The proto-oncogene SET interacts with muscarinic receptors and attenuates receptor signaling.

artículo científico publicado en 2006

Unsaturated fatty acids disrupt Smad signaling in gonadotrope cells leading to inhibition of FSHβ gene expression.

artículo científico publicado en 2013