Lista de obras -

Arachidonic acid enhances intracellular [Ca2+]i increase and mitochondrial depolarization induced by glutamate in cerebellar granule cells.

artículo científico publicado en 2006

Blockade of ADP-induced Ca2+-signal and platelet aggregation by lipoxygenase inhibitors.

artículo científico publicado en 1989

Blockade of mitochondrial Ca2+ uptake by mitochondrial inhibitors amplifies the glutamate-induced calcium response in cultured cerebellar granule cells.

artículo científico publicado en 1999

Changes in ATP content in cerebellar granule cells during hyperstimulation of glutamate receptors: possible role of NO and nitrite ions.

artículo científico publicado en 2007

Cortexin and combination of nitrite with cortexin decrease swelling and destruction of cerebellar neurons in hemorrhagic stroke.

artículo científico publicado en 2009

Decreased intraplatelet Ca2+ release and ATP secretion in pediatric nephrotic syndrome

artículo científico publicado en 1999

Dramatic effects of external alkalinity on neuronal calcium recovery following a short-duration glutamate challenge: the role of the plasma membrane Ca2+/H+ pump

artículo científico publicado en 1995

Effect of antibodies against AMPA glutamate receptors on brain neurons in primary cultures of the cerebellum and hippocampus.

artículo científico publicado en 2006

Effect of enteropeptidase on survival of cultured hippocampal neurons under conditions of glutamate toxicity.

artículo científico publicado en 2010

Effect of neurotrophin-3 precursor on glutamate-induced calcium homeostasis deregulation in rat cerebellum granule cells.

artículo científico publicado en 2015

Effects of selective inhibitors of neuronal and inducible NO-synthase on ATP content and survival of cultured rat cerebellar neurons during hyperstimulation of glutamate receptors.

artículo científico publicado en 2013

Effects of semax and its Pro-Gly-Pro fragment on calcium homeostasis of neurons and their survival under conditions of glutamate toxicity.

artículo científico publicado en 2007

Endothelial protein C receptor is expressed in rat cortical and hippocampal neurons and is necessary for protective effect of activated protein C at glutamate excitotoxicity.

artículo científico publicado en 2009

KB-R7943, an inhibitor of the reverse Na+ /Ca2+ exchanger, blocks N-methyl-D-aspartate receptor and inhibits mitochondrial complex I.

artículo científico publicado en 2011

LDL induces intracellular signalling and cell migration via atypical LDL-binding protein T-cadherin.

artículo científico publicado en 2005

Li+ protects nerve cells against destabilization of Ca2+ homeostasis and delayed death caused by removal of external Na+

artículo científico publicado en 1999

Mitochondrial H2O2 as an enable signal for triggering autophosphorylation of insulin receptor in neurons.

artículo científico publicado en 2013

Modulation of hippocampal neuron survival by thrombin and factor Xa.

artículo científico publicado en 2006

NF-κB-dependent and -independent pathways in the protective effects of activated protein C in hippocampal and cortical neurons at excitotoxicity.

artículo científico publicado en 2013

Na+/Ca2+ exchange and regulation of cytoplasmic concentration of calcium in rat cerebellar neurons treated with glutamate.

artículo científico publicado en 2007

Phosphono analogues of 2-oxoglutarate protect cerebellar granule neurons upon glutamate excitotoxicity.

artículo científico publicado en 2009

Protease-activated receptor (PAR)1-mediated anti-apoptotic effect of activated protein C on glutamate excitotoxicity in hippocampal neurons

artículo científico publicado en 2007

Proteinase-activated type 1 receptors are involved in the mechanism of protection of rat hippocampal neurons from glutamate toxicity.

artículo científico publicado en 2005

Role of Na+/Ca2+ exchange in regulation of neuronal Ca2+ homeostasis requires re-evaluation

artículo científico publicado en 1998

Role of protein kinase C in Ca(2+) homeostasis disorders in cultured rat neurons during hyperstimulation of glutamate receptors.

artículo científico publicado en 2008

Single fluorescent protein-based Ca2+ sensors with increased dynamic range.

artículo científico publicado en 2007

Study on ATP concentration changes in cytosol of individual cultured neurons during glutamate-induced deregulation of calcium homeostasis.

artículo científico publicado en 2014

The leading role of mitochondrial depolarization in the mechanism of glutamate-induced disruptions in Ca2+ homeostasis.

artículo científico publicado en 2002

[APOΕ gene polymorphism and markers of brain damage in the outcomes of severe traumatic brain injury in children]

scientific article published on 01 January 2020

[Biomarkers in children with traumatic brain injury]

artículo científico publicado en 2015

[The International Baikal seminar 'Innovations in neurology and neurosurgery'-2015']

artículo científico publicado en 2016

Lista de obras de

Arachidonic acid enhances intracellular [Ca2+]i increase and mitochondrial depolarization induced by glutamate in cerebellar granule cells.

Blockade of ADP-induced Ca2+-signal and platelet aggregation by lipoxygenase inhibitors.

Blockade of mitochondrial Ca2+ uptake by mitochondrial inhibitors amplifies the glutamate-induced calcium response in cultured cerebellar granule cells.

Changes in ATP content in cerebellar granule cells during hyperstimulation of glutamate receptors: possible role of NO and nitrite ions.

Cortexin and combination of nitrite with cortexin decrease swelling and destruction of cerebellar neurons in hemorrhagic stroke.

Decreased intraplatelet Ca2+ release and ATP secretion in pediatric nephrotic syndrome

Dramatic effects of external alkalinity on neuronal calcium recovery following a short-duration glutamate challenge: the role of the plasma membrane Ca2+/H+ pump

Effect of antibodies against AMPA glutamate receptors on brain neurons in primary cultures of the cerebellum and hippocampus.

Effect of enteropeptidase on survival of cultured hippocampal neurons under conditions of glutamate toxicity.

Effect of neurotrophin-3 precursor on glutamate-induced calcium homeostasis deregulation in rat cerebellum granule cells.

Effects of selective inhibitors of neuronal and inducible NO-synthase on ATP content and survival of cultured rat cerebellar neurons during hyperstimulation of glutamate receptors.

Effects of semax and its Pro-Gly-Pro fragment on calcium homeostasis of neurons and their survival under conditions of glutamate toxicity.

Endothelial protein C receptor is expressed in rat cortical and hippocampal neurons and is necessary for protective effect of activated protein C at glutamate excitotoxicity.

KB-R7943, an inhibitor of the reverse Na+ /Ca2+ exchanger, blocks N-methyl-D-aspartate receptor and inhibits mitochondrial complex I.

LDL induces intracellular signalling and cell migration via atypical LDL-binding protein T-cadherin.

Li+ protects nerve cells against destabilization of Ca2+ homeostasis and delayed death caused by removal of external Na+

Mitochondrial H2O2 as an enable signal for triggering autophosphorylation of insulin receptor in neurons.

Modulation of hippocampal neuron survival by thrombin and factor Xa.

NF-κB-dependent and -independent pathways in the protective effects of activated protein C in hippocampal and cortical neurons at excitotoxicity.

Na+/Ca2+ exchange and regulation of cytoplasmic concentration of calcium in rat cerebellar neurons treated with glutamate.

Phosphono analogues of 2-oxoglutarate protect cerebellar granule neurons upon glutamate excitotoxicity.

Protease-activated receptor (PAR)1-mediated anti-apoptotic effect of activated protein C on glutamate excitotoxicity in hippocampal neurons

Proteinase-activated type 1 receptors are involved in the mechanism of protection of rat hippocampal neurons from glutamate toxicity.

Role of Na+/Ca2+ exchange in regulation of neuronal Ca2+ homeostasis requires re-evaluation

Role of protein kinase C in Ca(2+) homeostasis disorders in cultured rat neurons during hyperstimulation of glutamate receptors.

Single fluorescent protein-based Ca2+ sensors with increased dynamic range.

Study on ATP concentration changes in cytosol of individual cultured neurons during glutamate-induced deregulation of calcium homeostasis.

The leading role of mitochondrial depolarization in the mechanism of glutamate-induced disruptions in Ca2+ homeostasis.

[APOΕ gene polymorphism and markers of brain damage in the outcomes of severe traumatic brain injury in children]

[Biomarkers in children with traumatic brain injury]

[The International Baikal seminar 'Innovations in neurology and neurosurgery'-2015']