Lista de obras - Wei Kong - Dominio Público Uruguay

ADAM17: A Molecular Switch to Control TNFR2 During Atherogenesis In Vivo

artículo científico publicado en 2017

ADAMTS-7 inhibits re-endothelialization of injured arteries and promotes vascular remodeling through cleavage of thrombospondin-1.

artículo científico publicado en 2015

COMP-prohibitin 2 interaction maintains mitochondrial homeostasis and controls smooth muscle cell identity.

artículo científico publicado en 2018

CYLD Deubiquitinates Nicotinamide Adenine Dinucleotide Phosphate Oxidase 4 Contributing to Adventitial Remodeling.

artículo científico publicado en 2017

Cartilage oligomeric matrix protein is a natural inhibitor of thrombin.

artículo científico

Cartilage oligomeric matrix protein is a novel notch ligand driving embryonic stem cell differentiation towards the smooth muscle lineage

artículo científico publicado en 2018

Cartilage oligomeric matrix protein prevents vascular aging and vascular smooth muscle cells senescence

artículo científico publicado en 2016

DeSiphering receptor core-induced and ligand-dependent conformational changes in arrestin via genetic encoded trimethylsilyl 1H-NMR probe

scientific article published on 25 September 2020

Deficiency of FAM3D (Family With Sequence Similarity 3, Member D), A Novel Chemokine, Attenuates Neutrophil Recruitment and Ameliorates Abdominal Aortic Aneurysm Development.

artículo científico publicado en 2018

FAM3D is essential for colon homeostasis and host defense against inflammation associated carcinogenesis

artículo científico publicado en 2020

Homocysteine directly interacts and activates the angiotensin II type I receptor to aggravate vascular injury

artículo científico publicado en 2018

Hyperhomocysteinaemia and vascular injury: advances in mechanisms and drug targets.

artículo científico publicado en 2017

Hyperhomocysteinemia exaggerates adventitial inflammation and angiotensin II-induced abdominal aortic aneurysm in mice

artículo científico publicado en 2012

Microsomal Prostaglandin E Synthase-1-Derived PGE2 Inhibits Vascular Smooth Muscle Cell Calcification

artículo científico publicado en 2015

PHB2 Maintains the Contractile Phenotype of VSMCs by Counteracting PKM2 Splicing

artículo científico publicado en 2022

Rapamycin prevents thoracic aortic aneurysm and dissection in mice

scientific article published on 22 September 2018

Shift of Macrophage Phenotype Due to Cartilage Oligomeric Matrix Protein Deficiency Drives Atherosclerotic Calcification.

artículo científico publicado en 2016

Targeting macrophage TFEB-14-3-3 epsilon Interface by naringenin inhibits abdominal aortic aneurysm

artículo científico publicado en 2022

Unspliced XBP1 Confers VSMC Homeostasis and Prevents Aortic Aneurysm Formation via FoxO4 Interaction

artículo científico publicado en 2017

Upregulation of a disintegrin and metalloproteinase with thrombospondin motifs-7 by miR-29 repression mediates vascular smooth muscle calcification.

artículo científico publicado en 2012

Lista de obras de Wei Kong

ADAM17: A Molecular Switch to Control TNFR2 During Atherogenesis In Vivo

ADAMTS-7 inhibits re-endothelialization of injured arteries and promotes vascular remodeling through cleavage of thrombospondin-1.

COMP-prohibitin 2 interaction maintains mitochondrial homeostasis and controls smooth muscle cell identity.

CYLD Deubiquitinates Nicotinamide Adenine Dinucleotide Phosphate Oxidase 4 Contributing to Adventitial Remodeling.

Cartilage oligomeric matrix protein is a natural inhibitor of thrombin.

Cartilage oligomeric matrix protein is a novel notch ligand driving embryonic stem cell differentiation towards the smooth muscle lineage

Cartilage oligomeric matrix protein prevents vascular aging and vascular smooth muscle cells senescence

DeSiphering receptor core-induced and ligand-dependent conformational changes in arrestin via genetic encoded trimethylsilyl 1H-NMR probe

Deficiency of FAM3D (Family With Sequence Similarity 3, Member D), A Novel Chemokine, Attenuates Neutrophil Recruitment and Ameliorates Abdominal Aortic Aneurysm Development.

FAM3D is essential for colon homeostasis and host defense against inflammation associated carcinogenesis

Homocysteine directly interacts and activates the angiotensin II type I receptor to aggravate vascular injury

Hyperhomocysteinaemia and vascular injury: advances in mechanisms and drug targets.

Hyperhomocysteinemia exaggerates adventitial inflammation and angiotensin II-induced abdominal aortic aneurysm in mice

Microsomal Prostaglandin E Synthase-1-Derived PGE2 Inhibits Vascular Smooth Muscle Cell Calcification

PHB2 Maintains the Contractile Phenotype of VSMCs by Counteracting PKM2 Splicing

Rapamycin prevents thoracic aortic aneurysm and dissection in mice

Shift of Macrophage Phenotype Due to Cartilage Oligomeric Matrix Protein Deficiency Drives Atherosclerotic Calcification.

Targeting macrophage TFEB-14-3-3 epsilon Interface by naringenin inhibits abdominal aortic aneurysm

Unspliced XBP1 Confers VSMC Homeostasis and Prevents Aortic Aneurysm Formation via FoxO4 Interaction

Upregulation of a disintegrin and metalloproteinase with thrombospondin motifs-7 by miR-29 repression mediates vascular smooth muscle calcification.