Lista de obras - Christopher M Adams - Dominio Público Uruguay

Activating transcription factor 4 (ATF4) promotes skeletal muscle atrophy by forming a heterodimer with the transcriptional regulator C/EBPβ

artículo científico publicado en 2020

Alterations in the muscle force transfer apparatus in aged rats during unloading and reloading: impact of microRNA-31

scientific article published on 01 July 2018

Altered mRNA expression after long-term soleus electrical stimulation training in humans with paralysis

artículo científico publicado en 2011

Amino Acid Sensing in Skeletal Muscle

artículo científico publicado en 2016

Atf4 regulates obesity, glucose homeostasis, and energy expenditure

artículo científico publicado en 2009

Control of skeletal muscle atrophy in response to disuse: clinical/preclinical contentions and fallacies of evidence

artículo científico publicado en 2016

Enhanced locomotion caused by loss of the Drosophila DEG/ENaC protein Pickpocket1.

artículo científico publicado en 2003

Evidence for metabolic aberrations in asymptomatic persons with type 2 diabetes after initiation of simvastatin therapy

artículo científico publicado en 2015

Gadd45a Protein Promotes Skeletal Muscle Atrophy by Forming a Complex with the Protein Kinase MEKK4

artículo científico publicado en 2016

Identification and Small Molecule Inhibition of an Activating Transcription Factor 4 (ATF4)-dependent Pathway to Age-related Skeletal Muscle Weakness and Atrophy.

artículo científico publicado en 2015

Inhibition of the Epithelial Na+ Channel by Interaction of Nedd4 with a PY Motif Deleted in Liddle's Syndrome ⬇️

artículo científico publicado el 6 de noviembre de 1998

Insulin signaling and the general amino acid control response. Two distinct pathways to amino acid synthesis and uptake.

artículo científico publicado en 2008

Interactions between subunits of the human epithelial sodium channel.

artículo científico publicado en 1997

Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel

artículo científico publicado en 1995

OPA1 deficiency promotes secretion of FGF21 from muscle that prevents obesity and insulin resistance.

artículo científico publicado en 2017

Obesity challenges the hepatoprotective function of the integrated stress response to asparaginase exposure in mice.

artículo científico publicado en 2017

Protons activate brain Na+ channel 1 by inducing a conformational change that exposes a residue associated with neurodegeneration.

artículo científico publicado en 1998

Rapid and Sensitive Quantification of Ursolic Acid and Oleanolic Acid in Human Plasma Using Ultra-performance Liquid Chromatography-Mass Spectrometry

artículo científico publicado en 2018

Ripped pocket and pickpocket, novel Drosophila DEG/ENaC subunits expressed in early development and in mechanosensory neurons.

artículo científico publicado en 1998

Role of ATF4 in skeletal muscle atrophy.

artículo científico publicado en 2017

Role of the transcription factor ATF4 in the anabolic actions of insulin and the anti-anabolic actions of glucocorticoids

artículo científico publicado en 2007

Skeletal muscle denervation causes skeletal muscle atrophy through a pathway that involves both Gadd45a and HDAC4.

scientific article published on 13 August 2013

Spermine oxidase maintains basal skeletal muscle gene expression and fiber size and is strongly repressed by conditions that cause skeletal muscle atrophy.

artículo científico publicado en 2014

Stress-induced skeletal muscle Gadd45a expression reprograms myonuclei and causes muscle atrophy.

artículo científico publicado en 2012

Systems-based discovery of tomatidine as a natural small molecule inhibitor of skeletal muscle atrophy

artículo científico publicado en 2014

Tetraethylammonium block of the BNC1 channel.

artículo científico publicado en 1999

Three mutations in sterol-sensing domain of SCAP block interaction with insig and render SREBP cleavage insensitive to sterols.

artículo científico publicado en 2002

Transcription factor ATF4 directs basal and stress-induced gene expression in the unfolded protein response and cholesterol metabolism in the liver.

artículo científico publicado en 2016

Ursolic acid increases skeletal muscle and brown fat and decreases diet-induced obesity, glucose intolerance and fatty liver disease

artículo científico publicado en 2012

Use of mRNA expression signatures to discover small molecule inhibitors of skeletal muscle atrophy

artículo científico publicado en 2015

mRNA expression signatures of human skeletal muscle atrophy identify a natural compound that increases muscle mass

artículo científico publicado en 2011

p53 and ATF4 mediate distinct and additive pathways to skeletal muscle atrophy during limb immobilization

artículo científico publicado en 2014

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

artículo científico publicado en 2016

Lista de obras de Christopher M Adams

Activating transcription factor 4 (ATF4) promotes skeletal muscle atrophy by forming a heterodimer with the transcriptional regulator C/EBPβ

Alterations in the muscle force transfer apparatus in aged rats during unloading and reloading: impact of microRNA-31

Altered mRNA expression after long-term soleus electrical stimulation training in humans with paralysis

Amino Acid Sensing in Skeletal Muscle

Atf4 regulates obesity, glucose homeostasis, and energy expenditure

Control of skeletal muscle atrophy in response to disuse: clinical/preclinical contentions and fallacies of evidence

Enhanced locomotion caused by loss of the Drosophila DEG/ENaC protein Pickpocket1.

Evidence for metabolic aberrations in asymptomatic persons with type 2 diabetes after initiation of simvastatin therapy

Gadd45a Protein Promotes Skeletal Muscle Atrophy by Forming a Complex with the Protein Kinase MEKK4

Identification and Small Molecule Inhibition of an Activating Transcription Factor 4 (ATF4)-dependent Pathway to Age-related Skeletal Muscle Weakness and Atrophy.

Inhibition of the Epithelial Na+ Channel by Interaction of Nedd4 with a PY Motif Deleted in Liddle's Syndrome ⬇️

Insulin signaling and the general amino acid control response. Two distinct pathways to amino acid synthesis and uptake.

Interactions between subunits of the human epithelial sodium channel.

Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel

OPA1 deficiency promotes secretion of FGF21 from muscle that prevents obesity and insulin resistance.

Obesity challenges the hepatoprotective function of the integrated stress response to asparaginase exposure in mice.

Protons activate brain Na+ channel 1 by inducing a conformational change that exposes a residue associated with neurodegeneration.

Rapid and Sensitive Quantification of Ursolic Acid and Oleanolic Acid in Human Plasma Using Ultra-performance Liquid Chromatography-Mass Spectrometry

Ripped pocket and pickpocket, novel Drosophila DEG/ENaC subunits expressed in early development and in mechanosensory neurons.

Role of ATF4 in skeletal muscle atrophy.

Role of the transcription factor ATF4 in the anabolic actions of insulin and the anti-anabolic actions of glucocorticoids

Skeletal muscle denervation causes skeletal muscle atrophy through a pathway that involves both Gadd45a and HDAC4.

Spermine oxidase maintains basal skeletal muscle gene expression and fiber size and is strongly repressed by conditions that cause skeletal muscle atrophy.

Stress-induced skeletal muscle Gadd45a expression reprograms myonuclei and causes muscle atrophy.

Systems-based discovery of tomatidine as a natural small molecule inhibitor of skeletal muscle atrophy

Tetraethylammonium block of the BNC1 channel.

Three mutations in sterol-sensing domain of SCAP block interaction with insig and render SREBP cleavage insensitive to sterols.

Transcription factor ATF4 directs basal and stress-induced gene expression in the unfolded protein response and cholesterol metabolism in the liver.

Ursolic acid increases skeletal muscle and brown fat and decreases diet-induced obesity, glucose intolerance and fatty liver disease

Use of mRNA expression signatures to discover small molecule inhibitors of skeletal muscle atrophy

mRNA expression signatures of human skeletal muscle atrophy identify a natural compound that increases muscle mass

p53 and ATF4 mediate distinct and additive pathways to skeletal muscle atrophy during limb immobilization

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)