Lista de obras - Rommy von Bernhardi - Dominio Público Uruguay

ATP is required and advances cytokine-induced gap junction formation in microglia in vitro.

artículo científico publicado en 2013

Acetylcholinesterase induces the expression of the beta-amyloid precursor protein in glia and activates glial cells in culture.

artículo científico publicado en 2003

Age-Dependent Changes in the Activation and Regulation of Microglia.

artículo científico publicado en 2016

Age-dependent changes on TGFβ1 Smad3 pathway modify the pattern of microglial cell activation

artículo científico publicado en 2013

Alzheimer's disease: redox dysregulation as a common denominator for diverse pathogenic mechanisms.

artículo científico

Astrocytes inhibit nitric oxide-dependent Ca(2+) dynamics in activated microglia: involvement of ATP released via pannexin 1 channels.

artículo científico publicado en 2013

Aβ potentiates inflammatory activation of glial cells induced by scavenger receptor ligands and inflammatory mediators in culture.

artículo científico publicado en 2012

Commentaries on Viewpoint: The ongoing need for good physiological investigation: Obstructive sleep apnea in HIV patients as a paradigm

artículo científico publicado en 2015

Connexin43 hemichannels mediate secondary cellular damage spread from the trauma zone to distal zones in astrocyte monolayers.

artículo científico publicado en 2015

D-serine released by astrocytes in brainstem regulates breathing response to CO2 levels

artículo científico publicado en 2017

Expression Pattern of Scavenger Receptors and Amyloid-β Phagocytosis of Astrocytes and Microglia in Culture are Modified by Acidosis: Implications for Alzheimer's Disease.

artículo científico publicado en 2016

Glial Cells and Integrity of the Nervous System

artículo científico publicado en 2016

Immobilized amyloid precursor protein constructs: a tool for the in vitro screening of glial cell reactivity

scientific article published on 01 September 2001

Immunotherapy in Alzheimer's disease: where do we stand? Where should we go?

artículo científico publicado en 2010

Impact of Aging in Microglia-Mediated D-Serine Balance in the CNS

artículo científico publicado en 2018

Is the supply of axoplasmic proteins a burden for the cell body? Morphometry of sensory neurons and amino acid incorporation into their cell bodies.

artículo científico publicado en 1989

Microglia Function in the Normal Brain.

artículo científico publicado en 2016

Microglial cell dysregulation in brain aging and neurodegeneration

artículo científico publicado en 2015

Microglial reactivity to beta-amyloid is modulated by astrocytes and proinflammatory factors

scientific article published on 01 October 2004

Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease.

artículo científico publicado en 2013

Modulation by astrocytes of microglial cell-mediated neuroinflammation: effect on the activation of microglial signaling pathways

artículo científico publicado en 2007

Modulation of interferon-γ-induced glial cell activation by transforming growth factor β1: a role for STAT1 and MAPK pathways.

artículo científico publicado en 2012

Oligodendrocytes damage in Alzheimer's disease: beta amyloid toxicity and inflammation.

artículo científico publicado en 2005

Phosphorylated tau potentiates Aβ-induced mitochondrial damage in mature neurons.

artículo científico publicado en 2014

Prenatal exposure to inflammatory conditions increases Cx43 and Panx1 unopposed channel opening and activation of astrocytes in the offspring effect on neuronal survival

artículo científico publicado en 2015

Prenatal to early postnatal nicotine exposure impairs central chemoreception and modifies breathing pattern in mouse neonates: a probable link to sudden infant death syndrome.

artículo científico publicado en 2008

Proliferating culture of aged microglia for the study of neurodegenerative diseases

scientific article published on 25 August 2011

Protection of rat primary hippocampal cultures from A beta cytotoxicity by pro-inflammatory molecules is mediated by astrocytes.

artículo científico publicado en 2005

Reduction of Blood Amyloid-β Oligomers in Alzheimer's Disease Transgenic Mice by c-Abl Kinase Inhibition

artículo científico publicado en 2016

Reduction of beta-amyloid-induced neurotoxicity on hippocampal cell cultures by moderate acidosis is mediated by transforming growth factor beta.

artículo científico publicado en 2008

Role of TGFβ signaling in the pathogenesis of Alzheimer's disease.

artículo científico publicado en 2015

Role of scavenger receptors in glia-mediated neuroinflammatory response associated with Alzheimer's disease.

artículo científico publicado en 2013

SR-A regulates the inflammatory activation of astrocytes

artículo científico publicado en 2013

Scavenger Receptor-A deficiency impairs immune response of microglia and astrocytes potentiating Alzheimer's disease pathophysiology.

artículo científico publicado en 2017

Thiazolidinediones promote axonal growth through the activation of the JNK pathway

artículo científico publicado en 2013

Transforming growth factor β1 modulates amyloid β-induced glial activation through the Smad3-dependent induction of MAPK phosphatase-1

artículo científico publicado en 2012

Transforming growth factor-beta 1 produced by hippocampal cells modulates microglial reactivity in culture.

artículo científico publicado en 2005

Understanding risk factors for Alzheimer's disease: interplay of neuroinflammation, connexin-based communication and oxidative stress.

artículo científico publicado en 2012

What Is Neural Plasticity?

artículo científico publicado en 2017

[Biological mechanisms involved in the spread of traumatic brain damage]

scientific article published on 07 September 2011

c-Abl tyrosine kinase modulates tau pathology and Cdk5 phosphorylation in AD transgenic mice

scientific journal article

Lista de obras de Rommy von Bernhardi

ATP is required and advances cytokine-induced gap junction formation in microglia in vitro.

Acetylcholinesterase induces the expression of the beta-amyloid precursor protein in glia and activates glial cells in culture.

Age-Dependent Changes in the Activation and Regulation of Microglia.

Age-dependent changes on TGFβ1 Smad3 pathway modify the pattern of microglial cell activation

Alzheimer's disease: redox dysregulation as a common denominator for diverse pathogenic mechanisms.

Astrocytes inhibit nitric oxide-dependent Ca(2+) dynamics in activated microglia: involvement of ATP released via pannexin 1 channels.

Aβ potentiates inflammatory activation of glial cells induced by scavenger receptor ligands and inflammatory mediators in culture.

Commentaries on Viewpoint: The ongoing need for good physiological investigation: Obstructive sleep apnea in HIV patients as a paradigm

Connexin43 hemichannels mediate secondary cellular damage spread from the trauma zone to distal zones in astrocyte monolayers.

D-serine released by astrocytes in brainstem regulates breathing response to CO2 levels

Expression Pattern of Scavenger Receptors and Amyloid-β Phagocytosis of Astrocytes and Microglia in Culture are Modified by Acidosis: Implications for Alzheimer's Disease.

Glial Cells and Integrity of the Nervous System

Immobilized amyloid precursor protein constructs: a tool for the in vitro screening of glial cell reactivity

Immunotherapy in Alzheimer's disease: where do we stand? Where should we go?

Impact of Aging in Microglia-Mediated D-Serine Balance in the CNS

Is the supply of axoplasmic proteins a burden for the cell body? Morphometry of sensory neurons and amino acid incorporation into their cell bodies.

Microglia Function in the Normal Brain.

Microglial cell dysregulation in brain aging and neurodegeneration

Microglial reactivity to beta-amyloid is modulated by astrocytes and proinflammatory factors

Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease.

Modulation by astrocytes of microglial cell-mediated neuroinflammation: effect on the activation of microglial signaling pathways

Modulation of interferon-γ-induced glial cell activation by transforming growth factor β1: a role for STAT1 and MAPK pathways.

Oligodendrocytes damage in Alzheimer's disease: beta amyloid toxicity and inflammation.

Phosphorylated tau potentiates Aβ-induced mitochondrial damage in mature neurons.

Prenatal exposure to inflammatory conditions increases Cx43 and Panx1 unopposed channel opening and activation of astrocytes in the offspring effect on neuronal survival

Prenatal to early postnatal nicotine exposure impairs central chemoreception and modifies breathing pattern in mouse neonates: a probable link to sudden infant death syndrome.

Proliferating culture of aged microglia for the study of neurodegenerative diseases

Protection of rat primary hippocampal cultures from A beta cytotoxicity by pro-inflammatory molecules is mediated by astrocytes.

Reduction of Blood Amyloid-β Oligomers in Alzheimer's Disease Transgenic Mice by c-Abl Kinase Inhibition

Reduction of beta-amyloid-induced neurotoxicity on hippocampal cell cultures by moderate acidosis is mediated by transforming growth factor beta.

Role of TGFβ signaling in the pathogenesis of Alzheimer's disease.

Role of scavenger receptors in glia-mediated neuroinflammatory response associated with Alzheimer's disease.

SR-A regulates the inflammatory activation of astrocytes

Scavenger Receptor-A deficiency impairs immune response of microglia and astrocytes potentiating Alzheimer's disease pathophysiology.

Thiazolidinediones promote axonal growth through the activation of the JNK pathway

Transforming growth factor β1 modulates amyloid β-induced glial activation through the Smad3-dependent induction of MAPK phosphatase-1

Transforming growth factor-beta 1 produced by hippocampal cells modulates microglial reactivity in culture.

Understanding risk factors for Alzheimer's disease: interplay of neuroinflammation, connexin-based communication and oxidative stress.

What Is Neural Plasticity?

[Biological mechanisms involved in the spread of traumatic brain damage]

c-Abl tyrosine kinase modulates tau pathology and Cdk5 phosphorylation in AD transgenic mice