Lista de obras - James W Crane - Dominio Público Uruguay

5-HT1A receptors mediate (+)8-OH-DPAT-stimulation of extracellular signal-regulated kinase (MAP kinase) in vivo in rat hypothalamus: time dependence and regional differences.

artículo científico publicado en 2007

Auditory Tones and Foot-Shock Recapitulate Spontaneous Sub-Threshold Activity in Basolateral Amygdala Principal Neurons and Interneurons.

artículo científico publicado en 2016

Baclofen acts in the central amygdala to reduce synaptic transmission and impair context fear conditioning.

artículo científico publicado en 2018

Chronic mild stress induces behavioral and physiological changes, and may alter serotonin 1A receptor function, in male and cycling female rats.

artículo científico publicado en 2004

Evidence that the bed nucleus of the stria terminalis contributes to the modulation of hypophysiotropic corticotropin-releasing factor cell responses to systemic interleukin-1beta.

artículo científico publicado en 2003

Inhibition dominates the early phase of up-states in the basolateral amygdala.

artículo científico publicado en 2010

Medial prefrontal cortex suppression of the hypothalamic–pituitary–adrenal axis response to a physical stressor, systemic delivery of interleukin‐1β ⬇️

artículo científico publicado el 1 de abril de 2003

Modulation of SK channel trafficking by beta adrenoceptors enhances excitatory synaptic transmission and plasticity in the amygdala.

artículo científico publicado en 2008

Noradrenaline modulates transmission at a central synapse by a presynaptic mechanism.

artículo científico publicado en 2007

Oscillations in the basolateral amygdala: aversive stimulation is state dependent and resets the oscillatory phase.

artículo científico publicado en 2009

Oxytocin receptor activation in the basolateral complex of the amygdala enhances discrimination between discrete cues and promotes configural processing of cues

artículo científico publicado en 2018

Patterns of neuronal activation in the rat brain and spinal cord in response to increasing durations of restraint stress.

artículo científico publicado en 2005

Presynaptic GABAB receptors reduce transmission at parabrachial synapses in the lateral central amygdala by inhibiting N-type calcium channels.

artículo científico publicado en 2016

Stressor categorization: acute physical and psychological stressors elicit distinctive recruitment patterns in the amygdala and in medullary noradrenergic cell groups.

artículo científico publicado en 2001

Systemic apomorphine alters HPA axis responses to interleukin-1 beta administration but not sound stress.

artículo científico publicado en 2003

Systemic blockade of complement C5a receptors reduces lipopolysacharride-induced responses in the paraventricular nucleus and the central amygdala

artículo científico publicado en 2007

The C5a anaphylatoxin receptor CD88 is expressed in presynaptic terminals of hippocampal mossy fibres.

artículo científico publicado en 2009

The central nucleus of the amygdala; a conduit for modulation of HPA axis responses to an immune challenge?

artículo científico

The complement factor C5a contributes to pathology in a rat model of amyotrophic lateral sclerosis

scientific article published on 01 December 2008

Therapeutic activity of C5a receptor antagonists in a rat model of neurodegeneration

artículo científico publicado en 2006

α2-adrenoceptor-mediated inhibition in the central amygdala blocks fear-conditioning.

artículo científico publicado en 2017

Lista de obras de James W Crane

5-HT1A receptors mediate (+)8-OH-DPAT-stimulation of extracellular signal-regulated kinase (MAP kinase) in vivo in rat hypothalamus: time dependence and regional differences.

Auditory Tones and Foot-Shock Recapitulate Spontaneous Sub-Threshold Activity in Basolateral Amygdala Principal Neurons and Interneurons.

Baclofen acts in the central amygdala to reduce synaptic transmission and impair context fear conditioning.

Chronic mild stress induces behavioral and physiological changes, and may alter serotonin 1A receptor function, in male and cycling female rats.

Evidence that the bed nucleus of the stria terminalis contributes to the modulation of hypophysiotropic corticotropin-releasing factor cell responses to systemic interleukin-1beta.

Inhibition dominates the early phase of up-states in the basolateral amygdala.

Medial prefrontal cortex suppression of the hypothalamic–pituitary–adrenal axis response to a physical stressor, systemic delivery of interleukin‐1β ⬇️

Modulation of SK channel trafficking by beta adrenoceptors enhances excitatory synaptic transmission and plasticity in the amygdala.

Noradrenaline modulates transmission at a central synapse by a presynaptic mechanism.

Oscillations in the basolateral amygdala: aversive stimulation is state dependent and resets the oscillatory phase.

Oxytocin receptor activation in the basolateral complex of the amygdala enhances discrimination between discrete cues and promotes configural processing of cues

Patterns of neuronal activation in the rat brain and spinal cord in response to increasing durations of restraint stress.

Presynaptic GABAB receptors reduce transmission at parabrachial synapses in the lateral central amygdala by inhibiting N-type calcium channels.

Stressor categorization: acute physical and psychological stressors elicit distinctive recruitment patterns in the amygdala and in medullary noradrenergic cell groups.

Systemic apomorphine alters HPA axis responses to interleukin-1 beta administration but not sound stress.

Systemic blockade of complement C5a receptors reduces lipopolysacharride-induced responses in the paraventricular nucleus and the central amygdala

The C5a anaphylatoxin receptor CD88 is expressed in presynaptic terminals of hippocampal mossy fibres.

The central nucleus of the amygdala; a conduit for modulation of HPA axis responses to an immune challenge?

The complement factor C5a contributes to pathology in a rat model of amyotrophic lateral sclerosis

Therapeutic activity of C5a receptor antagonists in a rat model of neurodegeneration

α2-adrenoceptor-mediated inhibition in the central amygdala blocks fear-conditioning.