Lista de obras -

A2A adenosine receptor deletion is protective in a mouse model of Tauopathy

artículo científico publicado en 2014

Abnormal Tau phosphorylation of the Alzheimer-type also occurs during mitosis.

artículo científico publicado en 2002

Activation of p65 NF-κB protein by p210BCR – ABL in a myeloid cell line (P210BCR – ABL activates p65 NF-κB) ⬇️

artículo científico publicado el 6 de noviembre de 1997

Alzheimer's disease-like tau neuropathology leads to memory deficits and loss of functional synapses in a novel mutated tau transgenic mouse without any motor deficits

artículo científico publicado en 2006

Beneficial effects of caffeine in a transgenic model of Alzheimer's disease-like tau pathology

artículo científico publicado en 2014

Beneficial effects of exercise in a transgenic mouse model of Alzheimer's disease-like Tau pathology.

artículo científico publicado en 2011

Biochemistry of Tau in Alzheimer's disease and related neurological disorders.

artículo científico publicado en 2008

Cellular contaminants of adeno-associated virus vector stocks can enhance transduction.

artículo científico publicado en 1999

Chloroquine and chloroquinoline derivatives as models for the design of modulators of amyloid Peptide precursor metabolism.

artículo científico publicado en 2015

Consensus brain-derived protein, extraction protocol for the study of human and murine brain proteome using both 2D-DIGE and mini 2DE immunoblotting. ⬇️

artículo científico publicado en 2014

Determination of the human c-Abl consensus DNA binding site

artículo científico publicado en 1998

Detrimental effects of diet-induced obesity on τ pathology are independent of insulin resistance in τ transgenic mice.

artículo científico publicado en 2012

Early Tau pathology involving the septo-hippocampal pathway in a Tau transgenic model: relevance to Alzheimer's disease.

artículo científico publicado en 2009

Erratum: A2A adenosine receptor deletion is protective in a mouse model of Tauopathy

article

From epidemiology to pathophysiology: what about caffeine in Alzheimer's disease?

artículo científico publicado en 2014

From tau phosphorylation to tau aggregation: what about neuronal death?

artículo científico publicado en 2010

Functional characterization of FTDP-17 tau gene mutations through their effects on Xenopus oocyte maturation.

artículo científico publicado en 2001

Highly successful therapeutic vaccinations combining dendritic cells and tumor cells secreting granulocyte macrophage colony-stimulating factor.

artículo científico publicado en 2004

Hippocampal BDNF expression in a tau transgenic mouse model.

artículo científico publicado en 2012

Imidazole-derived 2-[N-carbamoylmethyl-alkylamino]acetic acids, substrate-dependent modulators of insulin-degrading enzyme in amyloid-β hydrolysis

artículo científico publicado en 2014

Increased expression of BIN1 mediates Alzheimer genetic risk by modulating tau pathology

artículo científico publicado en 2013

Intracellular pH regulates amyloid precursor protein intracellular domain accumulation.

artículo científico publicado en 2007

Loss of medial septum cholinergic neurons in THY-Tau22 mouse model: what links with tau pathology?

artículo científico publicado en 2011

MUC1-C nuclear localization drives invasiveness of renal cancer cells through a sheddase/gamma secretase dependent pathway.

artículo científico publicado en 2014

Modelling Alzheimer-specific abnormal Tau phosphorylation independently of GSK3beta and PKA kinase activities.

artículo científico publicado en 2002

Molecular actors in Alzheimer's disease: which diagnostic and therapeutic consequences?

artículo científico publicado en 2010

NMDA receptor dysfunction contributes to impaired brain-derived neurotrophic factor-induced facilitation of hippocampal synaptic transmission in a Tau transgenic model.

artículo científico publicado en 2012

NMR spectroscopy of the neuronal tau protein: normal function and implication in Alzheimer's disease

artículo científico publicado en 2010

Neurofibrillary degeneration of the Alzheimer-type: an alternate pathway to neuronal apoptosis?

artículo científico publicado en 2003

PUFA induce antidepressant-like effects in parallel to structural and molecular changes in the hippocampus.

artículo científico publicado en 2008

Pathological tau phenotypes. The weight of mutations, polymorphisms, and differential neuronal vulnerabilities.

artículo científico publicado en 2000

Pin1 allows for differential Tau dephosphorylation in neuronal cells

artículo científico publicado en 2006

Pin1: a therapeutic target in Alzheimer neurodegeneration

artículo científico publicado en 2002

Progressive age-related cognitive decline in tau mice.

artículo científico publicado en 2013

Proteasome inhibition and Tau proteolysis: an unexpected regulation.

artículo científico publicado en 2005

Role and expression of CD40 on human retinal pigment epithelial cells

artículo científico publicado en 2000

Role of the Tau N-terminal region in microtubule stabilization revealed by new endogenous truncated forms

artículo científico publicado en 2015

Stable-tau overexpression in human neuroblastoma cells: an open door for explaining neuronal death in tauopathies.

artículo científico publicado en 2003

Tau deletion promotes brain insulin resistance

artículo científico publicado en 2017

Tau pathology modulates Pin1 post-translational modifications and may be relevant as biomarker

artículo científico publicado en 2012

The DNA Binding Domain of the Human c-Abl Tyrosine Kinase Preferentially Binds to DNA Sequences Containing an AAC Motif and to Distorted DNA Structures ⬇️

artículo científico publicado el 28 de abril de 1998

The FK506-binding protein FKBP52 in vitro induces aggregation of truncated Tau forms with prion-like behavior

artículo científico publicado en 2015

The caffeine-binding adenosine A2A receptor induces age-like HPA-axis dysfunction by targeting glucocorticoid receptor function.

artículo científico publicado en 2016

The complex p25/Cdk5 kinase in neurofibrillary degeneration and neuronal death: the missing link to cell cycle.

artículo científico publicado en 2007

The peptidylprolyl cis/trans-isomerase Pin1 modulates stress-induced dephosphorylation of Tau in neurons. Implication in a pathological mechanism related to Alzheimer disease

scientific journal article

Two-dimensional electrophoresis of tau mutants reveals specific phosphorylation pattern likely linked to early tau conformational changes.

artículo científico publicado en 2009

Variation of endothelial nitric oxide synthase synthesis in the median eminence during the rat estrous cycle: an additional argument for the implication of vascular blood vessel in the control of GnRH release.

artículo científico publicado en 2001

[Tau story: from frontotemporal dementia to other tauopathies]

scientific article published on 01 January 2002

p25/Cdk5-mediated retinoblastoma phosphorylation is an early event in neuronal cell death.

artículo científico publicado en 2005

Lista de obras de

A2A adenosine receptor deletion is protective in a mouse model of Tauopathy

Abnormal Tau phosphorylation of the Alzheimer-type also occurs during mitosis.

Activation of p65 NF-κB protein by p210BCR – ABL in a myeloid cell line (P210BCR – ABL activates p65 NF-κB) ⬇️

Alzheimer's disease-like tau neuropathology leads to memory deficits and loss of functional synapses in a novel mutated tau transgenic mouse without any motor deficits

Beneficial effects of caffeine in a transgenic model of Alzheimer's disease-like tau pathology

Beneficial effects of exercise in a transgenic mouse model of Alzheimer's disease-like Tau pathology.

Biochemistry of Tau in Alzheimer's disease and related neurological disorders.

Cellular contaminants of adeno-associated virus vector stocks can enhance transduction.

Chloroquine and chloroquinoline derivatives as models for the design of modulators of amyloid Peptide precursor metabolism.

Consensus brain-derived protein, extraction protocol for the study of human and murine brain proteome using both 2D-DIGE and mini 2DE immunoblotting. ⬇️

Determination of the human c-Abl consensus DNA binding site

Detrimental effects of diet-induced obesity on τ pathology are independent of insulin resistance in τ transgenic mice.

Early Tau pathology involving the septo-hippocampal pathway in a Tau transgenic model: relevance to Alzheimer's disease.

Erratum: A2A adenosine receptor deletion is protective in a mouse model of Tauopathy

From epidemiology to pathophysiology: what about caffeine in Alzheimer's disease?

From tau phosphorylation to tau aggregation: what about neuronal death?

Functional characterization of FTDP-17 tau gene mutations through their effects on Xenopus oocyte maturation.

Highly successful therapeutic vaccinations combining dendritic cells and tumor cells secreting granulocyte macrophage colony-stimulating factor.

Hippocampal BDNF expression in a tau transgenic mouse model.

Imidazole-derived 2-[N-carbamoylmethyl-alkylamino]acetic acids, substrate-dependent modulators of insulin-degrading enzyme in amyloid-β hydrolysis

Increased expression of BIN1 mediates Alzheimer genetic risk by modulating tau pathology

Intracellular pH regulates amyloid precursor protein intracellular domain accumulation.

Loss of medial septum cholinergic neurons in THY-Tau22 mouse model: what links with tau pathology?

MUC1-C nuclear localization drives invasiveness of renal cancer cells through a sheddase/gamma secretase dependent pathway.

Modelling Alzheimer-specific abnormal Tau phosphorylation independently of GSK3beta and PKA kinase activities.

Molecular actors in Alzheimer's disease: which diagnostic and therapeutic consequences?

NMDA receptor dysfunction contributes to impaired brain-derived neurotrophic factor-induced facilitation of hippocampal synaptic transmission in a Tau transgenic model.

NMR spectroscopy of the neuronal tau protein: normal function and implication in Alzheimer's disease

Neurofibrillary degeneration of the Alzheimer-type: an alternate pathway to neuronal apoptosis?

PUFA induce antidepressant-like effects in parallel to structural and molecular changes in the hippocampus.

Pathological tau phenotypes. The weight of mutations, polymorphisms, and differential neuronal vulnerabilities.

Pin1 allows for differential Tau dephosphorylation in neuronal cells

Pin1: a therapeutic target in Alzheimer neurodegeneration

Progressive age-related cognitive decline in tau mice.

Proteasome inhibition and Tau proteolysis: an unexpected regulation.

Role and expression of CD40 on human retinal pigment epithelial cells

Role of the Tau N-terminal region in microtubule stabilization revealed by new endogenous truncated forms

Stable-tau overexpression in human neuroblastoma cells: an open door for explaining neuronal death in tauopathies.

Tau deletion promotes brain insulin resistance

Tau pathology modulates Pin1 post-translational modifications and may be relevant as biomarker

The DNA Binding Domain of the Human c-Abl Tyrosine Kinase Preferentially Binds to DNA Sequences Containing an AAC Motif and to Distorted DNA Structures ⬇️

The FK506-binding protein FKBP52 in vitro induces aggregation of truncated Tau forms with prion-like behavior

The caffeine-binding adenosine A2A receptor induces age-like HPA-axis dysfunction by targeting glucocorticoid receptor function.

The complex p25/Cdk5 kinase in neurofibrillary degeneration and neuronal death: the missing link to cell cycle.

The peptidylprolyl cis/trans-isomerase Pin1 modulates stress-induced dephosphorylation of Tau in neurons. Implication in a pathological mechanism related to Alzheimer disease

Two-dimensional electrophoresis of tau mutants reveals specific phosphorylation pattern likely linked to early tau conformational changes.

Variation of endothelial nitric oxide synthase synthesis in the median eminence during the rat estrous cycle: an additional argument for the implication of vascular blood vessel in the control of GnRH release.

[Tau story: from frontotemporal dementia to other tauopathies]

p25/Cdk5-mediated retinoblastoma phosphorylation is an early event in neuronal cell death.