Lista de obras - Paolo Catarsi - Dominio Público Uruguay

Altered fibronectin expression and deposition by myeloproliferative neoplasm-derived mesenchymal stromal cells

artículo científico publicado en 2015

Angiotensin-converting enzyme (ACE) haplotypes and cyclosporine A (CsA) response: a model of the complex relationship between ACE quantitative trait locus and pathological phenotypes

artículo científico publicado en 2005

Apolipoprotein E in idiopathic nephrotic syndrome and focal segmental glomerulosclerosis.

artículo científico publicado en 2003

Arachidonic acid-evoked Ca2+ signals promote nitric oxide release and proliferation in human endothelial colony forming cells

scientific article published on 12 September 2016

Conjugated polymers optically regulate the fate of endothelial colony-forming cells

artículo científico publicado en 2019

Constitutive STAT5 phosphorylation in CD34+ cells of patients with primary myelofibrosis: Correlation with driver mutation status and disease severity

artículo científico publicado en 2019

Cyclosporin and organ specific toxicity: clinical aspects, pharmacogenetics and perspectives

artículo científico publicado en 2008

Cyclosporine in patients with steroid-resistant nephrotic syndrome: an open-label, nonrandomized, retrospective study

artículo científico publicado en 2004

Evidence that prefibrotic myelofibrosis is aligned along a clinical and biological continuum featuring primary myelofibrosis

artículo científico publicado en 2012

Functional and genetic aberrations of in vitro-cultured marrow-derived mesenchymal stromal cells of patients with classical Philadelphia-negative myeloproliferative neoplasms

artículo científico publicado en 2014

Impact of erythropoietin on the effects of irradiation under hypoxia.

artículo científico publicado en 2009

Increased plasma nicotinamide phosphoribosyltransferase is associated with a hyperproliferative phenotype and restrains disease progression in MPN-associated myelofibrosis.

artículo científico publicado en 2016

Involvement of TGFβ1 in autocrine regulation of proplatelet formation in healthy subjects and patients with primary myelofibrosis

artículo científico publicado en 2013

JAK2 V617F genotype is a strong determinant of blast transformation in primary myelofibrosis

artículo científico publicado en 2013

JAK2 exon 14 skipping in patients with primary myelofibrosis: a minor splice variant modulated by the JAK2-V617F allele burden

artículo científico publicado en 2015

JAK2(V617F) allele burden ⩾50% is associated with response to ruxolitinib in persons with MPN-associated myelofibrosis and splenomegaly requiring therapy.

artículo científico publicado en 2016

Kinetic and Angiogenic Activity of Circulating Endothelial Colony Forming Cells in Patients with Infantile Haemangioma Receiving Propranolol

artículo científico publicado en 2019

No association between the XPD Lys751Gln (rs13181) polymorphism and disease phenotype or leukemic transformation in primary myelofibrosis.

artículo científico publicado en 2013

Polymorphisms in the osteopontin promoter affect its transcriptional activity.

artículo científico publicado en 2004

Primary myelofibrosis: Older age and high JAK2V617F allele burden are associated with elevated plasma high-sensitivity C-reactive protein levels and a phenotype of progressive disease.

artículo científico publicado en 2017

Reduced CXCR4-expression on CD34-positive blood cells predicts outcomes of persons with primary myelofibrosis

artículo científico publicado en 2020

Reduced frequency of circulating CD4+CD25brightCD127lowFOXP3+ regulatory T cells in primary myelofibrosis

artículo científico publicado en 2016

Spleen endothelial cells from patients with myelofibrosis harbor the JAK2V617F mutation.

artículo científico publicado en 2012

The spleen of patients with myelofibrosis harbors defective mesenchymal stromal cells

artículo científico publicado en 2018

Tie2 Expressing Monocytes in the Spleen of Patients with Primary Myelofibrosis

artículo científico publicado en 2016

Lista de obras de Paolo Catarsi

Altered fibronectin expression and deposition by myeloproliferative neoplasm-derived mesenchymal stromal cells

Angiotensin-converting enzyme (ACE) haplotypes and cyclosporine A (CsA) response: a model of the complex relationship between ACE quantitative trait locus and pathological phenotypes

Apolipoprotein E in idiopathic nephrotic syndrome and focal segmental glomerulosclerosis.

Arachidonic acid-evoked Ca2+ signals promote nitric oxide release and proliferation in human endothelial colony forming cells

Conjugated polymers optically regulate the fate of endothelial colony-forming cells

Constitutive STAT5 phosphorylation in CD34+ cells of patients with primary myelofibrosis: Correlation with driver mutation status and disease severity

Cyclosporin and organ specific toxicity: clinical aspects, pharmacogenetics and perspectives

Cyclosporine in patients with steroid-resistant nephrotic syndrome: an open-label, nonrandomized, retrospective study

Evidence that prefibrotic myelofibrosis is aligned along a clinical and biological continuum featuring primary myelofibrosis

Functional and genetic aberrations of in vitro-cultured marrow-derived mesenchymal stromal cells of patients with classical Philadelphia-negative myeloproliferative neoplasms

Impact of erythropoietin on the effects of irradiation under hypoxia.

Increased plasma nicotinamide phosphoribosyltransferase is associated with a hyperproliferative phenotype and restrains disease progression in MPN-associated myelofibrosis.

Involvement of TGFβ1 in autocrine regulation of proplatelet formation in healthy subjects and patients with primary myelofibrosis

JAK2 V617F genotype is a strong determinant of blast transformation in primary myelofibrosis

JAK2 exon 14 skipping in patients with primary myelofibrosis: a minor splice variant modulated by the JAK2-V617F allele burden

JAK2(V617F) allele burden ⩾50% is associated with response to ruxolitinib in persons with MPN-associated myelofibrosis and splenomegaly requiring therapy.

Kinetic and Angiogenic Activity of Circulating Endothelial Colony Forming Cells in Patients with Infantile Haemangioma Receiving Propranolol

No association between the XPD Lys751Gln (rs13181) polymorphism and disease phenotype or leukemic transformation in primary myelofibrosis.

Polymorphisms in the osteopontin promoter affect its transcriptional activity.

Primary myelofibrosis: Older age and high JAK2V617F allele burden are associated with elevated plasma high-sensitivity C-reactive protein levels and a phenotype of progressive disease.

Reduced CXCR4-expression on CD34-positive blood cells predicts outcomes of persons with primary myelofibrosis

Reduced frequency of circulating CD4+CD25brightCD127lowFOXP3+ regulatory T cells in primary myelofibrosis

Spleen endothelial cells from patients with myelofibrosis harbor the JAK2V617F mutation.

The spleen of patients with myelofibrosis harbors defective mesenchymal stromal cells

Tie2 Expressing Monocytes in the Spleen of Patients with Primary Myelofibrosis