Lista de obras - Monika C Brunner-Weinzierl

A new role of CTLA-4 on B cells in thymus-dependent immune responses in vivo

artículo científico publicado en 2007

Activated protein C protects from GvHD via PAR2/PAR3 signalling in regulatory T-cells

artículo científico publicado en 2017

Acute psychological stress alerts the adaptive immune response: stress-induced mobilization of effector T cells.

artículo científico publicado en 2006

Acute psychological stress increases peripheral blood CD3+CD56+ natural killer T cells in healthy men: possible implications for the development and treatment of allergic and autoimmune disorders.

artículo científico publicado en 2013

Acute psychological stress induces a prolonged suppression of the production of reactive oxygen species by phagocytes.

artículo científico publicado en 2003

Acute psychological stress simultaneously alters hormone levels, recruitment of lymphocyte subsets, and production of reactive oxygen species.

artículo científico publicado en 2002

Altered Th1/Th2 balance associated with the immunosuppressive/protective effect of the H-2Ab allele on the response to allo-4-hydroxyphenylpyruvate dioxygenase.

artículo científico publicado en 1995

Astrocytes protect the CNS: antigen-specific T helper cell responses are inhibited by astrocyte-induced upregulation of CTLA-4 (CD152).

artículo científico publicado en 2004

Autoimmune pancreatitis in MRL/Mp mice is a T cell-mediated disease responsive to cyclosporine A and rapamycin treatment

article

Blockade of CTLA-4 decreases the generation of multifunctional memory CD4+ T cells in vivo

artículo científico publicado en 2011

CD152 (CTLA-4) determines CD4 T cell migration in vitro and in vivo

artículo científico publicado en 2009

CD152 (CTLA-4) determines the unequal resistance of Th1 and Th2 cells against activation-induced cell death by a mechanism requiring PI3 kinase function.

artículo científico

CD152 (CTLA-4) regulates effector functions of CD8+ T lymphocytes by repressing Eomesodermin.

artículo científico publicado en 2009

CD25 regulatory T cells determine secondary but not primary remission in EAE: impact on long-term disease progression.

artículo científico publicado en 2005

CD4+ T cells from human neonates and infants are poised spontaneously to run a nonclassical IL-4 program

artículo científico publicado en 2014

CTLA-4 (CD152) blockade does not cause a pro-inflammatory cytokine profile in regulatory T cells

artículo científico publicado en 2011

CTLA-4 (CD152) enhances the Tc17 differentiation program

scientific article published on 30 May 2014

CTLA-4 (CD152) inhibits T cell function by activating the ubiquitin ligase Itch.

artículo científico publicado en 2010

CTLA-4 (CD152): A versatile receptor for immune-based therapy

scientific article published on 01 April 2019

CTLA-4 and PD-1 Control of T-Cell Motility and Migration: Implications for Tumor Immunotherapy

scientific article published on 27 November 2018

CTLA-4 is expressed by activated mouse NK cells and inhibits NK Cell IFN-γ production in response to mature dendritic cells.

artículo científico publicado en 2014

CTLA-4-competent conventional T cells back up regulatory T cells to restrain memory T-helper type 2 cell responses

artículo científico publicado en 2019

CTLA4 blockade elicits paraneoplastic neurological disease in a mouse model.

artículo científico publicado en 2016

Clinically relevant doses of FLT3-kinase inhibitors quizartinib and midostaurin do not impair T-cell reactivity and function.

artículo científico publicado en 2014

Decrease of CD4(+)FOXP3(+) T regulatory cells in the peripheral blood of human subjects undergoing a mental stressor.

artículo científico publicado en 2009

Developmental induction of human T-cell responses against Candida albicans and Aspergillus fumigatus

scientific article published in Scientific Reports

Diversity of clonal T cell proliferation is mediated by differential expression of CD152 (CTLA-4) on the cell surface of activated individual T lymphocytes.

artículo científico publicado en 2003

Early changes in the metabolic profile of activated CD8(+) T cells

artículo científico publicado en 2016

Female-biased incidence of experimental autoimmune encephalomyelitis reflects sexually dimorphic expression of surface CTLA-4 (CD152) on T lymphocytes.

artículo científico publicado en 2010

Glycogen synthase kinase-3β is a crucial mediator of signal-induced RelB degradation ⬇️

artículo científico publicado el 10 de enero de 2011

IFN-γ is a therapeutic target in paraneoplastic cerebellar degeneration

artículo científico publicado en 2019

IL-1β and TGF-β act antagonistically in induction and differentially in propagation of human proinflammatory precursor CD4+ T cells.

artículo científico publicado en 2011

Immune privilege as an intrinsic CNS property: astrocytes protect the CNS against T-cell-mediated neuroinflammation

artículo científico publicado en 2013

Impact of the CTLA-4/CD28 axis on the processes of joint inflammation in rheumatoid arthritis.

artículo científico publicado en 2013

Migration of Th1 lymphocytes is regulated by CD152 (CTLA-4)-mediated signaling via PI3 kinase-dependent Akt activation

artículo científico publicado en 2012

Surface CD152 (CTLA-4) expression and signaling dictates longevity of CD28null T cells

artículo científico publicado en 2009

T Cells of Infants Are Mature, but Hyporeactive Due to Limited Ca2+ Influx

artículo científico publicado en 2016

The differentiation and plasticity of Tc17 cells are regulated by CTLA-4-mediated effects on STATs

artículo científico publicado en 2017

The integrative activity of the immune system

scientific article published on 01 December 1994

The tyrosine phosphatase SHP-2 regulates differentiation and apoptosis of individual primary T lymphocytes.

artículo científico publicado en 2007

Lista de obras de Monika C Brunner-Weinzierl

A new role of CTLA-4 on B cells in thymus-dependent immune responses in vivo

Activated protein C protects from GvHD via PAR2/PAR3 signalling in regulatory T-cells

Acute psychological stress alerts the adaptive immune response: stress-induced mobilization of effector T cells.

Acute psychological stress increases peripheral blood CD3+CD56+ natural killer T cells in healthy men: possible implications for the development and treatment of allergic and autoimmune disorders.

Acute psychological stress induces a prolonged suppression of the production of reactive oxygen species by phagocytes.

Acute psychological stress simultaneously alters hormone levels, recruitment of lymphocyte subsets, and production of reactive oxygen species.

Altered Th1/Th2 balance associated with the immunosuppressive/protective effect of the H-2Ab allele on the response to allo-4-hydroxyphenylpyruvate dioxygenase.

Astrocytes protect the CNS: antigen-specific T helper cell responses are inhibited by astrocyte-induced upregulation of CTLA-4 (CD152).

Autoimmune pancreatitis in MRL/Mp mice is a T cell-mediated disease responsive to cyclosporine A and rapamycin treatment

Blockade of CTLA-4 decreases the generation of multifunctional memory CD4+ T cells in vivo

CD152 (CTLA-4) determines CD4 T cell migration in vitro and in vivo

CD152 (CTLA-4) determines the unequal resistance of Th1 and Th2 cells against activation-induced cell death by a mechanism requiring PI3 kinase function.

CD152 (CTLA-4) regulates effector functions of CD8+ T lymphocytes by repressing Eomesodermin.

CD25 regulatory T cells determine secondary but not primary remission in EAE: impact on long-term disease progression.

CD4+ T cells from human neonates and infants are poised spontaneously to run a nonclassical IL-4 program

CTLA-4 (CD152) blockade does not cause a pro-inflammatory cytokine profile in regulatory T cells

CTLA-4 (CD152) enhances the Tc17 differentiation program

CTLA-4 (CD152) inhibits T cell function by activating the ubiquitin ligase Itch.

CTLA-4 (CD152): A versatile receptor for immune-based therapy

CTLA-4 and PD-1 Control of T-Cell Motility and Migration: Implications for Tumor Immunotherapy

CTLA-4 is expressed by activated mouse NK cells and inhibits NK Cell IFN-γ production in response to mature dendritic cells.

CTLA-4-competent conventional T cells back up regulatory T cells to restrain memory T-helper type 2 cell responses

CTLA4 blockade elicits paraneoplastic neurological disease in a mouse model.

Clinically relevant doses of FLT3-kinase inhibitors quizartinib and midostaurin do not impair T-cell reactivity and function.

Decrease of CD4(+)FOXP3(+) T regulatory cells in the peripheral blood of human subjects undergoing a mental stressor.

Developmental induction of human T-cell responses against Candida albicans and Aspergillus fumigatus

Diversity of clonal T cell proliferation is mediated by differential expression of CD152 (CTLA-4) on the cell surface of activated individual T lymphocytes.

Early changes in the metabolic profile of activated CD8(+) T cells

Female-biased incidence of experimental autoimmune encephalomyelitis reflects sexually dimorphic expression of surface CTLA-4 (CD152) on T lymphocytes.

Glycogen synthase kinase-3β is a crucial mediator of signal-induced RelB degradation ⬇️

IFN-γ is a therapeutic target in paraneoplastic cerebellar degeneration

IL-1β and TGF-β act antagonistically in induction and differentially in propagation of human proinflammatory precursor CD4+ T cells.

Immune privilege as an intrinsic CNS property: astrocytes protect the CNS against T-cell-mediated neuroinflammation

Impact of the CTLA-4/CD28 axis on the processes of joint inflammation in rheumatoid arthritis.

Migration of Th1 lymphocytes is regulated by CD152 (CTLA-4)-mediated signaling via PI3 kinase-dependent Akt activation

Surface CD152 (CTLA-4) expression and signaling dictates longevity of CD28null T cells

T Cells of Infants Are Mature, but Hyporeactive Due to Limited Ca2+ Influx

The differentiation and plasticity of Tc17 cells are regulated by CTLA-4-mediated effects on STATs

The integrative activity of the immune system

The tyrosine phosphatase SHP-2 regulates differentiation and apoptosis of individual primary T lymphocytes.