Lista de obras - Emma L Scotter - Dominio Público Uruguay

ALS mutant FUS disrupts nuclear localization and sequesters wild-type FUS within cytoplasmic stress granules

artículo científico publicado en 2013

ALS/FTD mutations in UBQLN2 impede autophagy by reducing autophagosome acidification through loss of function

artículo científico publicado en 2020

Allele-specific knockdown of ALS-associated mutant TDP-43 in neural stem cells derived from induced pluripotent stem cells

artículo científico publicado en 2014

An anti-inflammatory role for C/EBPδ in human brain pericytes

artículo científico publicado en 2015

Antisense oligonucleotide therapies for Amyotrophic Lateral Sclerosis: Existing and emerging targets

artículo científico publicado en 2019

C9ORF72 and UBQLN2 mutations are causes of amyotrophic lateral sclerosis in New Zealand: a genetic and pathologic study using banked human brain tissue.

artículo científico publicado en 2016

Cardiac glycosides target barrier inflammation of the vasculature, meninges and choroid plexus

artículo científico publicado en 2021

Concurrent stimulation of cannabinoid CB1 and dopamine D2 receptors enhances heterodimer formation: a mechanism for receptor cross-talk?

artículo científico publicado en 2005

Differential roles of the ubiquitin proteasome system and autophagy in the clearance of soluble and aggregated TDP-43 species

artículo científico publicado en 2014

Exome-wide rare variant analysis identifies TUBA4A mutations associated with familial ALS.

artículo científico publicado en 2014

Hexanucleotide repeats in ALS/FTD form length-dependent RNA foci, sequester RNA binding proteins, and are neurotoxic

artículo científico publicado en 2013

High throughput quantification of mutant huntingtin aggregates

artículo científico publicado en 2008

Induction of Krox-24 by endogenous cannabinoid type 1 receptors in Neuro2A cells is mediated by the MEK-ERK MAPK pathway and is suppressed by the phosphatidylinositol 3-kinase pathway.

artículo científico publicado en 2006

Markers for human brain pericytes and smooth muscle cells

artículo científico publicado en 2018

Motor neurone disease: bringing New Zealand patients onto the world stage

artículo científico publicado en 2015

Mutations in the vesicular trafficking protein annexin A11 are associated with amyotrophic lateral sclerosis

artículo científico publicado en 2017

Neuromuscular disease: new insights and avenues for therapy.

artículo científico

Neuroprotective potential of CB1 receptor agonists in an in vitro model of Huntington's disease.

artículo científico publicado en 2010

Novel mutations support a role for Profilin 1 in the pathogenesis of ALS.

artículo científico publicado en 2014

Single-cell image analysis reveals a protective role for microglia in glioblastoma

artículo científico publicado en 2021

Specific detection of CB1 receptors; cannabinoid CB1 receptor antibodies are not all created equal!

artículo científico publicado en 2008

TDP-43 Proteinopathy and ALS: Insights into Disease Mechanisms and Therapeutic Targets

artículo científico publicado en 2015

The endocannabinoid system as a target for the treatment of neurodegenerative disease

artículo científico publicado en 2010

The therapeutic potential of G-protein coupled receptors in Huntington's disease

artículo científico publicado en 2010

Wild type human TDP-43 potentiates ALS-linked mutant TDP-43 driven progressive motor and cortical neuron degeneration with pathological features of ALS.

artículo científico publicado en 2015

Lista de obras de Emma L Scotter

ALS mutant FUS disrupts nuclear localization and sequesters wild-type FUS within cytoplasmic stress granules

ALS/FTD mutations in UBQLN2 impede autophagy by reducing autophagosome acidification through loss of function

Allele-specific knockdown of ALS-associated mutant TDP-43 in neural stem cells derived from induced pluripotent stem cells

An anti-inflammatory role for C/EBPδ in human brain pericytes

Antisense oligonucleotide therapies for Amyotrophic Lateral Sclerosis: Existing and emerging targets

C9ORF72 and UBQLN2 mutations are causes of amyotrophic lateral sclerosis in New Zealand: a genetic and pathologic study using banked human brain tissue.

Cardiac glycosides target barrier inflammation of the vasculature, meninges and choroid plexus

Concurrent stimulation of cannabinoid CB1 and dopamine D2 receptors enhances heterodimer formation: a mechanism for receptor cross-talk?

Differential roles of the ubiquitin proteasome system and autophagy in the clearance of soluble and aggregated TDP-43 species

Exome-wide rare variant analysis identifies TUBA4A mutations associated with familial ALS.

Hexanucleotide repeats in ALS/FTD form length-dependent RNA foci, sequester RNA binding proteins, and are neurotoxic

High throughput quantification of mutant huntingtin aggregates

Induction of Krox-24 by endogenous cannabinoid type 1 receptors in Neuro2A cells is mediated by the MEK-ERK MAPK pathway and is suppressed by the phosphatidylinositol 3-kinase pathway.

Markers for human brain pericytes and smooth muscle cells

Motor neurone disease: bringing New Zealand patients onto the world stage

Mutations in the vesicular trafficking protein annexin A11 are associated with amyotrophic lateral sclerosis

Neuromuscular disease: new insights and avenues for therapy.

Neuroprotective potential of CB1 receptor agonists in an in vitro model of Huntington's disease.

Novel mutations support a role for Profilin 1 in the pathogenesis of ALS.

Single-cell image analysis reveals a protective role for microglia in glioblastoma

Specific detection of CB1 receptors; cannabinoid CB1 receptor antibodies are not all created equal!

TDP-43 Proteinopathy and ALS: Insights into Disease Mechanisms and Therapeutic Targets

The endocannabinoid system as a target for the treatment of neurodegenerative disease

The therapeutic potential of G-protein coupled receptors in Huntington's disease

Wild type human TDP-43 potentiates ALS-linked mutant TDP-43 driven progressive motor and cortical neuron degeneration with pathological features of ALS.