Lista de obras - D Jurk - Dominio Público Uruguay

17α-Estradiol Alleviates Age-related Metabolic and Inflammatory Dysfunction in Male Mice Without Inducing Feminization

artículo científico publicado en 2016

A Potent and Specific CD38 Inhibitor Ameliorates Age-Related Metabolic Dysfunction by Reversing Tissue NAD Decline

scholarly article by Mariana G Tarragó published in May 2018

A new gene set identifies senescent cells and predicts senescence-associated pathways across tissues

artículo científico publicado en 2022

A novel Sudan Black B-based analogue revives lipofuscin as a biomarker for in vivo senescence

artículo científico publicado en 2018

A senescent cell bystander effect: senescence-induced senescence

artículo científico publicado en 2012

A stochastic step model of replicative senescence explains ROS production rate in ageing cell populations

artículo científico publicado en 2012

Adult-onset, short-term dietary restriction reduces cell senescence in mice.

artículo científico publicado en 2010

Amelioration of age-related brain function decline by Bruton's tyrosine kinase inhibition

scientific article published on 17 November 2019

Anti-inflammatory treatment rescues memory deficits during aging in nfkb1-/- mice

scientific article published on 11 September 2020

Cellular Senescence: Defining a Path Forward

scientific article published on 01 October 2019

Cellular senescence drives age-dependent hepatic steatosis.

artículo científico publicado en 2017

Chronic inflammation induces telomere dysfunction and accelerates ageing in mice.

artículo científico publicado en 2014

Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice.

artículo científico publicado en 2016

Corrigendum: NFκB1 is a suppressor of neutrophil-driven hepatocellular carcinoma.

artículo científico publicado en 2015

DNA damage response and cellular senescence in tissues of aging mice

artículo científico publicado en 2009

DNA damage response at telomeres contributes to lung aging and chronic obstructive pulmonary disease.

artículo científico publicado en 2015

Expansion and Cell-Cycle Arrest: Common Denominators of Cellular Senescence

artículo científico publicado en 2019

Length-independent telomere damage drives post-mitotic cardiomyocyte senescence

artículo científico publicado en 2019

Mitochondria are required for pro-ageing features of the senescent phenotype.

artículo científico publicado en 2016

Mitochondria-to-nucleus retrograde signaling drives formation of cytoplasmic chromatin and inflammation in senescence

artículo científico publicado en 2020

NFκB1 is a suppressor of neutrophil-driven hepatocellular carcinoma ⬇️

artículo científico publicado en 2015

Obesity-Induced Cellular Senescence Drives Anxiety and Impairs Neurogenesis

artículo científico publicado en 2019

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis

artículo científico publicado en 2018

Oxidative stress and life histories: unresolved issues and current needs.

artículo científico publicado en 2015

Postmitotic neurons develop a p21-dependent senescence-like phenotype driven by a DNA damage response

artículo científico publicado en 2012

Quantitative assessment of markers for cell senescence.

artículo científico publicado en 2010

Rapamycin improves healthspan but not inflammaging in nfκb1-/- mice

scientific article published on 23 November 2018

SQSTM1/p62 mediates crosstalk between autophagy and the UPS in DNA repair

artículo científico publicado en 2016

Senescent human melanocytes drive skin ageing via paracrine telomere dysfunction

scientific article published on 21 October 2019

Senolytics improve physical function and increase lifespan in old age

artículo científico publicado en 2018

Sustained telomere length in hepatocytes and cholangiocytes with increasing age in normal liver.

artículo científico publicado en 2012

Targeting senescent cells alleviates obesity-induced metabolic dysfunction

artículo científico publicado en 2019

Telomeres are favoured targets of a persistent DNA damage response in ageing and stress-induced senescence.

artículo científico publicado en 2012

Temporal inhibition of autophagy reveals segmental reversal of ageing with increased cancer risk

artículo científico publicado en 2020

The DNA Damage Response in Neurons: Die by Apoptosis or Survive in a Senescence-Like State?

artículo científico publicado en 2017

Transplanted Senescent Cells Induce an Osteoarthritis-Like Condition in Mice.

artículo científico publicado en 2016

Whole-body senescent cell clearance alleviates age-related brain inflammation and cognitive impairment in mice

artículo científico publicado en 2021

Lista de obras de D Jurk

17α-Estradiol Alleviates Age-related Metabolic and Inflammatory Dysfunction in Male Mice Without Inducing Feminization

A Potent and Specific CD38 Inhibitor Ameliorates Age-Related Metabolic Dysfunction by Reversing Tissue NAD Decline

A new gene set identifies senescent cells and predicts senescence-associated pathways across tissues

A novel Sudan Black B-based analogue revives lipofuscin as a biomarker for in vivo senescence

A senescent cell bystander effect: senescence-induced senescence

A stochastic step model of replicative senescence explains ROS production rate in ageing cell populations

Adult-onset, short-term dietary restriction reduces cell senescence in mice.

Amelioration of age-related brain function decline by Bruton's tyrosine kinase inhibition

Anti-inflammatory treatment rescues memory deficits during aging in nfkb1-/- mice

Cellular Senescence: Defining a Path Forward

Cellular senescence drives age-dependent hepatic steatosis.

Chronic inflammation induces telomere dysfunction and accelerates ageing in mice.

Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice.

Corrigendum: NFκB1 is a suppressor of neutrophil-driven hepatocellular carcinoma.

DNA damage response and cellular senescence in tissues of aging mice

DNA damage response at telomeres contributes to lung aging and chronic obstructive pulmonary disease.

Expansion and Cell-Cycle Arrest: Common Denominators of Cellular Senescence

Length-independent telomere damage drives post-mitotic cardiomyocyte senescence

Mitochondria are required for pro-ageing features of the senescent phenotype.

Mitochondria-to-nucleus retrograde signaling drives formation of cytoplasmic chromatin and inflammation in senescence

NFκB1 is a suppressor of neutrophil-driven hepatocellular carcinoma ⬇️

Obesity-Induced Cellular Senescence Drives Anxiety and Impairs Neurogenesis

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis

Oxidative stress and life histories: unresolved issues and current needs.

Postmitotic neurons develop a p21-dependent senescence-like phenotype driven by a DNA damage response

Quantitative assessment of markers for cell senescence.

Rapamycin improves healthspan but not inflammaging in nfκb1-/- mice

SQSTM1/p62 mediates crosstalk between autophagy and the UPS in DNA repair

Senescent human melanocytes drive skin ageing via paracrine telomere dysfunction

Senolytics improve physical function and increase lifespan in old age

Sustained telomere length in hepatocytes and cholangiocytes with increasing age in normal liver.

Targeting senescent cells alleviates obesity-induced metabolic dysfunction

Telomeres are favoured targets of a persistent DNA damage response in ageing and stress-induced senescence.

Temporal inhibition of autophagy reveals segmental reversal of ageing with increased cancer risk

The DNA Damage Response in Neurons: Die by Apoptosis or Survive in a Senescence-Like State?

Transplanted Senescent Cells Induce an Osteoarthritis-Like Condition in Mice.

Whole-body senescent cell clearance alleviates age-related brain inflammation and cognitive impairment in mice