Lista de obras - Junji Xing - Dominio Público Uruguay

A PY-nuclear localization signal is required for nuclear accumulation of HCMV UL79 protein

artículo científico publicado en 2012

Antiserum to the recombinant truncated VP22 protein of herpes simplex virus type 1 that also recognizes full-length VP22 ⬇️

artículo científico publicado el 1 de enero de 2011

Characterization of nuclear import and export signals determining the subcellular localization of WD repeat-containing protein 42A (WDR42A)

artículo científico publicado en 2012

Correction for Xing et al., The Z Proteins of Pathogenic but Not Nonpathogenic Arenaviruses Inhibit RIG-i-Like Receptor-Dependent Interferon Production.

artículo científico publicado en 2015

Corrigendum: Identification of a role for TRIM29 in the control of innate immunity in the respiratory tract.

artículo científico publicado en 2016

Differential Inhibition of Macrophage Activation by Lymphocytic Choriomeningitis Virus and Pichinde Virus Is Mediated by the Z Protein N-Terminal Domain.

artículo científico publicado en 2015

EphA2 phosphorylates NLRP3 and inhibits inflammasomes in airway epithelial cells

scientific article published on 30 April 2020

Fecal bacteria from treatment-naive Crohn's disease patients can skew helper T cell responses.

artículo científico publicado en 2017

Granulysin Production and Anticryptococcal Activity Is Dependent upon a Far Upstream Enhancer That Binds STAT5 in Human Peripheral Blood CD4+ T Cells

artículo científico publicado en 2010

Herpes simplex virus 1 tegument protein US11 downmodulates the RLR signaling pathway via direct interaction with RIG-I and MDA-5

artículo científico publicado en 2012

Human Semaphorin-4A drives Th2 responses by binding to receptor ILT-4.

artículo científico publicado en 2018

Identification of a novel NLS of herpes simplex virus type 1 (HSV-1) VP19C and its nuclear localization is required for efficient production of HSV-1.

artículo científico publicado en 2012

Identification of a role for TRIM29 in the control of innate immunity in the respiratory tract

artículo científico publicado en 2016

Identification of the E3 Ligase TRIM29 as a Critical Checkpoint Regulator of NK Cell Functions

artículo científico publicado en 2019

In vitro and in vivo characterizations of pichinde viral nucleoprotein exoribonuclease functions.

artículo científico publicado en 2015

Live cell imaging fails to support viral-protein-mediated intercellular trafficking

artículo científico publicado en 2012

Molecular anatomy of subcellular localization of HSV-1 tegument protein US11 in living cells.

artículo científico publicado en 2010

Molecular determinants responsible for the subcellular localization of HSV-1 UL4 protein

artículo científico publicado en 2011

Screening and identification of host factors interacting with UL14 of herpes simplex virus 1.

artículo científico publicado en 2011

TRIM29 Negatively Regulates the Type I IFN Production in Response to RNA Virus

artículo científico publicado en 2018

TRIM29 promotes DNA virus infections by inhibiting innate immune response

artículo científico publicado en 2017

The Z proteins of pathogenic but not nonpathogenic arenaviruses inhibit RIG-I-like receptor-dependent interferon production

artículo científico publicado en 2014

The nucleolus and viral infection.

artículo científico publicado en 2010

Varicella-zoster virus immediate-early protein ORF61 abrogates the IRF3-mediated innate immune response through degradation of activated IRF3

artículo científico publicado en 2011

Lista de obras de Junji Xing

A PY-nuclear localization signal is required for nuclear accumulation of HCMV UL79 protein

Antiserum to the recombinant truncated VP22 protein of herpes simplex virus type 1 that also recognizes full-length VP22 ⬇️

Characterization of nuclear import and export signals determining the subcellular localization of WD repeat-containing protein 42A (WDR42A)

Correction for Xing et al., The Z Proteins of Pathogenic but Not Nonpathogenic Arenaviruses Inhibit RIG-i-Like Receptor-Dependent Interferon Production.

Corrigendum: Identification of a role for TRIM29 in the control of innate immunity in the respiratory tract.

Differential Inhibition of Macrophage Activation by Lymphocytic Choriomeningitis Virus and Pichinde Virus Is Mediated by the Z Protein N-Terminal Domain.

EphA2 phosphorylates NLRP3 and inhibits inflammasomes in airway epithelial cells

Fecal bacteria from treatment-naive Crohn's disease patients can skew helper T cell responses.

Granulysin Production and Anticryptococcal Activity Is Dependent upon a Far Upstream Enhancer That Binds STAT5 in Human Peripheral Blood CD4+ T Cells

Herpes simplex virus 1 tegument protein US11 downmodulates the RLR signaling pathway via direct interaction with RIG-I and MDA-5

Human Semaphorin-4A drives Th2 responses by binding to receptor ILT-4.

Identification of a novel NLS of herpes simplex virus type 1 (HSV-1) VP19C and its nuclear localization is required for efficient production of HSV-1.

Identification of a role for TRIM29 in the control of innate immunity in the respiratory tract

Identification of the E3 Ligase TRIM29 as a Critical Checkpoint Regulator of NK Cell Functions

In vitro and in vivo characterizations of pichinde viral nucleoprotein exoribonuclease functions.

Live cell imaging fails to support viral-protein-mediated intercellular trafficking

Molecular anatomy of subcellular localization of HSV-1 tegument protein US11 in living cells.

Molecular determinants responsible for the subcellular localization of HSV-1 UL4 protein

Screening and identification of host factors interacting with UL14 of herpes simplex virus 1.

TRIM29 Negatively Regulates the Type I IFN Production in Response to RNA Virus

TRIM29 promotes DNA virus infections by inhibiting innate immune response

The Z proteins of pathogenic but not nonpathogenic arenaviruses inhibit RIG-I-like receptor-dependent interferon production

The nucleolus and viral infection.

Varicella-zoster virus immediate-early protein ORF61 abrogates the IRF3-mediated innate immune response through degradation of activated IRF3