Lista de obras - Paola De Cesaris - Dominio Público Uruguay

Activation of Jun N-terminal Kinase/Stress-activated Protein Kinase Pathway by Tumor Necrosis Factor α Leads to Intercellular Adhesion Molecule-1 Expression

artículo científico publicado en 1999

Activation of inositol phospholipid turnover and calcium signaling in rat Sertoli cells by P2-purinergic receptors: modulation of follicle-stimulating hormone responses

article

Characterization of signaling pathways leading to Fas expression induced by TNF-alpha: pivotal role of NF-kappaB.

artículo científico publicado en 2004

Control and impairment of immune privilege in the testis and in semen.

artículo científico publicado en 2001

Endothelin induces functional hypertrophy of peritubular smooth muscle cells

artículo científico publicado en 2007

Identification and Characterization of an Ecto-ATPase Activity in Rat Sertoli Cells

artículo científico publicado en 1996

Inflammatory mediators increase surface expression of integrin ligands, adhesion to lymphocytes, and secretion of interleukin 6 in mouse Sertoli cells.

artículo científico publicado en 1995

PKCalpha-mediated ERK, JNK and p38 activation regulates the myogenic program in human rhabdomyosarcoma cells.

artículo científico publicado en 2002

Platelet-derived growth factor-BB-induced hypertrophy of peritubular smooth muscle cells is mediated by activation of p38 MAP-kinase and of Rho-kinase

artículo científico publicado en 2006

Spectrin, fodrin and protein 4.1-like proteins in differentiating rat germ cells

article

TLR stimulation of prostate tumor cells induces chemokine-mediated recruitment of specific immune cell types.

artículo científico publicado en 2010

TLR3 engagement induces IRF-3-dependent apoptosis in androgen-sensitive prostate cancer cells and inhibits tumour growth in vivo.

artículo científico publicado en 2014

The contractile phenotype of peritubular smooth muscle cells is locally controlled: possible implications in male fertility.

artículo científico publicado en 2005

Toll-like receptor 3 activation induces antiviral immune responses in mouse sertoli cells

scientific journal article

Toll-like receptor 3 regulates angiogenesis and apoptosis in prostate cancer cell lines through hypoxia-inducible factor 1 alpha

artículo científico publicado en 2010

Toll-like receptor 3 triggers apoptosis of human prostate cancer cells through a PKC-alpha-dependent mechanism.

artículo científico publicado en 2008

Toll-like receptors in prostate infection and cancer between bench and bedside

artículo científico publicado en 2013

Transfected poly(I:C) activates different dsRNA receptors, leading to apoptosis or immunoadjuvant response in androgen-independent prostate cancer cells

artículo científico publicado en 2015

Tumor Necrosis Factor-α Induces Interleukin-6 Production and Integrin Ligand Expression by Distinct Transduction Pathways

scientific article published in Journal of Biological Chemistry

c-Flip expression and function in fetal mouse gonocytes

scientific journal article

c-Flip overexpression reduces cardiac hypertrophy in response to pressure overload

article

c-FlipLis expressed in undifferentiated mouse male germ cells

artículo científico publicado en 2006

p21WAF1 expression induced by MEK/ERK pathway activation or inhibition correlates with growth arrest, myogenic differentiation and onco-phenotype reversal in rhabdomyosarcoma cells.

artículo científico publicado en 2005

Lista de obras de Paola De Cesaris

Activation of Jun N-terminal Kinase/Stress-activated Protein Kinase Pathway by Tumor Necrosis Factor α Leads to Intercellular Adhesion Molecule-1 Expression

Activation of inositol phospholipid turnover and calcium signaling in rat Sertoli cells by P2-purinergic receptors: modulation of follicle-stimulating hormone responses

Characterization of signaling pathways leading to Fas expression induced by TNF-alpha: pivotal role of NF-kappaB.

Control and impairment of immune privilege in the testis and in semen.

Endothelin induces functional hypertrophy of peritubular smooth muscle cells

Identification and Characterization of an Ecto-ATPase Activity in Rat Sertoli Cells

Inflammatory mediators increase surface expression of integrin ligands, adhesion to lymphocytes, and secretion of interleukin 6 in mouse Sertoli cells.

PKCalpha-mediated ERK, JNK and p38 activation regulates the myogenic program in human rhabdomyosarcoma cells.

Platelet-derived growth factor-BB-induced hypertrophy of peritubular smooth muscle cells is mediated by activation of p38 MAP-kinase and of Rho-kinase

Spectrin, fodrin and protein 4.1-like proteins in differentiating rat germ cells

TLR stimulation of prostate tumor cells induces chemokine-mediated recruitment of specific immune cell types.

TLR3 engagement induces IRF-3-dependent apoptosis in androgen-sensitive prostate cancer cells and inhibits tumour growth in vivo.

The contractile phenotype of peritubular smooth muscle cells is locally controlled: possible implications in male fertility.

Toll-like receptor 3 activation induces antiviral immune responses in mouse sertoli cells

Toll-like receptor 3 regulates angiogenesis and apoptosis in prostate cancer cell lines through hypoxia-inducible factor 1 alpha

Toll-like receptor 3 triggers apoptosis of human prostate cancer cells through a PKC-alpha-dependent mechanism.

Toll-like receptors in prostate infection and cancer between bench and bedside

Transfected poly(I:C) activates different dsRNA receptors, leading to apoptosis or immunoadjuvant response in androgen-independent prostate cancer cells

Tumor Necrosis Factor-α Induces Interleukin-6 Production and Integrin Ligand Expression by Distinct Transduction Pathways

c-Flip expression and function in fetal mouse gonocytes

c-Flip overexpression reduces cardiac hypertrophy in response to pressure overload

c-FlipLis expressed in undifferentiated mouse male germ cells

p21WAF1 expression induced by MEK/ERK pathway activation or inhibition correlates with growth arrest, myogenic differentiation and onco-phenotype reversal in rhabdomyosarcoma cells.