Lista de obras - Catherine A Blizzard - Dominio Público Uruguay

A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During Disease

artículo científico publicado en 2016

Amyotrophic lateral sclerosis associated mislocalisation of TDP-43 to the cytoplasm causes cortical hyperexcitability and reduced excitatory neurotransmission in the motor cortex

artículo científico publicado en 2020

Amyotrophic lateral sclerosis mutant TDP-43 may cause synaptic dysfunction through altered dendritic spine function

scientific article published on 17 May 2019

Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

artículo científico publicado en 2016

Axonal shearing in mature cortical neurons induces attempted regeneration and the reestablishment of neurite polarity

artículo científico publicado en 2009

Axonopathy and cytoskeletal disruption in degenerative diseases of the central nervous system.

artículo científico publicado en 2009

Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.

artículo científico publicado en 2017

Chronic excitotoxin-induced axon degeneration in a compartmented neuronal culture model

artículo científico publicado en 2012

Cortical Murine Neurons Lacking the Neurofilament Light Chain Protein Have an Attenuated Response to InjuryIn Vitro ⬇️

artículo científico publicado el 15 de noviembre de 2013

Degeneration of axons in spinal white matter in G93A mSOD1 mouse characterized by NFL and α-internexin immunoreactivity

artículo científico publicado en 2012

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

artículo científico publicado en 2021

Epothilone D accelerates disease progression in the SOD1G93A mouse model of Amyotrophic Lateral Sclerosis

artículo científico publicado en 2018

Excitotoxicity mediated by non-NMDA receptors causes distal axonopathy in long-term cultured spinal motor neurons.

artículo científico publicado en 2007

Focal damage to the adult rat neocortex induces wound healing accompanied by axonal sprouting and dendritic structural plasticity

artículo científico publicado en 2010

Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis - vulnerability of lower motor neurons to proximal excitotoxicity

artículo científico publicado en 2015

Inducing Chronic Excitotoxicity in the Mouse Spinal Cord to Investigate Lower Motor Neuron Degeneration

artículo científico publicado en 2016

Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities

artículo científico publicado en 2015

Microfluidic primary culture model of the lower motor neuron-neuromuscular junction circuit

artículo científico publicado en 2013

Mild Traumatic Brain Injury Leads to Decreased Inhibition and a Differential Response of Calretinin Positive Interneurons in the Injured Cortex.

artículo científico publicado en 2017

Mild and repetitive very mild axonal stretch injury triggers cystoskeletal mislocalization and growth cone collapse

artículo científico publicado en 2017

Reduced Excitability and Increased Neurite Complexity of Cortical Interneurons in a Familial Mouse Model of Amyotrophic Lateral Sclerosis

artículo científico publicado en 2018

Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.

artículo científico publicado en 2016

The Microtubule-Modulating Drug Epothilone D Alters Dendritic Spine Morphology in a Mouse Model of Mild Traumatic Brain Injury

artículo científico publicado en 2018

The Tasmanian devil transcriptome reveals Schwann cell origins of a clonally transmissible cancer

artículo científico publicado en 2010

The degree of astrocyte activation in multiple system atrophy is inversely proportional to the distance to α-synuclein inclusions

artículo científico publicado en 2015

The microtubule-stabilizing drug Epothilone D increases axonal sprouting following transection injury in vitro.

artículo científico publicado en 2015

The pathologic outcomes and efficacy of epothilone treatment following traumatic brain injury is determined by age

artículo científico publicado en 2020

Lista de obras de Catherine A Blizzard

A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During Disease

Amyotrophic lateral sclerosis associated mislocalisation of TDP-43 to the cytoplasm causes cortical hyperexcitability and reduced excitatory neurotransmission in the motor cortex

Amyotrophic lateral sclerosis mutant TDP-43 may cause synaptic dysfunction through altered dendritic spine function

Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Axonal shearing in mature cortical neurons induces attempted regeneration and the reestablishment of neurite polarity

Axonopathy and cytoskeletal disruption in degenerative diseases of the central nervous system.

Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.

Chronic excitotoxin-induced axon degeneration in a compartmented neuronal culture model

Cortical Murine Neurons Lacking the Neurofilament Light Chain Protein Have an Attenuated Response to InjuryIn Vitro ⬇️

Degeneration of axons in spinal white matter in G93A mSOD1 mouse characterized by NFL and α-internexin immunoreactivity

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

Epothilone D accelerates disease progression in the SOD1G93A mouse model of Amyotrophic Lateral Sclerosis

Excitotoxicity mediated by non-NMDA receptors causes distal axonopathy in long-term cultured spinal motor neurons.

Focal damage to the adult rat neocortex induces wound healing accompanied by axonal sprouting and dendritic structural plasticity

Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis - vulnerability of lower motor neurons to proximal excitotoxicity

Inducing Chronic Excitotoxicity in the Mouse Spinal Cord to Investigate Lower Motor Neuron Degeneration

Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities

Microfluidic primary culture model of the lower motor neuron-neuromuscular junction circuit

Mild Traumatic Brain Injury Leads to Decreased Inhibition and a Differential Response of Calretinin Positive Interneurons in the Injured Cortex.

Mild and repetitive very mild axonal stretch injury triggers cystoskeletal mislocalization and growth cone collapse

Reduced Excitability and Increased Neurite Complexity of Cortical Interneurons in a Familial Mouse Model of Amyotrophic Lateral Sclerosis

Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.

The Microtubule-Modulating Drug Epothilone D Alters Dendritic Spine Morphology in a Mouse Model of Mild Traumatic Brain Injury

The Tasmanian devil transcriptome reveals Schwann cell origins of a clonally transmissible cancer

The degree of astrocyte activation in multiple system atrophy is inversely proportional to the distance to α-synuclein inclusions

The microtubule-stabilizing drug Epothilone D increases axonal sprouting following transection injury in vitro.

The pathologic outcomes and efficacy of epothilone treatment following traumatic brain injury is determined by age