Lista de obras - Boris Calderon - Dominio Público Uruguay

A minor subset of Batf3-dependent antigen-presenting cells in islets of Langerhans is essential for the development of autoimmune diabetes

artículo científico publicado en 2014

Antibody Standardization to Benefit Biomedical Research

artículo científico publicado en 2015

Antigen presentation events in autoimmune diabetes.

artículo científico publicado en 2011

Beta cells transfer vesicles containing insulin to phagocytes for presentation to T cells.

artículo científico publicado en 2015

Cellular and molecular events in the localization of diabetogenic T cells to islets of Langerhans ⬇️

artículo científico publicado el 10 de enero de 2011

DNA double-strand breaks activate a multi-functional genetic program in developing lymphocytes

artículo científico publicado en 2008

Defining the transcriptional and cellular landscape of type 1 diabetes in the NOD mouse

artículo científico publicado en 2013

Dendritic cells in islets of Langerhans constitutively present beta cell-derived peptides bound to their class II MHC molecules

artículo científico publicado en 2008

Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation

scientific article published on January 2014

Entry of diabetogenic T cells into islets induces changes that lead to amplification of the cellular response

artículo científico publicado en 2011

Heavy/light chain assay for monitoring IgA multiple myeloma: digging out the IgA from the β region

artículo científico publicado en 2014

Immunological reversal of autoimmune diabetes without hematopoietic replacement of beta cells.

artículo científico publicado en 2006

In CD4+ T-cell-induced diabetes, macrophages are the final effector cells that mediate islet beta-cell killing: studies from an acute model.

artículo científico publicado en 2006

Inhibition of VEGFR-2 reverses type 1 diabetes in NOD mice by abrogating insulitis and restoring islet function

artículo científico publicado en 2013

Investigation of pre-existing reactivity to biotherapeutics can uncover potential immunogenic epitopes and predict immunogenicity risk

artículo científico publicado en 2019

Islet autoantibodies and type 1 diabetes: does the evidence support screening?

artículo científico publicado en 2013

Lymphocytes are detrimental during the early innate immune response against Listeria monocytogenes.

artículo científico publicado en 2006

Pathogenic CD4⁺ T cells recognizing an unstable peptide of insulin are directly recruited into islets bypassing local lymph nodes.

artículo científico publicado en 2013

RNA sensor-induced type I IFN prevents diabetes caused by a β cell-tropic virus in mice

artículo científico publicado en 2011

The central role of antigen presentation in islets of Langerhans in autoimmune diabetes

artículo científico publicado en 2013

The pancreas anatomy conditions the origin and properties of resident macrophages

artículo científico publicado en 2015

Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection.

artículo científico publicado en 2004

Unique autoreactive T cells recognize insulin peptides generated within the islets of Langerhans in autoimmune diabetes

artículo científico publicado en 2010

ZnT8-reactive T cells are weakly pathogenic in NOD mice but can participate in diabetes under inflammatory conditions

artículo científico publicado en 2014

Lista de obras de Boris Calderon

A minor subset of Batf3-dependent antigen-presenting cells in islets of Langerhans is essential for the development of autoimmune diabetes

Antibody Standardization to Benefit Biomedical Research

Antigen presentation events in autoimmune diabetes.

Beta cells transfer vesicles containing insulin to phagocytes for presentation to T cells.

Cellular and molecular events in the localization of diabetogenic T cells to islets of Langerhans ⬇️

DNA double-strand breaks activate a multi-functional genetic program in developing lymphocytes

Defining the transcriptional and cellular landscape of type 1 diabetes in the NOD mouse

Dendritic cells in islets of Langerhans constitutively present beta cell-derived peptides bound to their class II MHC molecules

Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation

Entry of diabetogenic T cells into islets induces changes that lead to amplification of the cellular response

Heavy/light chain assay for monitoring IgA multiple myeloma: digging out the IgA from the β region

Immunological reversal of autoimmune diabetes without hematopoietic replacement of beta cells.

In CD4+ T-cell-induced diabetes, macrophages are the final effector cells that mediate islet beta-cell killing: studies from an acute model.

Inhibition of VEGFR-2 reverses type 1 diabetes in NOD mice by abrogating insulitis and restoring islet function

Investigation of pre-existing reactivity to biotherapeutics can uncover potential immunogenic epitopes and predict immunogenicity risk

Islet autoantibodies and type 1 diabetes: does the evidence support screening?

Lymphocytes are detrimental during the early innate immune response against Listeria monocytogenes.

Pathogenic CD4⁺ T cells recognizing an unstable peptide of insulin are directly recruited into islets bypassing local lymph nodes.

RNA sensor-induced type I IFN prevents diabetes caused by a β cell-tropic virus in mice

The central role of antigen presentation in islets of Langerhans in autoimmune diabetes

The pancreas anatomy conditions the origin and properties of resident macrophages

Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection.

Unique autoreactive T cells recognize insulin peptides generated within the islets of Langerhans in autoimmune diabetes

ZnT8-reactive T cells are weakly pathogenic in NOD mice but can participate in diabetes under inflammatory conditions