Lista de obras - Eva Petermann - Dominio Público Uruguay

ATP-dependent selection between single nucleotide and long patch base excision repair.

artículo científico publicado en 2003

ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells

artículo científico publicado en 2015

ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling

artículo científico publicado en 2006

Adenovirus E1B-55K targets SMARCAL1 for degradation during infection and modulates cellular DNA replication

scientific article published on 14 June 2019

BET Inhibition Induces HEXIM1- and RAD51-Dependent Conflicts between Transcription and Replication

artículo científico publicado en 2018

BET inhibition as a single or combined therapeutic approach in primary paediatric B-precursor acute lymphoblastic leukaemia.

artículo científico publicado en 2013

BRCA2 and RAD51 promote double-strand break formation and cell death in response to gemcitabine.

artículo científico publicado en 2014

Cancer therapy and replication stress: forks on the road to perdition.

artículo científico

Chk1 regulates the density of active replication origins during the vertebrate S phase.

artículo científico publicado en 2007

DNA damage by X-rays and their impact on replication processes.

artículo científico publicado en 2012

DNA replication-associated lesions: importance in early tumorigenesis and cancer therapy.

artículo científico publicado en 2007

Diminished origin-licensing capacity specifically sensitizes tumor cells to replication stress.

scientific article published on 30 January 2013

Essential function of Chk1 can be uncoupled from DNA damage checkpoint and replication control

scholarly article

Evidence that the ATR/Chk1 pathway maintains normal replication fork progression during unperturbed S phase.

artículo científico publicado en 2006

Human single-stranded DNA binding protein 1 (hSSB1/NABP2) is required for the stability and repair of stalled replication forks.

artículo científico publicado en 2014

Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair.

artículo científico publicado en 2010

Increased global transcription activity as a mechanism of replication stress in cancer

artículo científico publicado en 2016

Loss of p53 suppresses replication-stress-induced DNA breakage in G1/S checkpoint deficient cells

artículo científico publicado en 2018

MYBL2 supports DNA double strand break repair in haematopoietic stem cells

Measurement of chromosomal DNA single-strand breaks and replication fork progression rates

artículo científico publicado en 2006

Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.

artículo científico publicado en 2018

Methylated DNA causes a physical block to replication forks independently of damage signalling, O(6)-methylguanine or DNA single-strand breaks and results in DNA damage.

artículo científico publicado en 2010

PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination.

scientific article published on 23 July 2009

PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation.

artículo científico publicado en 2018

Pathways of mammalian replication fork restart.

artículo científico publicado en 2010

PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts

artículo científico publicado en 2020

Replication fork dynamics and the DNA damage response.

artículo científico

Synthetic lethality in chronic lymphocytic leukaemia with DNA damage response defects by targeting the ATR pathway

artículo científico publicado en 2015

Tannins elevate the level of poly(ADP-ribose) in HeLa cell extracts.

artículo científico publicado en 2004

Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival ⬇️

artículo científico publicado el 14 de noviembre de 2011

The propeptide is not required to produce catalytically active neutral protease from Bacillus stearothermophilus.

artículo científico publicado en 2005

Lista de obras de Eva Petermann

ATP-dependent selection between single nucleotide and long patch base excision repair.

ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells

ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling

Adenovirus E1B-55K targets SMARCAL1 for degradation during infection and modulates cellular DNA replication

BET Inhibition Induces HEXIM1- and RAD51-Dependent Conflicts between Transcription and Replication

BET inhibition as a single or combined therapeutic approach in primary paediatric B-precursor acute lymphoblastic leukaemia.

BRCA2 and RAD51 promote double-strand break formation and cell death in response to gemcitabine.

Cancer therapy and replication stress: forks on the road to perdition.

Chk1 regulates the density of active replication origins during the vertebrate S phase.

DNA damage by X-rays and their impact on replication processes.

DNA replication-associated lesions: importance in early tumorigenesis and cancer therapy.

Diminished origin-licensing capacity specifically sensitizes tumor cells to replication stress.

Essential function of Chk1 can be uncoupled from DNA damage checkpoint and replication control

Evidence that the ATR/Chk1 pathway maintains normal replication fork progression during unperturbed S phase.

Human single-stranded DNA binding protein 1 (hSSB1/NABP2) is required for the stability and repair of stalled replication forks.

Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair.

Increased global transcription activity as a mechanism of replication stress in cancer

Loss of p53 suppresses replication-stress-induced DNA breakage in G1/S checkpoint deficient cells

MYBL2 supports DNA double strand break repair in haematopoietic stem cells

Measurement of chromosomal DNA single-strand breaks and replication fork progression rates

Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.

Methylated DNA causes a physical block to replication forks independently of damage signalling, O(6)-methylguanine or DNA single-strand breaks and results in DNA damage.

PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination.

PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation.

Pathways of mammalian replication fork restart.

PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts

Replication fork dynamics and the DNA damage response.

Synthetic lethality in chronic lymphocytic leukaemia with DNA damage response defects by targeting the ATR pathway

Tannins elevate the level of poly(ADP-ribose) in HeLa cell extracts.

Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival ⬇️

The propeptide is not required to produce catalytically active neutral protease from Bacillus stearothermophilus.