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Lista de obras de Raphael S Peres

CCR2 Expression in Neutrophils Plays a Critical Role in Their Migration Into the Joints in Rheumatoid Arthritis.

artículo científico publicado en 2015

Cigarette smoke induces miR-132 in Th17 cells that enhance osteoclastogenesis in inflammatory arthritis

scholarly article

Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway

artículo científico publicado en 2015

Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production

artículo científico publicado en 2015

Lapachol, a compound targeting pyrimidine metabolism, ameliorates experimental autoimmune arthritis

artículo científico publicado en 2017

Low expression of CD39 on regulatory T cells as a biomarker for resistance to methotrexate therapy in rheumatoid arthritis

artículo científico publicado en 2015

NLRP3 inflammasome-mediated neutrophil recruitment and hypernociception depend on leukotriene B4 in a murine model of gout

article

Neutrophil Recruitment and Articular Hyperalgesia in Antigen-Induced Arthritis are Modulated by the Cholinergic Anti-Inflammatory Pathway

artículo científico publicado en 2016

Pharmacological opportunities to control inflammatory diseases through inhibition of the leukocyte recruitment

artículo científico publicado en 2016

Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells.

artículo científico publicado en 2018

Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes

artículo científico publicado en 2018

TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis.

artículo científico publicado en 2018

Therapeutic Treatment of Arthritic Mice with 15-Deoxy Δ12,14-Prostaglandin J2 (15d-PGJ2) Ameliorates Disease through the Suppression of Th17 Cells and the Induction of CD4+CD25-FOXP3+ Cells.

scientific article published on 31 October 2016

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