Lista de obras - António Duarte - Dominio Público Uruguay

ALK1 signaling inhibits angiogenesis by cooperating with the Notch pathway.

artículo científico publicado en 2012

Bone marrow-derived endothelial progenitors expressing Delta-like 4 (Dll4) regulate tumor angiogenesis.

artículo científico publicado en 2011

Context- and cell-dependent effects of Delta-like 4 targeting in the bone marrow microenvironment

artículo científico publicado en 2012

Delta-like 4 is the essential, nonredundant ligand for Notch1 during thymic T cell lineage commitment.

artículo científico publicado en 2008

Delta-like 4/Notch signaling promotes Apc Min/+ tumor initiation through angiogenic and non-angiogenic related mechanisms

artículo científico publicado en 2017

Differential expression of Notch component and effector genes during ovarian follicle and corpus luteum development during the oestrous cycle

artículo científico publicado en 2014

Dosage-sensitive requirement for mouse Dll4 in artery development

scientific journal article

Dynamics of Notch pathway expression during mouse testis post-natal development and along the spermatogenic cycle

artículo científico publicado en 2013

Dynamics of Notch signalling in the mouse oviduct and uterus during the oestrous cycle

artículo científico publicado en 2015

Endothelial Dll4 overexpression reduces vascular response and inhibits tumor growth and metastasization in vivo

artículo científico publicado en 2017

Endothelial Jagged1 antagonizes Dll4 regulation of endothelial branching and promotes vascular maturation downstream of Dll4/Notch1.

artículo científico publicado en 2015

Endothelial Jagged1 promotes solid tumor growth through both pro-angiogenic and angiocrine functions.

artículo científico publicado en 2015

Expression of hes6, a new member of the Hairy/Enhancer-of-split family, in mouse development

artículo científico publicado en 2000

Identification and functional analysis of endothelial tip cell-enriched genes

artículo científico publicado en 2010

Immune response profile elicited by the model antigen ovalbumin expressed in fusion with the bacterial OprI lipoprotein.

artículo científico publicado en 2014

In vivo notch signaling blockade induces abnormal spermatogenesis in the mouse

artículo científico publicado en 2014

Incomplete Dll4/Notch signaling inhibition promotes functional angiogenesis supporting the growth of skin papillomas ⬇️

artículo científico publicado en 2015

Inhibition of Notch signaling by Dll4-Fc promotes reperfusion of acutely ischemic tissues.

artículo científico publicado en 2012

Loss of Notch signalling induced by Dll4 causes arterial calibre reduction by increasing endothelial cell response to angiogenic stimuli

artículo científico publicado en 2008

Low-dosage inhibition of Dll4 signaling promotes wound healing by inducing functional neo-angiogenesis

artículo científico publicado en 2012

Metastasis is impaired by endothelial-specific Dll4 loss-of-function through inhibition of epithelial-to-mesenchymal transition and reduction of cancer stem cells and circulating tumor cells

scientific article published on 22 May 2019

Nephropathy and defective spermatogenesis in mice transgenic for a single isoform of the Wilms' tumour suppressor protein, WT1-KTS, together with one disrupted Wt1 allele

artículo científico publicado en 2007

Notch Signaling Function in the Angiocrine Regulation of Tumor Development

scientific article published on 12 November 2020

Notch signaling dynamics in the adult healthy prostate and in prostatic tumor development.

artículo científico publicado en 2015

Notch-dependent VEGFR3 upregulation allows angiogenesis without VEGF-VEGFR2 signalling

scientific journal article

Notch1 is pan-endothelial at the onset of flow and regulated by flow.

artículo científico publicado en 2015

Overexpression of delta-like 4 induces arterialization and attenuates vessel formation in developing mouse embryos

scientific journal article

RNA binding by the Wilms tumor suppressor zinc finger proteins.

artículo científico publicado en 1996

Repression of promoters for the mouse insulin-like growth factor II-encoding gene (Igf-2) by products of the Wilms' tumour suppressor gene wt1

article

The Notch ligand Delta-like 4 negatively regulates endothelial tip cell formation and vessel branching.

artículo científico publicado en 2007

The notch ligand delta-like 4 regulates multiple stages of early hemato-vascular development

artículo científico publicado en 2012

Lista de obras de António Duarte

ALK1 signaling inhibits angiogenesis by cooperating with the Notch pathway.

Bone marrow-derived endothelial progenitors expressing Delta-like 4 (Dll4) regulate tumor angiogenesis.

Context- and cell-dependent effects of Delta-like 4 targeting in the bone marrow microenvironment

Delta-like 4 is the essential, nonredundant ligand for Notch1 during thymic T cell lineage commitment.

Delta-like 4/Notch signaling promotes Apc Min/+ tumor initiation through angiogenic and non-angiogenic related mechanisms

Differential expression of Notch component and effector genes during ovarian follicle and corpus luteum development during the oestrous cycle

Dosage-sensitive requirement for mouse Dll4 in artery development

Dynamics of Notch pathway expression during mouse testis post-natal development and along the spermatogenic cycle

Dynamics of Notch signalling in the mouse oviduct and uterus during the oestrous cycle

Endothelial Dll4 overexpression reduces vascular response and inhibits tumor growth and metastasization in vivo

Endothelial Jagged1 antagonizes Dll4 regulation of endothelial branching and promotes vascular maturation downstream of Dll4/Notch1.

Endothelial Jagged1 promotes solid tumor growth through both pro-angiogenic and angiocrine functions.

Expression of hes6, a new member of the Hairy/Enhancer-of-split family, in mouse development

Identification and functional analysis of endothelial tip cell-enriched genes

Immune response profile elicited by the model antigen ovalbumin expressed in fusion with the bacterial OprI lipoprotein.

In vivo notch signaling blockade induces abnormal spermatogenesis in the mouse

Incomplete Dll4/Notch signaling inhibition promotes functional angiogenesis supporting the growth of skin papillomas ⬇️

Inhibition of Notch signaling by Dll4-Fc promotes reperfusion of acutely ischemic tissues.

Loss of Notch signalling induced by Dll4 causes arterial calibre reduction by increasing endothelial cell response to angiogenic stimuli

Low-dosage inhibition of Dll4 signaling promotes wound healing by inducing functional neo-angiogenesis

Metastasis is impaired by endothelial-specific Dll4 loss-of-function through inhibition of epithelial-to-mesenchymal transition and reduction of cancer stem cells and circulating tumor cells

Nephropathy and defective spermatogenesis in mice transgenic for a single isoform of the Wilms' tumour suppressor protein, WT1-KTS, together with one disrupted Wt1 allele

Notch Signaling Function in the Angiocrine Regulation of Tumor Development

Notch signaling dynamics in the adult healthy prostate and in prostatic tumor development.

Notch-dependent VEGFR3 upregulation allows angiogenesis without VEGF-VEGFR2 signalling

Notch1 is pan-endothelial at the onset of flow and regulated by flow.

Overexpression of delta-like 4 induces arterialization and attenuates vessel formation in developing mouse embryos

RNA binding by the Wilms tumor suppressor zinc finger proteins.

Repression of promoters for the mouse insulin-like growth factor II-encoding gene (Igf-2) by products of the Wilms' tumour suppressor gene wt1

The Notch ligand Delta-like 4 negatively regulates endothelial tip cell formation and vessel branching.

The notch ligand delta-like 4 regulates multiple stages of early hemato-vascular development