Lista de obras - Stéphane Lourdel - Dominio Público Uruguay

A calcium-permeable non-selective cation channel in the thick ascending limb apical membrane of the mouse kidney

artículo científico publicado en 2011

A chloride channel at the basolateral membrane of the distal-convoluted tubule: a candidate ClC-K channel.

artículo científico publicado en 2003

A novel CLCN5 pathogenic mutation supports Dent disease with normal endosomal acidification

artículo científico publicado en 2018

CLCNKB mutations causing mild Bartter syndrome profoundly alter the pH and Ca2+ dependence of ClC-Kb channels

artículo científico publicado en 2013

Characterization of the mouse ClC-K1/Barttin chloride channel.

artículo científico publicado en 2013

ClC-5 mutations associated with Dent's disease: a major role of the dimer interface.

artículo científico publicado en 2011

ClC-K chloride channels: emerging pathophysiology of Bartter syndrome type 3.

artículo científico

Dual regulation of the native ClC-K2 chloride channel in the distal nephron by voltage and pH

scientific article published on September 2016

Exploration of the basolateral chloride channels in the renal tubule using

artículo científico publicado en 2004

Heterogeneity in the processing of CLCN5 mutants related to Dent disease.

artículo científico publicado en 2011

Heterogeneous distribution of chloride channels along the distal convoluted tubule probed by single-cell RT-PCR and patch clamp

scientific article published on 27 July 2004

How Bartter's and Gitelman's syndromes, and Dent's disease have provided important insights into the function of three renal chloride channels: ClC-Ka/b and ClC-5

scientific article published on 12 December 2005

Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis.

artículo científico publicado en 2005

Kir4.1/Kir5.1 channel forms the major K+ channel in the basolateral membrane of mouse renal collecting duct principal cells

artículo científico publicado en 2008

Mutation Update of the CLCN5 Gene Responsible for Dent Disease 1.

artículo científico

Novel CLCNKB mutations causing Bartter syndrome affect channel surface expression.

artículo científico publicado en 2013

Rattlesnake Phospholipase A2 Increases CFTR-Chloride Channel Current and Corrects ∆F508CFTR Dysfunction: Impact in Cystic Fibrosis

artículo científico publicado en 2016

Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome.

artículo científico publicado en 2011

Similar chloride channels in the connecting tubule and cortical collecting duct of the mouse kidney

artículo científico publicado en 2006

Voltage-dependent electrogenic chloride/proton exchange by endosomal CLC proteins

artículo científico publicado en 2005

Lista de obras de Stéphane Lourdel

A calcium-permeable non-selective cation channel in the thick ascending limb apical membrane of the mouse kidney

A chloride channel at the basolateral membrane of the distal-convoluted tubule: a candidate ClC-K channel.

A novel CLCN5 pathogenic mutation supports Dent disease with normal endosomal acidification

CLCNKB mutations causing mild Bartter syndrome profoundly alter the pH and Ca2+ dependence of ClC-Kb channels

Characterization of the mouse ClC-K1/Barttin chloride channel.

ClC-5 mutations associated with Dent's disease: a major role of the dimer interface.

ClC-K chloride channels: emerging pathophysiology of Bartter syndrome type 3.

Dual regulation of the native ClC-K2 chloride channel in the distal nephron by voltage and pH

Exploration of the basolateral chloride channels in the renal tubule using

Heterogeneity in the processing of CLCN5 mutants related to Dent disease.

Heterogeneous distribution of chloride channels along the distal convoluted tubule probed by single-cell RT-PCR and patch clamp

How Bartter's and Gitelman's syndromes, and Dent's disease have provided important insights into the function of three renal chloride channels: ClC-Ka/b and ClC-5

Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis.

Kir4.1/Kir5.1 channel forms the major K+ channel in the basolateral membrane of mouse renal collecting duct principal cells

Mutation Update of the CLCN5 Gene Responsible for Dent Disease 1.

Novel CLCNKB mutations causing Bartter syndrome affect channel surface expression.

Rattlesnake Phospholipase A2 Increases CFTR-Chloride Channel Current and Corrects ∆F508CFTR Dysfunction: Impact in Cystic Fibrosis

Renal phenotype in mice lacking the Kir5.1 (Kcnj16) K+ channel subunit contrasts with that observed in SeSAME/EAST syndrome.

Similar chloride channels in the connecting tubule and cortical collecting duct of the mouse kidney

Voltage-dependent electrogenic chloride/proton exchange by endosomal CLC proteins