Lista de obras - Ashok Kumar - Dominio Público Uruguay

A novel long non-coding RNA Myolinc regulates myogenesis through TDP-43 and Filip1.

artículo científico publicado en 2018

Assay for redox-sensitive kinases

artículo científico publicado en 1999

Canonical NF-κB signaling regulates satellite stem cell homeostasis and function during regenerative myogenesis

scientific article published on 01 January 2019

Curcumin (Diferuloylmethane) inhibition of tumor necrosis factor (TNF)-mediated adhesion of monocytes to endothelial cells by suppression of cell surface expression of adhesion molecules and of nuclear factor-kappaB activation

artículo científico publicado en 1998

Distinct roles of TRAF6 and TAK1 in the regulation of adipocyte survival, thermogenesis program, and high-fat diet-induced obesity.

artículo científico publicado en 2017

Distinct roles of TRAF6 at early and late stages of muscle pathology in the mdx model of Duchenne muscular dystrophy.

scientific article published on 24 October 2013

Distinct signaling pathways are activated in response to mechanical stress applied axially and transversely to skeletal muscle fibers.

artículo científico publicado en 2002

Dlk1 is necessary for proper skeletal muscle development and regeneration

artículo científico publicado en 2010

ER stress in skeletal muscle remodeling and myopathies

artículo científico publicado en 2017

Effect of Dalton's lymphoma on the antigen presentation of murine peritoneal macrophages ⬇️

artículo científico publicado el 8 de junio de 1995

Effect of cisplatin administration on the proliferation and differentiation of bone marrow cells of tumour‐bearing mice ⬇️

artículo científico publicado el 1 de junio de 1995

Effect of cisplatin and FK565 on the activation of tumor-associated and bone marrow-derived macrophages by Dalton's lymphoma

scientific article published on 01 January 1995

Effect of prolactin on nitric oxide and interleukin-1 production of murine peritoneal macrophages: Role of Ca2+ and protein kinase C ⬇️

artículo científico publicado el 1 de marzo de 1997

Effects of omega-3 on matrix metalloproteinase-9, myoblast transplantation and satellite cell activation in dystrophin-deficient muscle fibers

artículo científico publicado en 2017

Elevated levels of TWEAK in skeletal muscle promote visceral obesity, insulin resistance, and metabolic dysfunction.

artículo científico publicado en 2014

Elevated levels of active matrix metalloproteinase-9 cause hypertrophy in skeletal muscle of normal and dystrophin-deficient mdx mice.

artículo científico publicado en 2011

Emerging roles of ER stress and unfolded protein response pathways in skeletal muscle health and disease

artículo científico publicado en 2017

Emodin (3-methyl-1,6,8-trihydroxyanthraquinone) inhibits TNF-induced NF-kappaB activation, IkappaB degradation, and expression of cell surface adhesion proteins in human vascular endothelial cells.

artículo científico publicado en 1998

Fibroblast growth factor inducible 14 (Fn14) is required for the expression of myogenic regulatory factors and differentiation of myoblasts into myotubes. Evidence for TWEAK-independent functions of Fn14 during myogenesis

artículo científico publicado en 2007

Gene Profiling Studies in Skeletal Muscle by Quantitative Real-Time Polymerase Chain Reaction Assay ⬇️

artículo científico publicado el 1 de enero de 2012

Genomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in mice

artículo científico publicado en 2010

Human immunodeficiency virus-1-tat induces matrix metalloproteinase-9 in monocytes through protein tyrosine phosphatase-mediated activation of nuclear transcription factor NF-kappaB.

artículo científico publicado en 1999

Isolation, Culture, and Staining of Single Myofibers

artículo científico publicado en 2016

Isolation, Culturing, and Differentiation of Primary Myoblasts from Skeletal Muscle of Adult Mice

artículo científico publicado en 2017

Matrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy.

scientific article published on 28 April 2009

Matrix metalloproteinase-9 inhibition improves proliferation and engraftment of myogenic cells in dystrophic muscle of mdx mice

artículo científico publicado en 2013

Mechanical stress activates the nuclear factor-kappaB pathway in skeletal muscle fibers: a possible role in Duchenne muscular dystrophy.

artículo científico publicado en 2003

Mechanical stretch activates nuclear factor-kappaB, activator protein-1, and mitogen-activated protein kinases in lung parenchyma: implications in asthma

artículo científico publicado en 2003

MyD88 is required for satellite cell-mediated myofiber regeneration in dystrophin-deficient mdx mice

scientific article published on 01 October 2018

MyD88 promotes myoblast fusion in a cell-autonomous manner

artículo científico publicado en 2017

Noncoding RNAs in the regulation of skeletal muscle biology in health and disease.

artículo científico publicado en 2016

Nuclear factor-kappa B signaling in skeletal muscle atrophy.

artículo científico publicado en 2008

Osteopontin-Stimulated Expression of Matrix Metalloproteinase-9 Causes Cardiomyopathy in the mdx Model of Duchenne Muscular Dystrophy ⬇️

artículo científico publicado el 1 de septiembre de 2011

PERK regulates skeletal muscle mass and contractile function in adult mice

artículo científico publicado en 2018

Physiological Biomimetic Culture System for Pig and Human Heart Slices

scientific article published on 16 July 2019

Proinflammatory cytokine tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) suppresses satellite cell self-renewal through inversely modulating Notch and NF-κB signaling pathways

scientific article published on 22 October 2013

Protein-DNA array-based identification of transcription factor activities differentially regulated in skeletal muscle of normal and dystrophin-deficient mdx mice.

artículo científico publicado en 2008

Reciprocal Interaction between TRAF6 and Notch Signaling Regulates Adult Myofiber Regeneration upon Injury ⬇️

artículo científico publicado el 1 de octubre de 2012

Regulation of phosphatidylinositol 3-kinase (PI3K)/Akt and nuclear factor-kappa B signaling pathways in dystrophin-deficient skeletal muscle in response to mechanical stretch.

artículo científico publicado en 2006

Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program.

artículo científico publicado en 2013

Sanguinarine (Pseudochelerythrine) Is a Potent Inhibitor of NF-κB Activation, IκBα Phosphorylation, and Degradation ⬇️

artículo científico publicado el 28 de noviembre de 1997

Signaling Mechanisms in Mammalian Myoblast Fusion ⬇️

artículo científico publicado el 23 de abril de 2013

TAK1 modulates satellite stem cell homeostasis and skeletal muscle repair

artículo científico publicado en 2015

TAK1 regulates skeletal muscle mass and mitochondrial function

artículo científico publicado en 2018

TNF-like weak inducer of apoptosis (TWEAK) activates proinflammatory signaling pathways and gene expression through the activation of TGF-beta-activated kinase 1

artículo científico publicado en 2009

TNF-related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle-wasting cytokine

artículo científico publicado en 2007

TRAF6 coordinates the activation of autophagy and ubiquitin-proteasome systems in atrophying skeletal muscle ⬇️

artículo científico publicado el 1 de mayo de 2011

TRAF6 regulates satellite stem cell self-renewal and function during regenerative myogenesis.

artículo científico publicado en 2015

TWEAK and TRAF6 regulate skeletal muscle atrophy.

artículo científico publicado en 2012

TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.

artículo científico publicado en 2012

TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.

artículo científico publicado en 2013

TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction.

artículo científico publicado en 2014

Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice. ⬇️

artículo científico publicado en 2010

The E3 ubiquitin ligase TRAF6 intercedes in starvation-induced skeletal muscle atrophy through multiple mechanisms

artículo científico publicado en 2012

The PERK arm of the unfolded protein response regulates satellite cell-mediated skeletal muscle regeneration

artículo científico publicado en 2017

The TWEAK-Fn14 System: Breaking the Silence of Cytokine-Induced Skeletal Muscle Wasting ⬇️

artículo científico publicado el 1 de enero de 2012

The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscle

artículo científico publicado en 2014

The TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and disease

artículo científico publicado en 2014

The Toll-Like Receptor/MyD88/XBP1 Signaling Axis Mediates Skeletal Muscle Wasting during Cancer Cachexia

artículo científico publicado en 2019

Therapeutic potential of matrix metalloproteinases in Duchenne muscular dystrophy

artículo científico

Toll-like receptor signalling in regenerative myogenesis: friend and foe.

artículo científico publicado en 2016

Transgenic overexpression of pregnancy-associated plasma protein-A increases the somatic growth and skeletal muscle mass in mice.

artículo científico publicado en 2007

Tumor necrosis factor-alpha augments matrix metalloproteinase-9 production in skeletal muscle cells through the activation of transforming growth factor-beta-activated kinase 1 (TAK1)-dependent signaling pathway

artículo científico publicado en 2007

Tumor necrosis factor-like weak inducer of apoptosis inhibits skeletal myogenesis through sustained activation of nuclear factor-kappaB and degradation of MyoD protein.

artículo científico publicado en 2006

Tumor necrosis factor-α regulates distinct molecular pathways and gene networks in cultured skeletal muscle cells

artículo científico publicado en 2010

Wasting mechanisms in muscular dystrophy.

artículo científico publicado en 2013

Lista de obras de Ashok Kumar

A novel long non-coding RNA Myolinc regulates myogenesis through TDP-43 and Filip1.

Assay for redox-sensitive kinases

Canonical NF-κB signaling regulates satellite stem cell homeostasis and function during regenerative myogenesis

Curcumin (Diferuloylmethane) inhibition of tumor necrosis factor (TNF)-mediated adhesion of monocytes to endothelial cells by suppression of cell surface expression of adhesion molecules and of nuclear factor-kappaB activation

Distinct roles of TRAF6 and TAK1 in the regulation of adipocyte survival, thermogenesis program, and high-fat diet-induced obesity.

Distinct roles of TRAF6 at early and late stages of muscle pathology in the mdx model of Duchenne muscular dystrophy.

Distinct signaling pathways are activated in response to mechanical stress applied axially and transversely to skeletal muscle fibers.

Dlk1 is necessary for proper skeletal muscle development and regeneration

ER stress in skeletal muscle remodeling and myopathies

Effect of Dalton's lymphoma on the antigen presentation of murine peritoneal macrophages ⬇️

Effect of cisplatin administration on the proliferation and differentiation of bone marrow cells of tumour‐bearing mice ⬇️

Effect of cisplatin and FK565 on the activation of tumor-associated and bone marrow-derived macrophages by Dalton's lymphoma

Effect of prolactin on nitric oxide and interleukin-1 production of murine peritoneal macrophages: Role of Ca2+ and protein kinase C ⬇️

Effects of omega-3 on matrix metalloproteinase-9, myoblast transplantation and satellite cell activation in dystrophin-deficient muscle fibers

Elevated levels of TWEAK in skeletal muscle promote visceral obesity, insulin resistance, and metabolic dysfunction.

Elevated levels of active matrix metalloproteinase-9 cause hypertrophy in skeletal muscle of normal and dystrophin-deficient mdx mice.

Emerging roles of ER stress and unfolded protein response pathways in skeletal muscle health and disease

Emodin (3-methyl-1,6,8-trihydroxyanthraquinone) inhibits TNF-induced NF-kappaB activation, IkappaB degradation, and expression of cell surface adhesion proteins in human vascular endothelial cells.

Fibroblast growth factor inducible 14 (Fn14) is required for the expression of myogenic regulatory factors and differentiation of myoblasts into myotubes. Evidence for TWEAK-independent functions of Fn14 during myogenesis

Gene Profiling Studies in Skeletal Muscle by Quantitative Real-Time Polymerase Chain Reaction Assay ⬇️

Genomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in mice

Human immunodeficiency virus-1-tat induces matrix metalloproteinase-9 in monocytes through protein tyrosine phosphatase-mediated activation of nuclear transcription factor NF-kappaB.

Isolation, Culture, and Staining of Single Myofibers

Isolation, Culturing, and Differentiation of Primary Myoblasts from Skeletal Muscle of Adult Mice

Matrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy.

Matrix metalloproteinase-9 inhibition improves proliferation and engraftment of myogenic cells in dystrophic muscle of mdx mice

Mechanical stress activates the nuclear factor-kappaB pathway in skeletal muscle fibers: a possible role in Duchenne muscular dystrophy.

Mechanical stretch activates nuclear factor-kappaB, activator protein-1, and mitogen-activated protein kinases in lung parenchyma: implications in asthma

MyD88 is required for satellite cell-mediated myofiber regeneration in dystrophin-deficient mdx mice

MyD88 promotes myoblast fusion in a cell-autonomous manner

Noncoding RNAs in the regulation of skeletal muscle biology in health and disease.

Nuclear factor-kappa B signaling in skeletal muscle atrophy.

Osteopontin-Stimulated Expression of Matrix Metalloproteinase-9 Causes Cardiomyopathy in the mdx Model of Duchenne Muscular Dystrophy ⬇️

PERK regulates skeletal muscle mass and contractile function in adult mice

Physiological Biomimetic Culture System for Pig and Human Heart Slices

Proinflammatory cytokine tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) suppresses satellite cell self-renewal through inversely modulating Notch and NF-κB signaling pathways

Protein-DNA array-based identification of transcription factor activities differentially regulated in skeletal muscle of normal and dystrophin-deficient mdx mice.

Reciprocal Interaction between TRAF6 and Notch Signaling Regulates Adult Myofiber Regeneration upon Injury ⬇️

Regulation of phosphatidylinositol 3-kinase (PI3K)/Akt and nuclear factor-kappa B signaling pathways in dystrophin-deficient skeletal muscle in response to mechanical stretch.

Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program.

Sanguinarine (Pseudochelerythrine) Is a Potent Inhibitor of NF-κB Activation, IκBα Phosphorylation, and Degradation ⬇️

Signaling Mechanisms in Mammalian Myoblast Fusion ⬇️

TAK1 modulates satellite stem cell homeostasis and skeletal muscle repair

TAK1 regulates skeletal muscle mass and mitochondrial function

TNF-like weak inducer of apoptosis (TWEAK) activates proinflammatory signaling pathways and gene expression through the activation of TGF-beta-activated kinase 1

TNF-related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle-wasting cytokine

TRAF6 coordinates the activation of autophagy and ubiquitin-proteasome systems in atrophying skeletal muscle ⬇️

TRAF6 regulates satellite stem cell self-renewal and function during regenerative myogenesis.

TWEAK and TRAF6 regulate skeletal muscle atrophy.

TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.

TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.

TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction.

Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice. ⬇️

The E3 ubiquitin ligase TRAF6 intercedes in starvation-induced skeletal muscle atrophy through multiple mechanisms

The PERK arm of the unfolded protein response regulates satellite cell-mediated skeletal muscle regeneration

The TWEAK-Fn14 System: Breaking the Silence of Cytokine-Induced Skeletal Muscle Wasting ⬇️

The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscle

The TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and disease

The Toll-Like Receptor/MyD88/XBP1 Signaling Axis Mediates Skeletal Muscle Wasting during Cancer Cachexia

Therapeutic potential of matrix metalloproteinases in Duchenne muscular dystrophy

Toll-like receptor signalling in regenerative myogenesis: friend and foe.

Transgenic overexpression of pregnancy-associated plasma protein-A increases the somatic growth and skeletal muscle mass in mice.

Tumor necrosis factor-alpha augments matrix metalloproteinase-9 production in skeletal muscle cells through the activation of transforming growth factor-beta-activated kinase 1 (TAK1)-dependent signaling pathway

Tumor necrosis factor-like weak inducer of apoptosis inhibits skeletal myogenesis through sustained activation of nuclear factor-kappaB and degradation of MyoD protein.

Tumor necrosis factor-α regulates distinct molecular pathways and gene networks in cultured skeletal muscle cells

Wasting mechanisms in muscular dystrophy.