Lista de obras - Ilse Dewachter - Dominio Público Uruguay

A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model

artículo científico publicado en 2004

AAV-tau mediates pyramidal neurodegeneration by cell-cycle re-entry without neurofibrillary tangle formation in wild-type mice

artículo científico publicado en 2009

APP-dependent glial cell line-derived neurotrophic factor gene expression drives neuromuscular junction formation

artículo científico publicado en 2015

Activation of phagocytic activity in astrocytes by reduced expression of the inflammasome component ASC and its implication in a mouse model of Alzheimer disease

artículo científico publicado en 2016

Alzheimer's disease: old problem, new views from transgenic and viral models

scientific article published on 21 March 2010

Amyloid activates GSK-3beta to aggravate neuronal tauopathy in bigenic mice

artículo científico publicado en 2008

Amyloid precursor protein controls cholesterol turnover needed for neuronal activity

artículo científico publicado en 2013

Analysis by a highly sensitive split luciferase assay of the regions involved in APP dimerization and its impact on processing

artículo científico publicado en 2015

Axonal transport, tau protein, and neurodegeneration in Alzheimer's disease

artículo científico publicado en 2002

Beta-site amyloid precursor protein-cleaving enzyme-1 (BACE1)-mediated changes of endogenous amyloid beta in wild-type and transgenic mice in vivo

artículo científico publicado en 2008

Capacitive calcium entry is directly attenuated by mutant presenilin-1, independent of the expression of the amyloid precursor protein

scientific journal article

Cortical cells reveal APP as a new player in the regulation of GABAergic neurotransmission

artículo científico publicado en 2017

Deletion of the transient receptor potential cation channel TRPV4 impairs murine bladder voiding

artículo científico publicado en 2007

Focal glial activation coincides with increased BACE1 activation and precedes amyloid plaque deposition in APP[V717I] transgenic mice

artículo científico publicado en 2005

Glycines from the APP GXXXG/GXXXA Transmembrane Motifs Promote Formation of Pathogenic Aβ Oligomers in Cells

artículo científico publicado en 2016

Heterotypic seeding of Tau fibrillization by pre-aggregated Abeta provides potent seeds for prion-like seeding and propagation of Tau-pathology in vivo

artículo científico publicado en 2016

Neurodegeneration and neuroinflammation in cdk5/p25-inducible mice: a model for hippocampal sclerosis and neocortical degeneration

artículo científico publicado en 2008

Neurological characterization of mice deficient in GSK3α highlight pleiotropic physiological functions in cognition and pathological activity as Tau kinase

artículo científico publicado en 2013

Neuronal or glial expression of human apolipoprotein e4 affects parenchymal and vascular amyloid pathology differentially in different brain regions of double- and triple-transgenic mice

artículo científico publicado en 2006

Neuropathobiology in transgenic mice. The case of Alzheimer's disease.

artículo científico publicado en 2003

Nonsteroidal anti-inflammatory drugs and peroxisome proliferator-activated receptor-gamma agonists modulate immunostimulated processing of amyloid precursor protein through regulation of beta-secretase.

artículo científico publicado en 2003

Nonsteroidal anti-inflammatory drugs repress beta-secretase gene promoter activity by the activation of PPARgamma

artículo científico publicado en 2006

Presenilin transmembrane domain 8 conserved AXXXAXXXG motifs are required for the activity of the γ-secretase complex

artículo científico publicado en 2015

Reduction of beta-amyloid plaques in brain of transgenic mouse model of Alzheimer's disease by EFRH-phage immunization

artículo científico publicado en 2003

Secretases as targets for the treatment of Alzheimer's disease: the prospects

artículo científico publicado en 2002

Tau association with synaptic vesicles causes presynaptic dysfunction

artículo científico publicado en 2017

Tau interactome mapping based identification of Otub1 as Tau deubiquitinase involved in accumulation of pathological Tau forms in vitro and in vivo

artículo científico publicado en 2017

Tauopathy contributes to synaptic and cognitive deficits in a murine model for Alzheimer's disease

artículo científico publicado en 2014

Templated misfolding of Tau by prion-like seeding along neuronal connections impairs neuronal network function and associated behavioral outcomes in Tau transgenic mice

artículo científico publicado en 2015

The capsaicin receptor TRPV1 is a crucial mediator of the noxious effects of mustard oil

artículo científico publicado en 2011

Therapeutic effects of PKC activators in Alzheimer's disease transgenic mice

artículo científico publicado en 2004

Transgenic mouse models for APP processing and Alzheimer's disease: early and late defects.

artículo científico publicado en 2005

beta-site amyloid precursor protein cleaving enzyme 1 increases amyloid deposition in brain parenchyma but reduces cerebrovascular amyloid angiopathy in aging BACE x APP[V717I] double-transgenic mice

artículo científico publicado en 2004

Lista de obras de Ilse Dewachter

A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model

AAV-tau mediates pyramidal neurodegeneration by cell-cycle re-entry without neurofibrillary tangle formation in wild-type mice

APP-dependent glial cell line-derived neurotrophic factor gene expression drives neuromuscular junction formation

Activation of phagocytic activity in astrocytes by reduced expression of the inflammasome component ASC and its implication in a mouse model of Alzheimer disease

Alzheimer's disease: old problem, new views from transgenic and viral models

Amyloid activates GSK-3beta to aggravate neuronal tauopathy in bigenic mice

Amyloid precursor protein controls cholesterol turnover needed for neuronal activity

Analysis by a highly sensitive split luciferase assay of the regions involved in APP dimerization and its impact on processing

Axonal transport, tau protein, and neurodegeneration in Alzheimer's disease

Beta-site amyloid precursor protein-cleaving enzyme-1 (BACE1)-mediated changes of endogenous amyloid beta in wild-type and transgenic mice in vivo

Capacitive calcium entry is directly attenuated by mutant presenilin-1, independent of the expression of the amyloid precursor protein

Cortical cells reveal APP as a new player in the regulation of GABAergic neurotransmission

Deletion of the transient receptor potential cation channel TRPV4 impairs murine bladder voiding

Focal glial activation coincides with increased BACE1 activation and precedes amyloid plaque deposition in APP[V717I] transgenic mice

Glycines from the APP GXXXG/GXXXA Transmembrane Motifs Promote Formation of Pathogenic Aβ Oligomers in Cells

Heterotypic seeding of Tau fibrillization by pre-aggregated Abeta provides potent seeds for prion-like seeding and propagation of Tau-pathology in vivo

Neurodegeneration and neuroinflammation in cdk5/p25-inducible mice: a model for hippocampal sclerosis and neocortical degeneration

Neurological characterization of mice deficient in GSK3α highlight pleiotropic physiological functions in cognition and pathological activity as Tau kinase

Neuronal or glial expression of human apolipoprotein e4 affects parenchymal and vascular amyloid pathology differentially in different brain regions of double- and triple-transgenic mice

Neuropathobiology in transgenic mice. The case of Alzheimer's disease.

Nonsteroidal anti-inflammatory drugs and peroxisome proliferator-activated receptor-gamma agonists modulate immunostimulated processing of amyloid precursor protein through regulation of beta-secretase.

Nonsteroidal anti-inflammatory drugs repress beta-secretase gene promoter activity by the activation of PPARgamma

Presenilin transmembrane domain 8 conserved AXXXAXXXG motifs are required for the activity of the γ-secretase complex

Reduction of beta-amyloid plaques in brain of transgenic mouse model of Alzheimer's disease by EFRH-phage immunization

Secretases as targets for the treatment of Alzheimer's disease: the prospects

Tau association with synaptic vesicles causes presynaptic dysfunction

Tau interactome mapping based identification of Otub1 as Tau deubiquitinase involved in accumulation of pathological Tau forms in vitro and in vivo

Tauopathy contributes to synaptic and cognitive deficits in a murine model for Alzheimer's disease

Templated misfolding of Tau by prion-like seeding along neuronal connections impairs neuronal network function and associated behavioral outcomes in Tau transgenic mice

The capsaicin receptor TRPV1 is a crucial mediator of the noxious effects of mustard oil

Therapeutic effects of PKC activators in Alzheimer's disease transgenic mice

Transgenic mouse models for APP processing and Alzheimer's disease: early and late defects.

beta-site amyloid precursor protein cleaving enzyme 1 increases amyloid deposition in brain parenchyma but reduces cerebrovascular amyloid angiopathy in aging BACE x APP[V717I] double-transgenic mice