Lista de obras - Natalia I Dmitrieva - Dominio Público Uruguay

Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl

artículo científico publicado en 2008

Ataxia telangiectasia-mutated, a DNA damage-inducible kinase, contributes to high NaCl-induced nuclear localization of transcription factor TonEBP/OREBP.

artículo científico publicado en 2005

Cell cycle delay and apoptosis in response to osmotic stress.

artículo científico publicado en 2001

Cell cycle-dependent expression of thyroid hormone receptor-beta is a mechanism for variable hormone sensitivity

artículo científico publicado en 2004

Cells adapted to high NaCl have many DNA breaks and impaired DNA repair both in cell culture and in vivo

artículo científico publicado en 2004

Cellular response to hyperosmotic stresses

artículo científico publicado en 2007

Cross-Sectional Positive Association of Serum Lipids and Blood Pressure With Serum Sodium Within the Normal Reference Range of 135-145 mmol/L.

artículo científico publicado en 2016

DNA damage and osmotic regulation in the kidney

artículo científico publicado en 2005

DNA double-strand breaks induced by high NaCl occur predominantly in gene deserts

artículo científico publicado en 2011

Effects of expression of p53 and Gadd45 on osmotic tolerance of renal inner medullary cells.

artículo científico publicado en 2006

Elevated sodium and dehydration stimulate inflammatory signaling in endothelial cells and promote atherosclerosis

artículo científico publicado en 2015

High NaCl causes Mre11 to leave the nucleus, disrupting DNA damage signaling and repair

artículo científico publicado en 2003

High NaCl promotes cellular senescence

artículo científico publicado en 2007

High urea and NaCl carbonylate proteins in renal cells in culture and in vivo, and high urea causes 8-oxoguanine lesions in their DNA

artículo científico publicado en 2004

Hypertonic stress response

artículo científico publicado en 2005

Impaired angiogenesis and extracellular matrix metabolism in Autosomal-Dominant Hyper-IgE Syndrome

artículo científico publicado en 2020

Increased activity of TNAP compensates for reduced adenosine production and promotes ectopic calcification in the genetic disease ACDC.

artículo científico publicado en 2016

Increased insensible water loss contributes to aging related dehydration

artículo científico publicado en 2011

Involvement of endogenous digitalis-like factors in voluntary selection of alcohol by rats

artículo científico publicado en 1999

Ku86 preserves chromatin integrity in cells adapted to high NaCl

artículo científico publicado en 2005

MKP-1 inhibits high NaCl-induced activation of p38 but does not inhibit the activation of TonEBP/OREBP: opposite roles of p38alpha and p38delta

artículo científico publicado en 2008

Mediator of DNA damage checkpoint 1 (MDC1) contributes to high NaCl-induced activation of the osmoprotective transcription factor TonEBP/OREBP.

artículo científico publicado en 2010

Mitochondrial dysfunction is an early event in high-NaCl-induced apoptosis of mIMCD3 cells

artículo científico publicado en 2002

Mre11 is expressed in mammalian mitochondria where it binds to mitochondrial DNA.

artículo científico publicado en 2011

Mutations that prevent caspase cleavage of RIPK1 cause autoinflammatory disease

scientific article published on 11 December 2019

Osmotic Stress and DNA Damage

artículo científico publicado en 2007

Pax2 expression occurs in renal medullary epithelial cells in vivo and in cell culture, is osmoregulated, and promotes osmotic tolerance

artículo científico publicado en 2004

Proliferation and osmotic tolerance of renal inner medullary epithelial cells in vivo and in cell culture

artículo científico publicado en 2002

Protection of Renal Inner Medullary Epithelial Cells from Apoptosis by Hypertonic Stress-induced p53 Activation

scientific article published in Journal of Biological Chemistry

Rapid activation of G2/M checkpoint after hypertonic stress in renal inner medullary epithelial (IME) cells is protective and requires p38 kinase

artículo científico publicado en 2001

Rate of increase of osmolality determines osmotic tolerance of mouse inner medullary epithelial cells

artículo científico publicado en 2002

STAT3 modulates reprogramming efficiency of human somatic cells; Insights from autosomal dominant Hyper IgE syndrome caused by STAT3 mutations

artículo científico publicado en 2020

Secretion of von Willebrand factor by endothelial cells links sodium to hypercoagulability and thrombosis

artículo científico publicado en 2014

Suboptimal hydration remodels metabolism, promotes degenerative diseases, and shortens life

artículo científico publicado en 2019

The Saltiness of the Sea Breaks DNA in Marine Invertebrates: Possible Implications for Animal Evolution

artículo científico publicado en 2006

Toxicity of acetaminophen, salicylic acid, and caffeine for first-passage rat renal inner medullary collecting duct cells

artículo científico publicado en 2003

Lista de obras de Natalia I Dmitrieva

Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl

Ataxia telangiectasia-mutated, a DNA damage-inducible kinase, contributes to high NaCl-induced nuclear localization of transcription factor TonEBP/OREBP.

Cell cycle delay and apoptosis in response to osmotic stress.

Cell cycle-dependent expression of thyroid hormone receptor-beta is a mechanism for variable hormone sensitivity

Cells adapted to high NaCl have many DNA breaks and impaired DNA repair both in cell culture and in vivo

Cellular response to hyperosmotic stresses

Cross-Sectional Positive Association of Serum Lipids and Blood Pressure With Serum Sodium Within the Normal Reference Range of 135-145 mmol/L.

DNA damage and osmotic regulation in the kidney

DNA double-strand breaks induced by high NaCl occur predominantly in gene deserts

Effects of expression of p53 and Gadd45 on osmotic tolerance of renal inner medullary cells.

Elevated sodium and dehydration stimulate inflammatory signaling in endothelial cells and promote atherosclerosis

High NaCl causes Mre11 to leave the nucleus, disrupting DNA damage signaling and repair

High NaCl promotes cellular senescence

High urea and NaCl carbonylate proteins in renal cells in culture and in vivo, and high urea causes 8-oxoguanine lesions in their DNA

Hypertonic stress response

Impaired angiogenesis and extracellular matrix metabolism in Autosomal-Dominant Hyper-IgE Syndrome

Increased activity of TNAP compensates for reduced adenosine production and promotes ectopic calcification in the genetic disease ACDC.

Increased insensible water loss contributes to aging related dehydration

Involvement of endogenous digitalis-like factors in voluntary selection of alcohol by rats

Ku86 preserves chromatin integrity in cells adapted to high NaCl

MKP-1 inhibits high NaCl-induced activation of p38 but does not inhibit the activation of TonEBP/OREBP: opposite roles of p38alpha and p38delta

Mediator of DNA damage checkpoint 1 (MDC1) contributes to high NaCl-induced activation of the osmoprotective transcription factor TonEBP/OREBP.

Mitochondrial dysfunction is an early event in high-NaCl-induced apoptosis of mIMCD3 cells

Mre11 is expressed in mammalian mitochondria where it binds to mitochondrial DNA.

Mutations that prevent caspase cleavage of RIPK1 cause autoinflammatory disease

Osmotic Stress and DNA Damage

Pax2 expression occurs in renal medullary epithelial cells in vivo and in cell culture, is osmoregulated, and promotes osmotic tolerance

Proliferation and osmotic tolerance of renal inner medullary epithelial cells in vivo and in cell culture

Protection of Renal Inner Medullary Epithelial Cells from Apoptosis by Hypertonic Stress-induced p53 Activation

Rapid activation of G2/M checkpoint after hypertonic stress in renal inner medullary epithelial (IME) cells is protective and requires p38 kinase

Rate of increase of osmolality determines osmotic tolerance of mouse inner medullary epithelial cells

STAT3 modulates reprogramming efficiency of human somatic cells; Insights from autosomal dominant Hyper IgE syndrome caused by STAT3 mutations

Secretion of von Willebrand factor by endothelial cells links sodium to hypercoagulability and thrombosis

Suboptimal hydration remodels metabolism, promotes degenerative diseases, and shortens life

The Saltiness of the Sea Breaks DNA in Marine Invertebrates: Possible Implications for Animal Evolution

Toxicity of acetaminophen, salicylic acid, and caffeine for first-passage rat renal inner medullary collecting duct cells