Lista de obras - Linda M. Scott - Dominio Público Uruguay

Acquired mutations that affect pre-mRNA splicing in hematologic malignancies and solid tumors

artículo científico publicado en 2013

Alpha4 and beta2 integrins have nonredundant roles for asthma development, but for optimal allergen sensitization only alpha4 is critical.

artículo científico publicado en 2007

Deletion of alpha4 integrins from adult hematopoietic cells reveals roles in homeostasis, regeneration, and homing.

artículo científico publicado en 2003

Deregulated JAK/STAT signalling in lymphomagenesis, and its implications for the development of new targeted therapies

artículo científico

Inhibition of the Bcl-xL deamidation pathway in myeloproliferative disorders

artículo científico publicado en 2008

JAK2 V617F impairs hematopoietic stem cell function in a conditional knock-in mouse model of JAK2 V617F-positive essential thrombocythemia

artículo científico publicado en 2010

Lack of alpha4 integrin expression in stem cells restricts competitive function and self-renewal activity

artículo científico publicado en 2005

Lymphoid malignancies: Another face to the Janus kinases.

artículo científico

MPL mutations in myeloproliferative disorders: analysis of the PT-1 cohort.

artículo científico publicado en 2008

Molecular pathways in bone marrow homing: dominant role of alpha(4)beta(1) over beta(2)-integrins and selectins

artículo científico publicado en 2001

Mutation of JAK2 in the myeloproliferative disorders: timing, clonality studies, cytogenetic associations, and role in leukemic transformation

artículo científico publicado en 2006

Mutations in exon 12 of JAK2 are mainly found in JAK2 V617F-negative polycythaemia vera patients.

artículo científico publicado en 2008

Mutations of JAK2 in acute lymphoblastic leukaemias associated with Down's syndrome

artículo científico publicado en 2008

Prevalance of JAK2 V617F and exon 12 mutations in polycythaemia vera

artículo científico publicado en 2007

Progenitor genotyping reveals a complex clonal architecture in a subset of CALR-mutated myeloproliferative neoplasms.

artículo científico publicado en 2017

Rapid identification of JAK2 exon 12 mutations using high resolution melting analysis

artículo científico publicado en 2008

Revisiting the case for genetically engineered mouse models in human myelodysplastic syndrome research

artículo científico publicado en 2015

SOCS3 tyrosine phosphorylation as a potential bio-marker for myeloproliferative neoplasms associated with mutant JAK2 kinases

artículo científico publicado en 2009

Synergistic mobilization of hemopoietic progenitor cells using concurrent beta1 and beta2 integrin blockade or beta2-deficient mice.

artículo científico publicado en 2001

The JAK2 46/1 haplotype predisposes to MPL-mutated myeloproliferative neoplasms

artículo científico publicado en 2010

The JAK2 exon 12 mutations: A comprehensive review ⬇️

artículo científico publicado el 14 de junio de 2011

The V617F JAK2 mutation is uncommon in cancers and in myeloid malignancies other than the classic myeloproliferative disorders

artículo científico publicado en 2005

The frequency of JAK2 exon 12 mutations in idiopathic erythrocytosis patients with low serum erythropoietin levels

artículo científico publicado en 2007

Transgenic Cre expression mice for generation of erythroid-specific gene alterations

artículo científico publicado en 2004

VCAM-1 expression in adult hematopoietic and nonhematopoietic cells is controlled by tissue-inductive signals and reflects their developmental origin.

artículo científico publicado en 2005

Lista de obras de Linda M. Scott

Acquired mutations that affect pre-mRNA splicing in hematologic malignancies and solid tumors

Alpha4 and beta2 integrins have nonredundant roles for asthma development, but for optimal allergen sensitization only alpha4 is critical.

Deletion of alpha4 integrins from adult hematopoietic cells reveals roles in homeostasis, regeneration, and homing.

Deregulated JAK/STAT signalling in lymphomagenesis, and its implications for the development of new targeted therapies

Inhibition of the Bcl-xL deamidation pathway in myeloproliferative disorders

JAK2 V617F impairs hematopoietic stem cell function in a conditional knock-in mouse model of JAK2 V617F-positive essential thrombocythemia

Lack of alpha4 integrin expression in stem cells restricts competitive function and self-renewal activity

Lymphoid malignancies: Another face to the Janus kinases.

MPL mutations in myeloproliferative disorders: analysis of the PT-1 cohort.

Molecular pathways in bone marrow homing: dominant role of alpha(4)beta(1) over beta(2)-integrins and selectins

Mutation of JAK2 in the myeloproliferative disorders: timing, clonality studies, cytogenetic associations, and role in leukemic transformation

Mutations in exon 12 of JAK2 are mainly found in JAK2 V617F-negative polycythaemia vera patients.

Mutations of JAK2 in acute lymphoblastic leukaemias associated with Down's syndrome

Prevalance of JAK2 V617F and exon 12 mutations in polycythaemia vera

Progenitor genotyping reveals a complex clonal architecture in a subset of CALR-mutated myeloproliferative neoplasms.

Rapid identification of JAK2 exon 12 mutations using high resolution melting analysis

Revisiting the case for genetically engineered mouse models in human myelodysplastic syndrome research

SOCS3 tyrosine phosphorylation as a potential bio-marker for myeloproliferative neoplasms associated with mutant JAK2 kinases

Synergistic mobilization of hemopoietic progenitor cells using concurrent beta1 and beta2 integrin blockade or beta2-deficient mice.

The JAK2 46/1 haplotype predisposes to MPL-mutated myeloproliferative neoplasms

The JAK2 exon 12 mutations: A comprehensive review ⬇️

The V617F JAK2 mutation is uncommon in cancers and in myeloid malignancies other than the classic myeloproliferative disorders

The frequency of JAK2 exon 12 mutations in idiopathic erythrocytosis patients with low serum erythropoietin levels

Transgenic Cre expression mice for generation of erythroid-specific gene alterations

VCAM-1 expression in adult hematopoietic and nonhematopoietic cells is controlled by tissue-inductive signals and reflects their developmental origin.