A computational framework for a Lyapunov-enabled analysis of biochemical reaction networks

A mathematical model exhibiting the effect of DNA methylation on the stability boundary in cell-fate networks

A note on dynamical compensation and its relation to parameter identifiability

A novel COVID-19 epidemiological model with explicit susceptible and asymptomatic isolation compartments reveals unexpected consequences of timing social distancing

A synthetic distributed genetic multi-bit counter

An explicit formula for minimizing the infected peak in an SIR epidemic model when using a fixed number of complete lockdowns

Bell-shaped dose response for a system with no IFFLs

Combining model-based and data-driven models: an application to synthetic biology resource competition

Cumulative dose responses for adapting biological systems

Delaying Cancer Progression by Integrating Toxicity Constraints in a Model of Adaptive Therapy

Distributed implementation of Boolean functions by transcriptional synthetic circuits

Identifying competition phenotypes in synthetic biochemical circuits

Immunobiochemical reconstruction of influenza lung infection - Melanoma skin cancer interactions

Incoherent feedforward motifs as immune change detectors

Inferring reaction network structure from single-cell, multiplex data, using toric systems theory

Integrating multimodal data sets into a mathematical framework to describe and predict therapeutic resistance in cancer

Minimizing the infected peak utilizing a single lockdown: a technical result regarding equal peaks

Paradoxical gene regulation explained by competition for genomic sites

Perfect adaptation of CD8+ T cell responses to constant antigen input over a wide range of affinity is overcome by costimulation

The interaction between dynamic ligand signaling and epigenetics in Notch-induced cancer metastasis

Understanding therapeutic tolerance through a mathematical model of drug-induced resistance

Universal Features of Epidemic Models Under Social Distancing Guidelines

Validation of a Mathematical Model of Cancer Incorporating Spontaneous and Induced Evolution to Drug Resistance