Lista de obras -

2019 - A year of celebrations

scientific article published on 28 October 2019

A fluorescent reporter reveals on/off regulation of the Shigella type III secretion apparatus during entry and cell-to-cell spread

artículo científico publicado en 2014

A secreted anti-activator, OspD1, and its chaperone, Spa15, are involved in the control of transcription by the type III secretion apparatus activity in Shigella flexneri

artículo científico publicado en 2005

An injected bacterial effector targets chromatin access for transcription factor NF-kappaB to alter transcription of host genes involved in immune responses

artículo científico publicado en 2006

Analysis of virulence plasmid gene expression defines three classes of effectors in the type III secretion system of Shigella flexneri

artículo científico publicado en 2005

B lymphocytes undergo TLR2-dependent apoptosis upon Shigella infection

artículo científico publicado en 2014

Cell regulation

scientific article published on 21 March 2008

Chaperones of the type III secretion pathway: jacks of all trades

artículo científico publicado en 2002

Characterization of the promoter, MxiE box and 5' UTR of genes controlled by the activity of the type III secretion apparatus in Shigella flexneri

artículo científico publicado en 2012

Conversion of PtdIns(4,5)P(2) into PtdIns(5)P by the S.flexneri effector IpgD reorganizes host cell morphology

artículo científico publicado en 2002

Distinct mutations led to inactivation of type 1 fimbriae expression in Shigella spp

artículo científico publicado en 2015

Escape of Actively Secreting Shigella flexneri from ATG8/LC3-Positive Vacuoles Formed during Cell-To-Cell Spread Is Facilitated by IcsB and VirA.

artículo científico publicado en 2015

Frameshifting by transcriptional slippage is involved in production of MxiE, the transcription activator regulated by the activity of the type III secretion apparatus in Shigella flexneri.

artículo científico publicado en 2005

Glycan-Glycan Interaction Determines Shigella Tropism toward Human T Lymphocytes

artículo científico publicado en 2018

Identification of substrates and chaperone from the Yersinia enterocolitica 1B Ysa type III secretion system

artículo científico publicado en 2003

Identification of the cis-acting site involved in activation of promoters regulated by activity of the type III secretion apparatus in Shigella flexneri

artículo científico publicado en 2002

Injection of T3SS effectors not resulting in invasion is the main targeting mechanism of Shigella toward human lymphocytes

artículo científico

IpaD is localized at the tip of the Shigella flexneri type III secretion apparatus

artículo científico publicado en 2006

IpgB1 and IpgB2, two homologous effectors secreted via the Mxi-Spa type III secretion apparatus, cooperate to mediate polarized cell invasion and inflammatory potential of Shigella flexenri

artículo científico publicado en 2007

MxiC is secreted by and controls the substrate specificity of the Shigella flexneri type III secretion apparatus

artículo científico publicado en 2008

MxiK and MxiN interact with the Spa47 ATPase and are required for transit of the needle components MxiH and MxiI, but not of Ipa proteins, through the type III secretion apparatus of Shigella flexneri

artículo científico publicado en 2003

Optimization of virulence functions through glucosylation of Shigella LPS.

artículo científico publicado en 2005

Regulation of transcription by the activity of the Shigella flexneri type III secretion apparatus.

artículo científico publicado en 2002

Shigella spp. and enteroinvasive Escherichia coli pathogenicity factors

artículo científico publicado en 2005

Shigella type III secretion effectors: how, where, when, for what purposes?

artículo científico publicado en 2009

Spa15 of Shigella flexneri, a third type of chaperone in the type III secretion pathway

artículo científico publicado en 2002

Structural basis for the inhibition of host protein ubiquitination by Shigella effector kinase OspG

artículo científico publicado en 2014

Structural mimicry for vinculin activation by IpaA, a virulence factor of Shigella flexneri

artículo científico publicado en 2006

Structure of Spa15, a type III secretion chaperone from Shigella flexneri with broad specificity

artículo científico publicado en 2004

Structure of the Shigella T3SS effector IpaH defines a new class of E3 ubiquitin ligases

artículo científico publicado en 2008

The 33 carboxyl-terminal residues of Spa40 orchestrate the multi-step assembly process of the type III secretion needle complex in Shigella flexneri

artículo científico publicado en 2010

The Evolutionary History of Shigella and Enteroinvasive Escherichia coli Revised ⬇️

artículo científico publicado el 1 de agosto de 2003

The Shigella flexneri effector OspG interferes with innate immune responses by targeting ubiquitin-conjugating enzymes

artículo científico publicado en 2005

The various and varying roles of specific chaperones in type III secretion systems.

artículo científico publicado en 2003

Transcriptional slippage controls production of type III secretion apparatus components in Shigella flexneri

artículo científico publicado en 2006

Transcriptional slippage in mxiE controls transcription and translation of the downstream mxiD gene, which encodes a component of the Shigella flexneri type III secretion apparatus

artículo científico publicado en 2006

Two msbB genes encoding maximal acylation of lipid A are required for invasive Shigella flexneri to mediate inflammatory rupture and destruction of the intestinal epithelium

artículo científico publicado en 2002

Type III secretion effectors of the IpaH family are E3 ubiquitin ligases

artículo científico publicado en 2007

[Control of the host inflammatory response during Shigella flexneri infection]

artículo científico publicado en 2006

Lista de obras de

2019 - A year of celebrations

A fluorescent reporter reveals on/off regulation of the Shigella type III secretion apparatus during entry and cell-to-cell spread

A secreted anti-activator, OspD1, and its chaperone, Spa15, are involved in the control of transcription by the type III secretion apparatus activity in Shigella flexneri

An injected bacterial effector targets chromatin access for transcription factor NF-kappaB to alter transcription of host genes involved in immune responses

Analysis of virulence plasmid gene expression defines three classes of effectors in the type III secretion system of Shigella flexneri

B lymphocytes undergo TLR2-dependent apoptosis upon Shigella infection

Cell regulation

Chaperones of the type III secretion pathway: jacks of all trades

Characterization of the promoter, MxiE box and 5' UTR of genes controlled by the activity of the type III secretion apparatus in Shigella flexneri

Conversion of PtdIns(4,5)P(2) into PtdIns(5)P by the S.flexneri effector IpgD reorganizes host cell morphology

Distinct mutations led to inactivation of type 1 fimbriae expression in Shigella spp

Escape of Actively Secreting Shigella flexneri from ATG8/LC3-Positive Vacuoles Formed during Cell-To-Cell Spread Is Facilitated by IcsB and VirA.

Frameshifting by transcriptional slippage is involved in production of MxiE, the transcription activator regulated by the activity of the type III secretion apparatus in Shigella flexneri.

Glycan-Glycan Interaction Determines Shigella Tropism toward Human T Lymphocytes

Identification of substrates and chaperone from the Yersinia enterocolitica 1B Ysa type III secretion system

Identification of the cis-acting site involved in activation of promoters regulated by activity of the type III secretion apparatus in Shigella flexneri

Injection of T3SS effectors not resulting in invasion is the main targeting mechanism of Shigella toward human lymphocytes

IpaD is localized at the tip of the Shigella flexneri type III secretion apparatus

IpgB1 and IpgB2, two homologous effectors secreted via the Mxi-Spa type III secretion apparatus, cooperate to mediate polarized cell invasion and inflammatory potential of Shigella flexenri

MxiC is secreted by and controls the substrate specificity of the Shigella flexneri type III secretion apparatus

MxiK and MxiN interact with the Spa47 ATPase and are required for transit of the needle components MxiH and MxiI, but not of Ipa proteins, through the type III secretion apparatus of Shigella flexneri

Optimization of virulence functions through glucosylation of Shigella LPS.

Regulation of transcription by the activity of the Shigella flexneri type III secretion apparatus.

Shigella spp. and enteroinvasive Escherichia coli pathogenicity factors

Shigella type III secretion effectors: how, where, when, for what purposes?

Spa15 of Shigella flexneri, a third type of chaperone in the type III secretion pathway

Structural basis for the inhibition of host protein ubiquitination by Shigella effector kinase OspG

Structural mimicry for vinculin activation by IpaA, a virulence factor of Shigella flexneri

Structure of Spa15, a type III secretion chaperone from Shigella flexneri with broad specificity

Structure of the Shigella T3SS effector IpaH defines a new class of E3 ubiquitin ligases

The 33 carboxyl-terminal residues of Spa40 orchestrate the multi-step assembly process of the type III secretion needle complex in Shigella flexneri

The Evolutionary History of Shigella and Enteroinvasive Escherichia coli Revised ⬇️

The Shigella flexneri effector OspG interferes with innate immune responses by targeting ubiquitin-conjugating enzymes

The various and varying roles of specific chaperones in type III secretion systems.

Transcriptional slippage controls production of type III secretion apparatus components in Shigella flexneri

Transcriptional slippage in mxiE controls transcription and translation of the downstream mxiD gene, which encodes a component of the Shigella flexneri type III secretion apparatus

Two msbB genes encoding maximal acylation of lipid A are required for invasive Shigella flexneri to mediate inflammatory rupture and destruction of the intestinal epithelium

Type III secretion effectors of the IpaH family are E3 ubiquitin ligases

[Control of the host inflammatory response during Shigella flexneri infection]