Lista de obras -

A functional cancer genomics screen identifies a druggable synthetic lethal interaction between MSH3 and PRKDC.

artículo científico publicado en 2014

A simple and fast method for the simultaneous detection of nine fibroblast growth factor receptor 3 mutations in bladder cancer and voided urine

artículo científico publicado en 2005

Chromosome 9 deletions are more frequent than FGFR3 mutations in flat urothelial hyperplasias of the bladder.

artículo científico publicado en 2006

FGFR3 mutations and a normal CK20 staining pattern define low-grade noninvasive urothelial bladder tumours

artículo científico publicado en 2007

FGFR3 mutations in seborrheic keratoses are already present in flat lesions and associated with age and localization

scientific article published on 22 June 2007

FGFR3 mutations indicate better survival in invasive upper urinary tract and bladder tumours.

artículo científico publicado en 2008

Fibroblast growth factor receptor 3 mutations in bladder tumors correlate with low frequency of chromosome alterations

artículo científico publicado en 2008

High frequency of FGFR3 mutations in adenoid seborrheic keratoses

artículo científico publicado en 2006

Induction of CAMEL/NY-ESO-ORF2-specific CD8+ T cells upon stimulation with dendritic cells infected with a modified Ad5 vector expressing a chimeric Ad5/35 fiber

artículo científico publicado en 2004

Low frequency of molecular changes and tumor recurrence in inverted papillomas of the urinary tract.

artículo científico publicado en 2007

Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia.

artículo científico publicado en 2015

Minimal Reduction of PU.1 Is Sufficient to Induce a Preleukemic State and Promote Development of Acute Myeloid Leukemia ⬇️

artículo científico publicado en 2015

Mosaicism of activating FGFR3 mutations in human skin causes epidermal nevi

artículo científico publicado en 2006

PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance

artículo científico publicado en 2010

Prediction of Progression of Non–Muscle-Invasive Bladder Cancer by WHO 1973 and 2004 Grading and by FGFR3 Mutation Status: A Prospective Study

article

The ATPase activity of MLH1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination

artículo científico publicado en 2012

The absence of BRAF, FGFR3, and PIK3CA mutations differentiates lentigo simplex from melanocytic nevus and solar lentigo

artículo científico publicado en 2009

[Correlation of FGFR3 mutations and chromosomal alterations in bladder cancer]

artículo científico publicado en 2006

[Urothelial neoplasms in individuals younger than 20 years show very few genetic alterations and have a favourable clinical outcome]

artículo científico publicado en 2006

Lista de obras de

A functional cancer genomics screen identifies a druggable synthetic lethal interaction between MSH3 and PRKDC.

A simple and fast method for the simultaneous detection of nine fibroblast growth factor receptor 3 mutations in bladder cancer and voided urine

Chromosome 9 deletions are more frequent than FGFR3 mutations in flat urothelial hyperplasias of the bladder.

FGFR3 mutations and a normal CK20 staining pattern define low-grade noninvasive urothelial bladder tumours

FGFR3 mutations in seborrheic keratoses are already present in flat lesions and associated with age and localization

FGFR3 mutations indicate better survival in invasive upper urinary tract and bladder tumours.

Fibroblast growth factor receptor 3 mutations in bladder tumors correlate with low frequency of chromosome alterations

High frequency of FGFR3 mutations in adenoid seborrheic keratoses

Induction of CAMEL/NY-ESO-ORF2-specific CD8+ T cells upon stimulation with dendritic cells infected with a modified Ad5 vector expressing a chimeric Ad5/35 fiber

Low frequency of molecular changes and tumor recurrence in inverted papillomas of the urinary tract.

Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia.

Minimal Reduction of PU.1 Is Sufficient to Induce a Preleukemic State and Promote Development of Acute Myeloid Leukemia ⬇️

Mosaicism of activating FGFR3 mutations in human skin causes epidermal nevi

PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance

Prediction of Progression of Non–Muscle-Invasive Bladder Cancer by WHO 1973 and 2004 Grading and by FGFR3 Mutation Status: A Prospective Study

The ATPase activity of MLH1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination

The absence of BRAF, FGFR3, and PIK3CA mutations differentiates lentigo simplex from melanocytic nevus and solar lentigo

[Correlation of FGFR3 mutations and chromosomal alterations in bladder cancer]

[Urothelial neoplasms in individuals younger than 20 years show very few genetic alterations and have a favourable clinical outcome]