Lista de obras -

A key role for NOX4 in epithelial cell death during development of lung fibrosis.

artículo científico publicado en 2011

BARD1 mediates TGF-β signaling in pulmonary fibrosis.

artículo científico publicado en 2015

Bcl-2 overexpression in type II epithelial cells does not prevent hyperoxia-induced acute lung injury in mice

artículo científico publicado en 2010

Bcl-2 protects against hyperoxia-induced apoptosis through inhibition of the mitochondria-dependent pathway

artículo científico publicado en 2007

CD4+CD25-mTGFbeta+ T cells induced by nasal application of ovalbumin transfer tolerance in a therapeutic model of asthma

artículo científico publicado en 2010

CD40-CD40 Ligand Disruption Does Not Prevent Hyperoxia-Induced Injury ⬇️

artículo científico publicado el 1 de enero de 2002

Early life exposure to nicotine modifies lung gene response after elastase-induced emphysema

artículo científico publicado en 2022

Gestation and lactation exposure to nicotine induces transient postnatal changes in lung alveolar development

scientific article published on 22 January 2020

Glucocorticoids aggravate hyperoxia-induced lung injury through decreased nuclear factor-kappa B activity

artículo científico publicado en 2002

Hyperoxia-mediated oxidative stress increases expression of UCP3 mRNA and protein in skeletal muscle

artículo científico publicado en 2005

In vivo investigations on anti-fibrotic potential of proteasome inhibition in lung and skin fibrosis.

artículo científico publicado en 2008

Macrophage-specific NOX2 contributes to the development of lung emphysema through modulation of SIRT1/MMP-9 pathways

artículo científico publicado en 2014

Mitochondrial cytochromecrelease is a key event in hyperoxia-induced lung injury: protection by cyclosporin A ⬇️

artículo científico publicado el 3 de octubre de 2003

NADPH oxidase-1 plays a crucial role in hyperoxia-induced acute lung injury in mice

scientific journal article

NOX-4 is expressed in thickened pulmonary arteries in idiopathic pulmonary fibrosis

artículo científico publicado en 2011

NOX1 is responsible for cell death through STAT3 activation in hyperoxia and is associated with the pathogenesis of acute respiratory distress syndrome

artículo científico publicado en 2014

Nicotine mediates oxidative stress and apoptosis through cross talk between NOX1 and Bcl-2 in lung epithelial cells

artículo científico publicado en 2014

Poly(ADP-ribose) polymerase-1 (PARP-1) controls lung cell proliferation and repair after hyperoxia-induced lung damage

scientific article published on 15 June 2007

Poly(ADP-ribose)polymerase activation mediates lung epithelial cell death in vitro but is not essential in hyperoxia-induced lung injury

artículo científico publicado en 2005

Prevention of hyperoxia-induced bronchial hyperreactivity by sildenafil and vasoactive intestinal peptide: impact of preserved lung function and structure

artículo científico publicado en 2014

Simultaneous isolation of endothelial and alveolar epithelial type I and type II cells during mouse lung development in the absence of a transgenic reporter

scientific article published on 05 February 2020

The role of endothelin-1 in hyperoxia-induced lung injury in mice

artículo científico publicado en 2006

Lista de obras de

A key role for NOX4 in epithelial cell death during development of lung fibrosis.

BARD1 mediates TGF-β signaling in pulmonary fibrosis.

Bcl-2 overexpression in type II epithelial cells does not prevent hyperoxia-induced acute lung injury in mice

Bcl-2 protects against hyperoxia-induced apoptosis through inhibition of the mitochondria-dependent pathway

CD4+CD25-mTGFbeta+ T cells induced by nasal application of ovalbumin transfer tolerance in a therapeutic model of asthma

CD40-CD40 Ligand Disruption Does Not Prevent Hyperoxia-Induced Injury ⬇️

Early life exposure to nicotine modifies lung gene response after elastase-induced emphysema

Gestation and lactation exposure to nicotine induces transient postnatal changes in lung alveolar development

Glucocorticoids aggravate hyperoxia-induced lung injury through decreased nuclear factor-kappa B activity

Hyperoxia-mediated oxidative stress increases expression of UCP3 mRNA and protein in skeletal muscle

In vivo investigations on anti-fibrotic potential of proteasome inhibition in lung and skin fibrosis.

Macrophage-specific NOX2 contributes to the development of lung emphysema through modulation of SIRT1/MMP-9 pathways

Mitochondrial cytochromecrelease is a key event in hyperoxia-induced lung injury: protection by cyclosporin A ⬇️

NADPH oxidase-1 plays a crucial role in hyperoxia-induced acute lung injury in mice

NOX-4 is expressed in thickened pulmonary arteries in idiopathic pulmonary fibrosis

NOX1 is responsible for cell death through STAT3 activation in hyperoxia and is associated with the pathogenesis of acute respiratory distress syndrome

Nicotine mediates oxidative stress and apoptosis through cross talk between NOX1 and Bcl-2 in lung epithelial cells

Poly(ADP-ribose) polymerase-1 (PARP-1) controls lung cell proliferation and repair after hyperoxia-induced lung damage

Poly(ADP-ribose)polymerase activation mediates lung epithelial cell death in vitro but is not essential in hyperoxia-induced lung injury

Prevention of hyperoxia-induced bronchial hyperreactivity by sildenafil and vasoactive intestinal peptide: impact of preserved lung function and structure

Simultaneous isolation of endothelial and alveolar epithelial type I and type II cells during mouse lung development in the absence of a transgenic reporter

The role of endothelin-1 in hyperoxia-induced lung injury in mice