Lista de obras -

A modified version of a Fos-associated cluster in HBZ affects Jun transcriptional potency

artículo científico publicado en 2006

Activation of HTLV-I gene transcription by protein tyrosine phosphatase inhibitors

artículo científico publicado en 2004

An interaction between the human T cell leukemia virus type 1 basic leucine zipper factor (HBZ) and the KIX domain of p300/CBP contributes to the down-regulation of tax-dependent viral transcription by HBZ

artículo científico publicado en 2008

Construction of a reporter vector for analysis of bidirectional transcriptional activity of retrovirus LTR.

artículo científico publicado en 2014

Detection of the HIV-1 minus-strand-encoded antisense protein and its association with autophagy

artículo científico publicado en 2013

Detection, characterization and regulation of antisense transcripts in HIV-1.

artículo científico publicado en 2007

Does chronic infection in retroviruses have a sense?

artículo científico publicado en 2015

Does the HBZ gene represent a new potential target for the treatment of adult T-cell leukemia?

artículo científico publicado en 2007

Functional comparison of antisense proteins of HTLV-1 and HTLV-2 in viral pathogenesis

artículo científico publicado en 2013

HIV-1 antisense transcription is preferentially activated in primary monocyte-derived cells.

artículo científico publicado en 2012

HTLV-I antisense transcripts initiating in the 3'LTR are alternatively spliced and polyadenylated

artículo científico publicado en 2006

Hijacking of the AP-1 Signaling Pathway during Development of ATL.

artículo científico publicado en 2017

Human T-cell leukemia virus type 1 (HTLV-1) bZIP factor requires cellular transcription factor JunD to upregulate HTLV-1 antisense transcription from the 3' long terminal repeat

artículo científico publicado en 2012

Human T-cell leukemia virus type 1 (HTLV-1) bZIP protein interacts with the cellular transcription factor CREB to inhibit HTLV-1 transcription.

artículo científico publicado en 2006

Human T-cell leukemia virus type 3 (HTLV-3) and HTLV-4 antisense-transcript-encoded proteins interact and transactivate Jun family-dependent transcription via their atypical bZIP motif

artículo científico publicado en 2014

Making sense out of antisense transcription in human T-cell lymphotropic viruses (HTLVs).

artículo científico publicado en 2011

Propensity for HBZ-SP1 isoform of HTLV-I to inhibit c-Jun activity correlates with sequestration of c-Jun into nuclear bodies rather than inhibition of its DNA-binding activity

artículo científico publicado en 2009

Reviving an old HIV-1 gene: the HIV-1 antisense protein

artículo científico publicado en 2015

Roles of HTLV-1 basic Zip Factor (HBZ) in Viral Chronicity and Leukemic Transformation. Potential New Therapeutic Approaches to Prevent and Treat HTLV-1-Related Diseases

artículo científico publicado en 2015

The HBZ-SP1 isoform of human T-cell leukemia virus type I represses JunB activity by sequestration into nuclear bodies.

artículo científico publicado en 2007

Upregulation of human T-cell leukemia virus type 1 antisense transcription by the viral tax protein

scientific article published on 10 December 2008

Lista de obras de

A modified version of a Fos-associated cluster in HBZ affects Jun transcriptional potency

Activation of HTLV-I gene transcription by protein tyrosine phosphatase inhibitors

An interaction between the human T cell leukemia virus type 1 basic leucine zipper factor (HBZ) and the KIX domain of p300/CBP contributes to the down-regulation of tax-dependent viral transcription by HBZ

Construction of a reporter vector for analysis of bidirectional transcriptional activity of retrovirus LTR.

Detection of the HIV-1 minus-strand-encoded antisense protein and its association with autophagy

Detection, characterization and regulation of antisense transcripts in HIV-1.

Does chronic infection in retroviruses have a sense?

Does the HBZ gene represent a new potential target for the treatment of adult T-cell leukemia?

Functional comparison of antisense proteins of HTLV-1 and HTLV-2 in viral pathogenesis

HIV-1 antisense transcription is preferentially activated in primary monocyte-derived cells.

HTLV-I antisense transcripts initiating in the 3'LTR are alternatively spliced and polyadenylated

Hijacking of the AP-1 Signaling Pathway during Development of ATL.

Human T-cell leukemia virus type 1 (HTLV-1) bZIP factor requires cellular transcription factor JunD to upregulate HTLV-1 antisense transcription from the 3' long terminal repeat

Human T-cell leukemia virus type 1 (HTLV-1) bZIP protein interacts with the cellular transcription factor CREB to inhibit HTLV-1 transcription.

Human T-cell leukemia virus type 3 (HTLV-3) and HTLV-4 antisense-transcript-encoded proteins interact and transactivate Jun family-dependent transcription via their atypical bZIP motif

Making sense out of antisense transcription in human T-cell lymphotropic viruses (HTLVs).

Propensity for HBZ-SP1 isoform of HTLV-I to inhibit c-Jun activity correlates with sequestration of c-Jun into nuclear bodies rather than inhibition of its DNA-binding activity

Reviving an old HIV-1 gene: the HIV-1 antisense protein

Roles of HTLV-1 basic Zip Factor (HBZ) in Viral Chronicity and Leukemic Transformation. Potential New Therapeutic Approaches to Prevent and Treat HTLV-1-Related Diseases

The HBZ-SP1 isoform of human T-cell leukemia virus type I represses JunB activity by sequestration into nuclear bodies.

Upregulation of human T-cell leukemia virus type 1 antisense transcription by the viral tax protein