Lista de obras -

A GM-CSF/IL-33 pathway facilitates allergic airway responses to sub-threshold house dust mite exposure

artículo científico publicado en 2014

Acute, but not resolved, influenza A infection enhances susceptibility to house dust mite-induced allergic disease

artículo científico publicado en 2009

B7RP-1 is not required for the generation of Th2 responses in a model of allergic airway inflammation but is essential for the induction of inhalation tolerance.

artículo científico publicado en 2005

Concurrent blockade of platelet-activating factor and histamine prevents life-threatening peanut-induced anaphylactic reactions

artículo científico publicado en 2009

Cutaneous antigen priming via gene gun leads to skin-selective Th2 immune-inflammatory responses

artículo científico publicado en 2005

Effect of GM-CSF on immune, inflammatory, and clinical responses to ragweed in a novel mouse model of mucosal sensitization

artículo científico publicado en 2003

Evaluation of inducible costimulator/B7-related protein-1 as a therapeutic target in a murine model of allergic airway inflammation

artículo científico publicado en 2003

House dust mite facilitates ovalbumin-specific allergic sensitization and airway inflammation

artículo científico publicado en 2005

IL-33, but not thymic stromal lymphopoietin or IL-25, is central to mite and peanut allergic sensitization.

artículo científico publicado en 2012

Impact of CD40 ligand, B cells, and mast cells in peanut-induced anaphylactic responses

artículo científico publicado en 2007

In vivo-to-in silico iterations to investigate aeroallergen-host interactions

artículo científico publicado en 2008

Indigenous enteric eosinophils control DCs to initiate a primary Th2 immune response in vivo

artículo científico publicado en 2014

Intranasal exposure of mice to house dust mite elicits allergic airway inflammation via a GM-CSF-mediated mechanism

artículo científico publicado en 2004

Prediction of clinical peanut allergy status among children in Hamilton, Ontario using chart review data collected during 2012-2015.

artículo científico publicado en 2017

Therapeutic potential of anti-IL-6 therapies for granulocytic airway inflammation in asthma

artículo científico publicado en 2015

Transient corticosteroid treatment permanently amplifies the Th2 response in a murine model of asthma

artículo científico publicado en 2004

Lista de obras de

A GM-CSF/IL-33 pathway facilitates allergic airway responses to sub-threshold house dust mite exposure

Acute, but not resolved, influenza A infection enhances susceptibility to house dust mite-induced allergic disease

B7RP-1 is not required for the generation of Th2 responses in a model of allergic airway inflammation but is essential for the induction of inhalation tolerance.

Concurrent blockade of platelet-activating factor and histamine prevents life-threatening peanut-induced anaphylactic reactions

Cutaneous antigen priming via gene gun leads to skin-selective Th2 immune-inflammatory responses

Effect of GM-CSF on immune, inflammatory, and clinical responses to ragweed in a novel mouse model of mucosal sensitization

Evaluation of inducible costimulator/B7-related protein-1 as a therapeutic target in a murine model of allergic airway inflammation

House dust mite facilitates ovalbumin-specific allergic sensitization and airway inflammation

IL-33, but not thymic stromal lymphopoietin or IL-25, is central to mite and peanut allergic sensitization.

Impact of CD40 ligand, B cells, and mast cells in peanut-induced anaphylactic responses

In vivo-to-in silico iterations to investigate aeroallergen-host interactions

Indigenous enteric eosinophils control DCs to initiate a primary Th2 immune response in vivo

Intranasal exposure of mice to house dust mite elicits allergic airway inflammation via a GM-CSF-mediated mechanism

Prediction of clinical peanut allergy status among children in Hamilton, Ontario using chart review data collected during 2012-2015.

Therapeutic potential of anti-IL-6 therapies for granulocytic airway inflammation in asthma

Transient corticosteroid treatment permanently amplifies the Th2 response in a murine model of asthma