Lista de obras -

Cell-Mediated Release of Nanoparticles as a Preferential Option for Future Treatment of Melanoma

artículo científico publicado en 2020

Chronic Resveratrol Treatment Inhibits MRC5 Fibroblast SASP-Related Protumoral Effects on Melanoma Cells.

artículo científico publicado en 2017

Chronic Resveratrol Treatment Reduces the Pro-angiogenic Effect of Human Fibroblast "Senescent-Associated Secretory Phenotype" on Endothelial Colony-Forming Cells: The Role of IL8

artículo científico publicado en 2019

Correction: Desmoglein-2-Integrin Beta-8 Interaction Regulates Actin Assembly in Endothelial Cells: Deregulation in Systemic Sclerosis.

artículo científico publicado en 2013

Delivery systems of CRISPR/Cas9-based cancer gene therapy

article

Desmoglein-2-Integrin Beta-8 Interaction Regulates Actin Assembly in Endothelial Cells: Deregulation in Systemic Sclerosis ⬇️

artículo científico publicado el 11 de julio de 2013

Differential uPAR recruitment in caveolar-lipid rafts by GM1 and GM3 gangliosides regulates endothelial progenitor cells angiogenesis

artículo científico publicado en 2014

EGFR/uPAR interaction as druggable target to overcome vemurafenib acquired resistance in melanoma cells

artículo científico publicado en 2019

ENDOTHELIAL PROGENITOR CELLS AS SHUTTLE OF ANTICANCER AGENTS.

artículo científico publicado en 2016

Endothelial progenitor cell–dependent angiogenesis requires localization of the full-length form of uPAR in caveolae ⬇️

artículo científico publicado el 29 de julio de 2011

Erratum: Publisher Correction to: delivery systems of CRISPR/Cas9-based cancer gene therapy

article

GDF5 regulates TGFß-dependent angiogenesis in breast carcinoma MCF-7 cells: in vitro and in vivo control by anti-TGFß peptides

artículo científico publicado en 2012

Inhibition of uPAR-TGFβ crosstalk blocks MSC-dependent EMT in melanoma cells

artículo científico publicado en 2015

Lipid rafts: integrated platforms for vascular organization offering therapeutic opportunities

artículo científico

Mature and progenitor endothelial cells perform angiogenesis also under protease inhibition: the amoeboid angiogenesis.

artículo científico publicado en 2018

Melanoma cell therapy: Endothelial progenitor cells as shuttle of the MMP12 uPAR-degrading enzyme

artículo científico publicado en 2014

Modulation of the angiogenic phenotype of normal and systemic sclerosis endothelial cells by gain-loss of function of pentraxin 3 and matrix metalloproteinase 12

artículo científico publicado en 2010

Proteomic identification of VEGF-dependent protein enrichment to membrane caveolar-raft microdomains in endothelial progenitor cells.

artículo científico publicado en 2013

Reduction of in vitro invasion and in vivo cartilage degradation in a SCID mouse model by loss of function of the fibrinolytic system of rheumatoid arthritis synovial fibroblasts.

artículo científico publicado en 2011

Systemic sclerosis endothelial cells recruit and activate dermal fibroblasts by induction of a connective tissue growth factor (CCN2)/transforming growth factor β-dependent mesenchymal-to-mesenchymal transition

artículo científico publicado en 2013

Th1-Induced CD106 Expression Mediates Leukocytes Adhesion on Synovial Fibroblasts from Juvenile Idiopathic Arthritis Patients.

artículo científico publicado en 2016

Th17 lymphocyte-dependent degradation of joint cartilage by synovial fibroblasts in a humanized mouse model of arthritis and reversal by secukinumab

artículo científico publicado en 2020

The receptor for urokinase-plasminogen activator (uPAR) controls plasticity of cancer cell movement in mesenchymal and amoeboid migration style

artículo científico publicado en 2014

The urokinase receptor system, a key regulator at the intersection between inflammation, immunity, and coagulation.

artículo científico publicado en 2011

Tumor-tropic endothelial colony forming cells (ECFCs) loaded with near-infrared sensitive Au nanoparticles: A "cellular stove" approach to the photoablation of melanoma.

artículo científico publicado en 2016

Urokinase and its receptor in follicular and inflammatory cysts of the jaws.

artículo científico publicado en 2010

uPA/uPAR system activation drives a glycolytic phenotype in melanoma cells.

artículo científico publicado en 2017

uPAR Knockout Results in a Deep Glycolytic and OXPHOS Reprogramming in Melanoma and Colon Carcinoma Cell Lines

artículo científico publicado en 2020

uPAR-expressing melanoma exosomes promote angiogenesis by VE-Cadherin, EGFR and uPAR overexpression and rise of ERK1,2 signaling in endothelial cells

scientific article published on 25 November 2020

Lista de obras de

Cell-Mediated Release of Nanoparticles as a Preferential Option for Future Treatment of Melanoma

Chronic Resveratrol Treatment Inhibits MRC5 Fibroblast SASP-Related Protumoral Effects on Melanoma Cells.

Chronic Resveratrol Treatment Reduces the Pro-angiogenic Effect of Human Fibroblast "Senescent-Associated Secretory Phenotype" on Endothelial Colony-Forming Cells: The Role of IL8

Correction: Desmoglein-2-Integrin Beta-8 Interaction Regulates Actin Assembly in Endothelial Cells: Deregulation in Systemic Sclerosis.

Delivery systems of CRISPR/Cas9-based cancer gene therapy

Desmoglein-2-Integrin Beta-8 Interaction Regulates Actin Assembly in Endothelial Cells: Deregulation in Systemic Sclerosis ⬇️

Differential uPAR recruitment in caveolar-lipid rafts by GM1 and GM3 gangliosides regulates endothelial progenitor cells angiogenesis

EGFR/uPAR interaction as druggable target to overcome vemurafenib acquired resistance in melanoma cells

ENDOTHELIAL PROGENITOR CELLS AS SHUTTLE OF ANTICANCER AGENTS.

Endothelial progenitor cell–dependent angiogenesis requires localization of the full-length form of uPAR in caveolae ⬇️

Erratum: Publisher Correction to: delivery systems of CRISPR/Cas9-based cancer gene therapy

GDF5 regulates TGFß-dependent angiogenesis in breast carcinoma MCF-7 cells: in vitro and in vivo control by anti-TGFß peptides

Inhibition of uPAR-TGFβ crosstalk blocks MSC-dependent EMT in melanoma cells

Lipid rafts: integrated platforms for vascular organization offering therapeutic opportunities

Mature and progenitor endothelial cells perform angiogenesis also under protease inhibition: the amoeboid angiogenesis.

Melanoma cell therapy: Endothelial progenitor cells as shuttle of the MMP12 uPAR-degrading enzyme

Modulation of the angiogenic phenotype of normal and systemic sclerosis endothelial cells by gain-loss of function of pentraxin 3 and matrix metalloproteinase 12

Proteomic identification of VEGF-dependent protein enrichment to membrane caveolar-raft microdomains in endothelial progenitor cells.

Reduction of in vitro invasion and in vivo cartilage degradation in a SCID mouse model by loss of function of the fibrinolytic system of rheumatoid arthritis synovial fibroblasts.

Systemic sclerosis endothelial cells recruit and activate dermal fibroblasts by induction of a connective tissue growth factor (CCN2)/transforming growth factor β-dependent mesenchymal-to-mesenchymal transition

Th1-Induced CD106 Expression Mediates Leukocytes Adhesion on Synovial Fibroblasts from Juvenile Idiopathic Arthritis Patients.

Th17 lymphocyte-dependent degradation of joint cartilage by synovial fibroblasts in a humanized mouse model of arthritis and reversal by secukinumab

The receptor for urokinase-plasminogen activator (uPAR) controls plasticity of cancer cell movement in mesenchymal and amoeboid migration style

The urokinase receptor system, a key regulator at the intersection between inflammation, immunity, and coagulation.

Tumor-tropic endothelial colony forming cells (ECFCs) loaded with near-infrared sensitive Au nanoparticles: A "cellular stove" approach to the photoablation of melanoma.

Urokinase and its receptor in follicular and inflammatory cysts of the jaws.

uPA/uPAR system activation drives a glycolytic phenotype in melanoma cells.

uPAR Knockout Results in a Deep Glycolytic and OXPHOS Reprogramming in Melanoma and Colon Carcinoma Cell Lines

uPAR-expressing melanoma exosomes promote angiogenesis by VE-Cadherin, EGFR and uPAR overexpression and rise of ERK1,2 signaling in endothelial cells