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A Promising New Approach for the Treatment of Inflammatory Pain: Transfer of Stem Cell-Derived Tyrosine Hydroxylase-Positive Cells

artículo científico publicado en 2018

Acute and long-term effects of IL-6 on cultured dorsal root ganglion neurones from adult rat.

artículo científico publicado en 2005

Antinociceptive effects of tumor necrosis factor alpha neutralization in a rat model of antigen-induced arthritis: evidence of a neuronal target

artículo científico publicado en 2008

Calcitonin gene-related peptide enhances TTX-resistant sodium currents in cultured dorsal root ganglion neurons from adult rats

artículo científico publicado en 2005

Contribution of Inflammation and Bone Destruction to Pain in Arthritis: A Study in Murine Glucose-6-Phosphate Isomerase-Induced Arthritis

artículo científico publicado en 2019

Effects of differently activated rodent macrophages on sensory neurons: implications for arthritis pain.

artículo científico publicado en 2015

Experimental arthritis causes tumor necrosis factor-α-dependent infiltration of macrophages into rat dorsal root ganglia which correlates with pain-related behavior

scientific article published on 26 June 2009

Expression pattern of neuronal and skeletal muscle voltage-gated Na+ channels in the developing mouse heart

scientific journal article

Fibroblast-like synovial cells from normal and inflamed knee joints differently affect the expression of pain-related receptors in sensory neurones: a co-culture study

artículo científico publicado en 2007

Gain-of-function mutation in SCN11A causes itch and affects neurogenic inflammation and muscle function in Scn11a+/L799P mice

scientific article published on 20 August 2020

Interleukin-17 sensitizes joint nociceptors to mechanical stimuli and contributes to arthritic pain through neuronal interleukin-17 receptors in rodents

artículo científico publicado en 2012

Interleukin-17A is involved in mechanical hyperalgesia but not in the severity of murine antigen-induced arthritis

artículo científico publicado en 2017

Interleukin-6-dependent influence of nociceptive sensory neurons on antigen-induced arthritis.

artículo científico publicado en 2015

Joint pain

artículo científico publicado en 2009

Long-Lasting Activation of the Transcription Factor CREB in Sensory Neurons by Interleukin-1β During Antigen-Induced Arthritis in Rats: A Mechanism of Persistent Arthritis Pain?

artículo científico publicado en 2016

Molecular effects of interleukin-1β on dorsal root ganglion neurons: Prevention of ligand-induced internalization of the bradykinin 2 receptor and downregulation of G protein-coupled receptor kinase 2

artículo científico publicado el 29 de septiembre de 2010

Neuronal IL-17 receptor upregulates TRPV4 but not TRPV1 receptors in DRG neurons and mediates mechanical but not thermal hyperalgesia.

artículo científico publicado en 2012

Pain-related behaviors associated with persistence of mechanical hyperalgesia post antigen-induced arthritis in rats

artículo científico publicado en 2020

Prostaglandin E2 increases the expression of the neurokinin1 receptor in adult sensory neurones in culture: a novel role of prostaglandins

artículo científico publicado en 2003

Spinal interleukin-6 is an amplifier of arthritic pain in the rat

artículo científico publicado en 2012

The cytokine TNFalpha increases the proportion of DRG neurones expressing the TRPV1 receptor via the TNFR1 receptor and ERK activation

scientific article published on 01 August 2007

The proportion of TRPV1 protein-positive lumbar DRG neurones does not increase in the course of acute and chronic antigen-induced arthritis in the knee joint of the rat.

artículo científico publicado en 2004

The role of interleukin-1β in arthritic pain: main involvement in thermal, but not mechanical, hyperalgesia in rat antigen-induced arthritis

scientific article published on 01 December 2012

Transient Receptor Potential vanilloid 4 ion channel in C-fibres is involved in mechanonociception of the normal and inflamed joint

artículo científico publicado en 2019